The effects of low dose insulin infusions on the renin angiotensin and sympathetic nervous systems in normal man

1991 ◽  
Vol 21 (4) ◽  
pp. 430-435 ◽  
Author(s):  
D. P. ROONEY ◽  
J. D. M. EDGAR ◽  
B. SHERIDAN ◽  
A. B. ATKINSON ◽  
P. M. BELL
2017 ◽  
Vol 35 ◽  
pp. e347-e348
Author(s):  
C. Fania ◽  
F. Saladini ◽  
L. Mos ◽  
A. Bortolazzi ◽  
G. Zanata ◽  
...  

2011 ◽  
Vol 2011 ◽  
pp. 1-17 ◽  
Author(s):  
Mohammed H. Abdulla ◽  
Munavvar A. Sattar ◽  
Edward J. Johns

This paper explores the possible relationships between dietary fructose and altered neurohumoral regulation of renal haemodynamic and excretory function in this model of metabolic syndrome. Fructose consumption induces hyperinsulinemia, hypertriglyceridaemia, insulin resistance, and hypertension. The pathogenesis of fructose-induced hypertension is dubious and involves numerous pathways acting both singly and together. In addition, hyperinsulinemia and hypertension contribute significantly to progressive renal disease in fructose-fed rats. Moreover, increased activity of the renin-angiotensin and sympathetic nervous systems leading to downregulation of receptors may be responsible for the blunted vascular sensitivity to angiotensin II and catecholamines, respectively. Various approaches have been suggested to prevent the development of fructose-induced hypertension and/or metabolic alteration. In this paper, we address the role played by the renin-angiotensin and sympathetic nervous systems in the haemodynamic alterations that occur due to prolonged consumption of fructose.


1998 ◽  
Vol 38 (1) ◽  
pp. 221-228 ◽  
Author(s):  
David Ernest Newby ◽  
Rajiv Jalan ◽  
Satoko Masumori ◽  
Peter Clive Hayes ◽  
Nicholas Antony Boon ◽  
...  

2012 ◽  
Vol 98 (1) ◽  
pp. 57-66 ◽  
Author(s):  
Joelma M. C. Gomide ◽  
Rodrigo C. de Menezes ◽  
Luciano G. Fernandes ◽  
Fernanda C. Silva ◽  
Leonardo M. Cardoso ◽  
...  

2004 ◽  
Vol 22 (Suppl. 2) ◽  
pp. S348
Author(s):  
M. Castellano ◽  
A. Padovani ◽  
M. Giacchè ◽  
A. Pezzini ◽  
F. Rivadossi ◽  
...  

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