Pheochromocytoma with Acute Non-cardiac Pulmonary Edema: A Report of One Case and The Review of Literature

2021 ◽  
pp. 1-14
Author(s):  
Yuan Liu ◽  
Ning Wang ◽  
Shi Li ◽  
Ling Jiang ◽  
Chunfang Liu ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Review Of Literature

scholarly journals Giant Negative T Waves and QT Prolongation in Non-cardiogenic Pulmonary Edema: A Case Report and Review of Literature

Cureus ◽  
2018 ◽  
Author(s):  
Abdallah M Mansour ◽  
Obai Abdullah ◽  
Haytham Allaham ◽  
Cristina Danila ◽  
Sudarshan Balla
Keyword(s):  
Case Report ◽  
Pulmonary Edema ◽  
Qt Prolongation ◽  
Cardiogenic Pulmonary Edema ◽  
Review Of Literature ◽  
T Waves

scholarly journals Pulmonary Edema in Hb S/β+ thalassemia Patient Leading to Acute Chest Syndrome. A Case Report and Review of Literature

2020 ◽  
Vol 8 (10) ◽  
pp. 332-334
Author(s):  
Hira Chaudhary ◽  
Peter Gillette ◽  
Michael Ashamalla ◽  
Angeleque Hartt ◽  
Moro Salifu ◽  
...  
Keyword(s):  
Case Report ◽  
Pulmonary Edema ◽  
Acute Chest Syndrome ◽  
Review Of Literature ◽  
Thalassemia Patient ◽  
Hb S

scholarly journals Negative Pressure Pulmonary Edema- Case Series and Review of Literature

2012 ◽  
Vol 9 (4) ◽  
pp. 310-314 ◽  
Author(s):  
B Bhattarai ◽  
S Shrestha
Keyword(s):  
Pulmonary Edema ◽  
Negative Pressure ◽  
Case Series ◽  
Upper Airway ◽  
Residual Effects ◽  
Review Of Literature ◽  
Predisposing Factor ◽  
Negative Pressure Pulmonary Edema ◽  
Life Threatening ◽  
Life Threatening Complication

Post obstructive pulmonary edema (POPE) also known as Negative pressure pulmonary edema (NPPE)is potentially life threatening complication. It occurs in about 0.1% of anesthetics and is related to upper airway obstruction. Two types have been described in literature. Different etiology has been attributed to development of Negative pressure pulmonary edema. Early identification and treatment of predisposing factor along with proper monitoring of this complication early treatment should be instituted because resolution is also fast and in most cases without residual effects. DOI: http://dx.doi.org/10.3126/kumj.v9i4.6352 Kathmandu Univ Med J 2011;9(4):310-4

Pulmonary Edema and Simultaneous Cardiac Dysfunction After Epileptic Seizures

2013 ◽  
Vol 40 (06) ◽  
pp. 896-897
Author(s):  
V. Pelliccia ◽  
C. Pizzanelli ◽  
S. Pini ◽  
P. Malacarne ◽  
U. Bonuccelli
Keyword(s):  
Pulmonary Edema ◽  
Cardiac Dysfunction ◽  
Epileptic Seizures

Ultrastructural Localization of Phycocyanin in the Acidophilic, Thermophilic Alga, Cyanidium Caldarium

1973 ◽  
Vol 31 ◽  
pp. 462-463
Author(s):  
M. R. Edwards ◽  
J. D. Mainwaring
Keyword(s):  
Yellowstone National Park ◽  
National Park ◽  
Ultrastructural Localization ◽  
Review Of Literature ◽  
Cyanidium Caldarium ◽  
Taxonomic Rank ◽  
Thin Sections ◽  
Standard Methods ◽  
Laboratory Cultures

Although the general ultrastructure of Cyanidium caldarium, an acidophilic, thermophilic alga of questionable taxonomic rank, has been extensively studied (see review of literature in reference 1), some peculiar ultrastructural features of the chloroplast of this alga have not been noted by other investigators.Cells were collected and prepared for thin sections at the Yellowstone National Park and were also grown in laboratory cultures (45-52°C; pH 2-5). Fixation (glutaraldehyde-osmium), dehydration (ethanol), and embedding (Epon 812) were accomplished by standard methods. Replicas of frozenfracture d- etched cells were obtained in a Balzers apparatus. In addition, cells were examined after disruption in a French Press.

Oxygen radicals participate in alloxan-induced pulmonary EDEMA

1990 ◽  
Vol 48 (3) ◽  
pp. 298-299
Author(s):  
Tomoo Kawada ◽  
Michio Arakawa ◽  
Kenjiro Kambara ◽  
Takashi Segawa ◽  
Fumio Ando ◽  
...  
Keyword(s):  
Pulmonary Edema ◽  
Oxygen Radicals ◽  
Bolus Injection ◽  
Physiological Saline ◽  
Baseline Period ◽  
Alveolar Epithelial Cells ◽  
Control Group ◽  
Intervention Period ◽  
Lung Water ◽  
Electron Microscopic

We know that alloxan causes increased-permeability pulmonary edema and that alloxan generates oxygen radicals (H2O2, O2−, ·OH) in blood. Therefore, we hypothesize that alloxan-generated oxygen radicals damage pulmonary capillary endothelial cells, and, possibly, alveolar epithelial cells as well. We examined whether oxygen radical scavengers, such as catalase or dimethylsulfoxide (DMSO), protected against alloxaninduced pulmonary edema.Five dogs in each following group were anesthetized: control group: physiological saline (20ml/kg/h); alloxan group: physiological saline + alloxan (75mg/kg) bolus injection at the beginning of the experiment; catalase group: physiological saline + catalase (150,000u/kg) bolus injection before injection of alloxan; DMSO group: physiological saline + DMSO (0.4mg/kg) bolus injection before alloxan. All dogs had 30-min baseline period and 3-h intervention period. Hemodynamics and circulating substances were measured at the specific points of time. At the end of intervention period, the dogs were killed and had the lungs removed for electron microscopic study and lung water measurement with direct destructive method.

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