scholarly journals Congestive Cardiac Failure: A Case Report

2021 ◽  
pp. 209-212
Author(s):  
Julie Vanlalsawmi ◽  
Deeplata Mendhe ◽  
Pratibha Wankhede
Keyword(s):  
Heart Failure ◽  
Intensive Care Unit ◽  
Cardiac Failure ◽  
Ischemic Cardiomyopathy ◽  
Hospital Readmissions ◽  
Chief Complaint ◽  
Congestive Cardiac Failure ◽  
Emergency Unit ◽  
Economic Strain ◽  
Case Presentation

Introduction: Congestive Cardiac Failure (CCF) is an anomalous clinical condition involving insufficient heart pumping and filling. Cardiac failure causes the heart to be unable to provide enough blood to meet the tissue's oxygen needs. Heart disease is the most common explanation why older people are admitted to hospitals or are in need of palliation. This puts a huge economic strain on the health care system. The dynamic, progressive nature of heart failure also leads to poor results, with hospital readmissions being the costliest. About half of those patient die within 5 years after diagnosis. Case presentation [1]: A male patient of 62years from Aarvi Naka was admitted to Medicine Intensive Care Unit (MICU), AVBRH on 11th January 2020 with a known case of Ischemic Cardiomyopathy which was diagnosed itself at AVBRH on 22nd October 2018 and a known case of diabetes and hypertension for 10 years. My patient was brought to AVBRH Emergency Unit on 11th January 2020 with a chief complaint of breathlessness for 2 days, sweating over both feet for 5 days and generalized weakness for 2 weeks. He was having difficulty in breathing for about 2 days which eventually become severe on 11th January 2020 evening and was brought immediately to AVBRH and got admitted on the same day. The patient was delirious and vomit two times on admission.

Responses of vasoactive hormones in congestive cardiac failure

1987 ◽  
Vol 65 (8) ◽  
pp. 1706-1711 ◽  
Author(s):  
C. I. Johnston ◽  
L. F. Arnolda ◽  
K. Tsunoda ◽  
P. A. Phillips ◽  
G. P. Hodsman
Keyword(s):  
Heart Failure ◽  
Cardiac Failure ◽  
Atrial Natriuretic Factor ◽  
Peripheral Resistance ◽  
Congestive Cardiac Failure ◽  
Renin Angiotensin ◽  
Natriuretic Factor ◽  
Vasoactive Hormones ◽  
Atrial Natriuretic

Congestive cardiac failure causes activation of various neurohumoral responses that increase total peripheral resistance and promote salt and water retention. These effects increase blood pressure and organ perfusion in the short term, but ultimately cause further cardiac decompensation by increasing ventricular afterload and cardiac work. The role of the renin–angiotensin–aldosterone system and the catecholamines is partially understood, and blockade of these systems as a treatment of heart failure is now established. The role of vasopressin in heart failure is more controversial, but there is now compelling evidence that vasopressin may have important vasoconstrictor actions in addition to its fluid retaining properties. Atrial natriuretic factor is a newly described cardiac hormone released from the atrium. Atrial natriuretic factor causes natriuresis, diuresis, vasodilatation, suppression of thirst, and suppression of both renin and aldosterone. These actions largely counteract the effects of the renin–angiotensin system and vasopressin. Plasma atrial natriuretic factor has been reported to be markedly elevated in human and experimental heart failure, and may act to limit the neurohumoral response to reduced cardiac output. This review summarizes our understanding of the vasoactive hormones and reports experimental evidence supporting a pathophysiological role for vasopressin and atrial natriuretic factor in congestive cardiac failure.

scholarly journals Acute kidney injury with cardiorenal syndrome; experience from a tertiary care renal unit in Pakistan

2020 ◽  
Vol 10 (4) ◽  
pp. e29-e29
Author(s):  
Rubina Naqvi
Keyword(s):  
Myocardial Infarction ◽  
Heart Failure ◽  
Acute Myocardial Infarction ◽  
Acute Kidney Injury ◽  
Infective Endocarditis ◽  
Cardiac Failure ◽  
Kidney Injury ◽  
Congestive Cardiac Failure ◽  
Fluid Replacement ◽  
Partial Recovery

