Evolutionary consequences of colour perception of Sapajus apella (Primates, Platyrrhini)

The argument at Phaedo 74 B 4‐C 6 that the equal itself is ‘something different from’ sets of physical equals depends on Leibniz's Law: there is a property that perceptible equals have that the equal itself does not have. What I call the ‘epistemic interpretation’ holds that the property is an epistemic one: having appeared unequal. The ‘ontological interpretation’ holds that the property is not epistemic, but simply the property of being unequal (that is: physical equals suffer the compresence of opposites, while the equal itself does not). The most natural reading of the text favours the epistemic interpretation; scholarly support for the ontological interpretation is based on the widely held view that on the epistemic interpretation the argument is manifestly invalid. But this view implicitly relies on an impoverished sense of ‘appearing’ as equivalent to ‘being thought’. Drawing on an analogy with colour perception, I elaborate an experiential sense of ‘appearing’ which makes Plato's argument on the epistemic interpretation philosophically defensible.

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Diagnosis and treatment of endometriosis in a hooded capuchin ( Sapajus apella )

Journal of Medical Primatology ◽

10.1111/jmp.12513 ◽

2021 ◽

Author(s):

Anneke Moresco ◽

Sushan Han ◽

Gwen Jankowski ◽

Abigail Peterson

Keyword(s):

Diagnosis And Treatment ◽

Sapajus Apella ◽

Hooded Capuchin

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Boon and Bane of DNA Double-Strand Breaks

International Journal of Molecular Sciences ◽

10.3390/ijms22105171 ◽

2021 ◽

Vol 22 (10) ◽

pp. 5171

Author(s):

Ingo Schubert

Keyword(s):

Negative Impact ◽

Biological Effects ◽

Double Strand Breaks ◽

Immunoglobulin Gene ◽

Dna Double Strand Breaks ◽

Endogenous Factors ◽

Strand Breaks ◽

Chromatin Elimination ◽

Evolutionary Consequences ◽

Cell Lethality

DNA double-strand breaks (DSBs), interrupting the genetic information, are elicited by various environmental and endogenous factors. They bear the risk of cell lethality and, if mis-repaired, of deleterious mutation. This negative impact is contrasted by several evolutionary achievements for DSB processing that help maintaining stable inheritance (correct repair, meiotic cross-over) and even drive adaptation (immunoglobulin gene recombination), differentiation (chromatin elimination) and speciation by creating new genetic diversity via DSB mis-repair. Targeted DSBs play a role in genome editing for research, breeding and therapy purposes. Here, I survey possible causes, biological effects and evolutionary consequences of DSBs, mainly for students and outsiders.

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