Green Synthesized CeO2 Nanowires immobilized with Alginate-Ascorbic Acid biopolymer for Advance Oxidative Degradation of Crystal Violet

Author(s):  
Aditi Rana ◽  
Imran Hasan ◽  
Bon Heun Koo ◽  
Rais Ahmad Khan
2020 ◽  
Vol 317 ◽  
pp. 114216 ◽  
Author(s):  
Imran Hasan ◽  
Shubham Walia ◽  
Khadijah H. Alharbi ◽  
Maymonah Abu Khanjer ◽  
Ali Alsalme ◽  
...  

1974 ◽  
Vol 20 (7) ◽  
pp. 790-793 ◽  
Author(s):  
Samuel Meites ◽  
Carlotta Thompson ◽  
Ralph W Roach

Abstract Results with two ultramicro methods for plasma uric acid analysis with uricase are described: a crystal violet (gentian violet) method, modified from an automated procedure by Gochman and Schmitz [Clin. Chem. 17, 1154 (1971)] and adapted to manual analysis, and a polarographic oxygen-sensing system (Beckman Instruments, Inc.). The coefficient of variation (CV) for the crystal violet method varied between 2.1 and 5.7% for an 11-month period. Within-run precision (CV) of the polarographic method was 3.3%; day-to-day CV varied between 3.4 and 9.4%. Percentage recovery for the crystal violet method ranged between 97 and 106%, and for the polarographic method between 92 and 100%. Results of the two methods correlate well, but 5-15% lower values are obtained by the crystal violet method than by the polarographic method. Bilirubin, hemolysis, or usual plasma concentrations of ascorbic acid do not interfere significantly with either method. Advantages and disadvantages of both methods are discussed. The polarographic method requires more uricase, but is the better of the two methods.


ChemInform ◽  
2001 ◽  
Vol 32 (4) ◽  
pp. no-no
Author(s):  
Janusz Madaj ◽  
Yoko Nishikawa ◽  
V. Prakash Reddy ◽  
Peter Rinaldi ◽  
Tadao Kurata ◽  
...  

1978 ◽  
Vol 174 (3) ◽  
pp. 693-697 ◽  
Author(s):  
K Hirota ◽  
H A Itano ◽  
T S Vedvick

Substitutions on the benzene ring of nitrosobenzene did not have the same effect on oxidative haemolysis as substitutions on phenylhydrazine. We previously found that the haemolytic effect of arylhydrazines paralleled their oxidative conversion into ligands of ferrihaemoglobin. In contrast, although most substituted nitrosobenzenes that are ligands of ferrohaemoglobin caused haemolysis and most that are not ligands failed to cause nitrosoarenes appeared to be related more closely to the ease of their reduction to arylhydroxylamines than to their properties as ligands. We propose a mechanism of oxidative degradation whereby the cyclic formation of phenylhydroxylamine from nitrosobenzene within an erythrocyte leads to the accumulation of H2O2, which then reacts with ferrohaemoglobin to initiate the oxidative cleavage of haem. The posulated active intermediate in this reaction is the same as that previously proposed in the oxidative degradation of haemoglobin by phenylhydrzine and in the coupled oxidation of ascorbic acid and haemoglobin.


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