Introduction: Acute kidney injury (AKI) is a commonly recognized clinical problem after many morbid conditions related to heart like congenital heart disease surgery, acute or chronic congestive heart failure, acute myocardial infarction, infective endocarditis or cardiomyopathies. Cardio-renal syndrome (CRS) includes a spectrum of disorders involving both the heart and kidneys simultaneously; here acute or chronic dysfunction in one organ may induce acute or chronic dysfunction in the other. Objectives: To report here, case series of patients with AKI developing in association with CRS. We aim to report different causes of CRS and outcome of patients in this group of patients. Patients and Methods: Subjects for the study reported here comprised a cohort of 34 patients coming to this institution with AKI in association of CRS. AKI was defined according to KDIGO guidelines and CRS based on consensus conference of ADQI in 2012. Type 1or type2 CRS are included in the study. All patients had normal size kidneys on ultrasonography. Results: Thirty-four patients with AKI and CRS were brought to this institute from January 1990 to December 2014; this was contributing 1% to medical causes of total AKI. Among these 25 were males and 9 females; mean age of these patients was 54.06±14.106 years. Causes of CRS were acute myocardial infarction (ST elevated), congestive cardiac failure, infective endocarditis and dilated cardiomyopathy. More than two third of patients were either oliguric or anuric on presentation. Fluid replacement and/or inotropic support required in 79%. Renal replacement therapy in form of hemodialysis was conducted in 64.7% and intermittent peritoneal dialysis in one patient. Complete renal recovery was observed in 19 (56%) patients, while 12 (35%) died during acute phase of illness. CKD-V developed in one patient, 2 patients lost long term follow up, but became dialysis free and renal functions were in improving trends, they were labeled as partial recovery. Secondary insults like hypotension, aggressive diuresis, and volume loss from gastro-intestinal tract or infection were evaluated for any co-relation with outcome but statistically no significant difference was found. Conclusion: CRS can be severe life-threatening condition especially when patients present with circulatory collapse. Diuretics must be used cautiously in patients with congestive cardiac failure. Infective endocarditis with acute right heart failure can lead to CRS.

scholarly journals Infantile hepatic hemangiomas associated with high-output cardiac failure and pulmonary hypertension

2019 ◽  
Vol 19 (1) ◽  
Author(s):  
Xin-tong Zhang ◽  
Wei-dong Ren ◽  
Guang Song ◽  
Yang-jie Xiao ◽  
Fei-fei Sun ◽  
...  
Keyword(s):  
Heart Failure ◽  
Pulmonary Hypertension ◽  
Cardiac Failure ◽  
Ductus Arteriosus ◽  
Hepatic Hemangioma ◽  
Case Presentation ◽  
High Output Heart Failure ◽  
Poor Outcomes ◽  
Congenial Heart Disease ◽  
High Output

Abstract Background Infantile hepatic hemangioma (IHH) is a rare endothelial cell neoplasm, which may be concurrent with severe complications and result in poor outcomes. Moreover, the coexistence of IHH and congenial heart disease is even rarer. Case presentation We present a 10-day-old male born with IHH associated with patent ductus arteriosus (PDA), atrial septal defect (ASD) and pulmonary hypertension. Moreover, we reviewed a series of studies of IHH-associated high-output cardiac failure between 1974 and 2018, and summarized the treatment outcomes. Conclusions Infantile hepatic hemangioma (IHH) has been known to induce high-output heart failure. There is no literature to summarize the severity of its impact on heart, which can lead to a high mortality rate. When IHH is detected by ultrasound, the heart should be evaluated to facilitate treatment. The outcomes of IHH associated with heart failure are good.

