scholarly journals A fine-scale marine mammal movement model for assessing long-term aggregate noise exposure

2022 ◽  
Vol 464 ◽  
pp. 109798
Author(s):  
Ruth Joy ◽  
Robert S. Schick ◽  
Michael Dowd ◽  
Tetyana Margolina ◽  
John E. Joseph ◽  
...  
2019 ◽  
Vol 45 (2) ◽  
pp. 125-232 ◽  
Author(s):  
Brandon L. Southall ◽  
James J. Finneran ◽  
Colleen Reichmuth ◽  
Paul E. Nachtigall ◽  
Darlene R. Ketten ◽  
...  

Author(s):  
Bok Youn Kim ◽  
Cheon Tae Kim ◽  
Jung Jeung Lee ◽  
Hong Chin Park ◽  
Chang Yoon Kim ◽  
...  

2021 ◽  
Vol 17 (1) ◽  
pp. 52
Author(s):  
Brandon L. Southall
Keyword(s):  

Noise Mapping ◽  
2014 ◽  
Vol 1 (1) ◽  
Author(s):  
Ioannis Karipidis ◽  
Danielle Vienneau ◽  
Manuel Habermacher ◽  
Micha Köpflii ◽  
Mark Brink ◽  
...  

AbstractIn 2014 the three-year interdisciplinary study SiRENE (Short and Long Term Effects of Traffic Noise Exposure) was launched in Switzerland. The goal of SiRENE is to investigate acute, short- and long-term e_ects of road, railway and aircraft noise exposure on annoyance, sleep disturbances and cardio-metabolic risk.The study is based on a detailed Swiss-wide assessment of transportation noise exposure, including diurnal distributions. The exposure analysis comprises current as well as historical exposure calculations for up to 20 years in the past.We present the major challenges of compiling sufficient data to support a Swiss-wide model for all buildings and including all transport infrastructure as a basis for the subsequent SiRENE sound exposure analysis for the years 2011, 2001 and 1991. The task is particularly challenging for the early years due to poor data quality and/or lack of availability. We address the integration of geo-referenced input datasets from various sources and time periods, the assignment of tra_c noise exposure from façade points to dwelling units, as well as the processing of traffic information and statistics. Preliminary results of the noise exposure calculations are presented.


PeerJ ◽  
2018 ◽  
Vol 6 ◽  
pp. e5209 ◽  
Author(s):  
Alfonso Garmendia ◽  
Hugo Merle ◽  
Pablo Ruiz ◽  
Maria Ferriol

Although polyploidy is considered a ubiquitous process in plants, the establishment of new polyploid species may be hindered by ecological competition with parental diploid taxa. In such cases, the adaptive processes that result in the ecological divergence of diploids and polyploids can lead to their co-existence. In contrast, non-adaptive processes can lead to the co-existence of diploids and polyploids or to differentiated distributions, particularly when the minority cytotype disadvantage effect comes into play. Although large-scale studies of cytotype distributions have been widely conducted, the segregation of sympatric cytotypes on fine scales has been poorly studied. We analysed the spatial distribution and ecological requirements of the tetraploidCentaurea seridisand the diploidCentaurea asperain east Spain on a large scale, and also microspatially in contact zones where both species hybridise and give rise to sterile triploid hybrids. On the fine scale, the position of eachCentaureaindividual was recorded along with soil parameters, accompanying species cover and plant richness. On the east Spanish coast, a slight latitudinal gradient was found. TetraploidC. seridisindividuals were located northerly and diploidC. asperaindividuals southerly. Tetraploids were found only in the habitats with strong anthropogenic disturbance. In disturbed locations with well-developed semi-fixed or fixed dunes, diploids and tetraploids could co-exist and hybridise. However, on a fine scale, although taxa were spatially segregated in contact zones, they were not ecologically differentiated. This finding suggests the existence of non-adaptive processes that have led to their co-existence. Triploid hybrids were closer to diploid allogamous mothers (C. aspera) than to tetraploid autogamous fathers (C. seridis). This may result in a better ability to compete for space in the tetraploid minor cytotype, which might facilitate its long-term persistence.


2021 ◽  
Vol 11 ◽  
Author(s):  
Andi Zhang ◽  
Tianyuan Zou ◽  
Dongye Guo ◽  
Quan Wang ◽  
Yilin Shen ◽  
...  

As a stressor widely existing in daily life, noise can cause great alterations to the immune system and result in many physical and mental disorders, including noise-induced deafness, sleep disorders, cardiovascular diseases, endocrine diseases and other problems. The immune system plays a major role in maintaining homeostasis by recognizing and removing harmful substances in the body. Many studies have shown that noise may play vital roles in the occurrence and development of some immune diseases. In humans, both innate immunity and specific immunity can be influenced by noise, and different exposure durations and intensities of noise may exert various effects on the immune system. Short-term or low-intensity noise can enhance immune function, while long-term or high-intensity noise suppresses it. Noise can lead to the occurrence of noise-induced hearing loss (NIHL) through the production of autoantibodies such as anti-Hsp70 and anti-Hsp60 and exert adverse effects related to other immune-related diseases such as some autoimmune diseases and non-Hodgkin lymphoma. The neuroendocrine system, mainly including the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic-adrenal-medullary (SAM) system, is involved in the mechanisms of immune-related diseases induced by noise and gut microbiota dysfunction. In addition, noise exposure during pregnancy may be harmful to the immune system of the fetus. On the other hand, some studies have shown that music can improve immune function and alleviate the adverse effects caused by noise.


2021 ◽  
pp. 2004594
Author(s):  
Shuo Liu ◽  
Youn-Hee Lim ◽  
Marie Pedersen ◽  
Jeanette T. Jørgensen ◽  
Heresh Amini ◽  
...  

BackgroundWhile air pollution has been linked to the development of chronic obstructive pulmonary disease (COPD), evidence on the role of environmental noise is just emerging. We examined the associations of long-term exposure to air pollution and road traffic noise with COPD incidence.MethodsWe defined COPD incidence for 24 538 female nurses from the Danish Nurse Cohort (age>44 years) as the first hospital contact between baseline (1993 or 1999) and 2015. We estimated residential annual mean concentrations of particulate matter with diameter<2.5 µm (PM2.5) since 1990 and nitrogen dioxide (NO2) since 1970 by the Danish DEHM/UBM/AirGIS modeling system, and road traffic noise (Lden) since 1970 by the Nord2000 model. Time-varying Cox regression models were applied to assess the associations of air pollution and road traffic noise with COPD incidence.Results977 nurses developed COPD during 18.6 years’ mean follow-up. We observed associations with COPD for all three exposures with hazard ratios (HRs) and 95% confidence intervals (CIs) of 1.19 (1.01, 1.41) per 6.26 µg·m−3 for PM2.5, 1.13 (1.05, 1.20) per 8.19 µg·m−3 for NO2, and 1.15 (1.06, 1.25) per 10 dB for Lden. Associations with NO2 and Lden attenuated slightly after mutual adjustment, but were robust to adjustment for PM2.5. Associations with PM2.5 were attenuated to null after adjustment for either NO2 or Lden. No potential interaction effect was observed between air pollutants and noise.ConclusionsLong-term exposure to air pollution, especially traffic-related NO2, and road traffic noise were independently associated with COPD.


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