570 Age-related trend in hospital readmissions for heart failure: an analysis from regional administrative databases

2006 ◽  
Vol 5 (1) ◽  
pp. 128-129
Author(s):  
G PULIGNANO ◽  
A DILENARDA ◽  
F OLIVA ◽  
G GIGLI ◽  
S LOTTAROLI ◽  
...  
Keyword(s):  
Heart Failure ◽  
Hospital Readmissions ◽  
Administrative Databases ◽  
Age Related

Quantitative Relation Between Myocardial Viability and Improvement in Heart Failure Symptoms After Revascularization in Patients With Ischemic Cardiomyopathy

Circulation ◽  
1995 ◽  
Vol 92 (12) ◽  
pp. 3436-3444 ◽  
Author(s):  
Marcelo F. Di Carli ◽  
Farbod Asgarzadie ◽  
Heinrich R. Schelbert ◽  
Richard C. Brunken ◽  
Hillel Laks ◽  
...  
Keyword(s):  
Heart Failure ◽  
Myocardial Viability ◽  
Ischemic Cardiomyopathy ◽  
Quantitative Relation

scholarly journals Implications of multiple late gadolinium enhancement lesions on the frequency of left ventricular reverse remodeling and prognosis in patients with non‐ischemic cardiomyopathy

2021 ◽  
Vol 23 (1) ◽  
Author(s):  
Shingo Ota ◽  
Makoto Orii ◽  
Tsuyoshi Nishiguchi ◽  
Mao Yokoyama ◽  
Ryoko Matsushita ◽  
...  
Keyword(s):  
Heart Failure ◽  
Late Gadolinium Enhancement ◽  
Ischemic Cardiomyopathy ◽  
Left Ventricular ◽  
Reverse Remodeling ◽  
Cardiac Events ◽  
Gadolinium Enhancement ◽  
Systolic Volume ◽  
Linear Pattern ◽  
Multiple Lesions

Abstract Background Non-ischemic cardiomyopathy (NICM) is a heterogeneous disease, and its prognosis varies. Although late gadolinium enhancement (LGE)-cardiovascular magnetic resonance (CMR) demonstrates a linear pattern in the mid-wall of the septum or multiple LGE lesions in patients with NICM, the therapeutic response and prognosis of multiple LGE lesions have not been elucidated. This study aimed to investigate the frequency of left ventricular (LV) reverse remodeling (LVRR) and prognosis in patients with NICM who have multiple LGE lesions. Methods This single-center retrospective study included 101 consecutive patients with NICM who were divided into 3 groups according to LGE-CMR results: patients without LGE (no LGE group = 48 patients), patients with a typical mid-wall LGE pattern (n = 29 patients), and patients with multiple LGE lesions (n = 24 patients). LVRR was defined as an increase in LV ejection fraction (LVEF) ≥ 10 % and a final value of LVEF > 35 %, which was accompanied by a decrease in LV end-systolic volume ≥ 15 % at 12-month follow-up using echocardiography. The frequency of composite cardiac events, defined as sudden cardiac death (SCD), aborted SCD (non-fatal ventricular fibrillation, sustained ventricular tachycardia, or adequate implantable cardioverter-defibrillator therapies), and heart failure death or hospitalization for worsening heart failure, were summarized and compared between the groups. Results Among the 3 groups, the frequency of LVRR was significantly lower in the multiple lesions group than in the no LGE and mid-wall groups (no LGE vs. mid-wall vs. multiple lesions: 49 % vs. 52 % vs. 19 %, p = 0.03). There were 24 composite cardiac events among the patients: 2 in patients without LGE (hospitalization for worsening heart failure; 2), 7 in patients of the mid-wall group (SCD; 1, aborted SCD; 1 and hospitalization for worsening heart failure; 5), and 15 in patients of the multiple lesions group (SCD; 1, aborted SCD; 8 and hospitalization for worsening heart failure; 6). The multiple LGE lesions was an independent predictor of composite cardiac events (hazard ratio: 11.40 [95 % confidence intervals: 1.49−92.01], p = 0.020). Conclusions Patients with multiple LGE lesions have a higher risk of cardiac events and poorer LVRR. The LGE pattern may be useful for an improved risk stratification in patients with NICM.

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