Iron and chemical control of breathing

Pneumologie ◽  
2010 ◽  
Vol 64 (01) ◽  
Author(s):  
J Antosiewicz ◽  
M Walski ◽  
M Pokorski
Keyword(s):  
Chemical Control ◽  
Control Of Breathing

Chemical Control of Breathing in Early Life

1988 ◽  
Vol 533 (1 The Sudden In) ◽  
pp. 276-288 ◽  
Author(s):  
DAVID J. HENDERSON-SMART ◽  
GARY L. COHEN
Keyword(s):  
Chemical Control ◽  
Early Life ◽  
Control Of Breathing

Abnormal Breathing during Sleep and Chemical Control of Breathing during Wakefulness in Patients with Sleep Apnea Syndrome

1989 ◽  
Vol 139 (1) ◽  
pp. 164-169 ◽  
Author(s):  
Fumio Kunitomo ◽  
Hiroshi Kimura ◽  
Koichiro Tatsumi ◽  
Shinya Okita ◽  
Hirokazu Tojima ◽  
...  
Keyword(s):  
Sleep Apnea ◽  
Chemical Control ◽  
Sleep Apnea Syndrome ◽  
Control Of Breathing ◽  
Abnormal Breathing ◽  
Apnea Syndrome

THE INTERACTION OF CENTRAL AND PERIPHERAL CHEMICAL CONTROL OF BREATHING

1940 ◽  
Vol 130 (1) ◽  
pp. 155-170 ◽  
Author(s):  
Robert Gesell ◽  
Jack Lapides ◽  
Manuel Levin
Keyword(s):  
Chemical Control ◽  
Control Of Breathing

Development of the chemical control of breathing in the Virginia opossum

1972 ◽  
Vol 14 (3) ◽  
pp. 267-277 ◽  
Author(s):  
J.P. Farber ◽  
H.N. Hultgren ◽  
S.M. Tenney
Keyword(s):  
Chemical Control ◽  
Control Of Breathing ◽  
Virginia Opossum

Luft's syndrome: O2 cost of exercise and chemical control of breathing

1975 ◽  
Vol 39 (5) ◽  
pp. 857-859 ◽  
Author(s):  
N. H. Edelman ◽  
T. V. Santiago ◽  
H. L. Conn
Keyword(s):  
Chemical Control ◽  
Ventilatory Response ◽  
Normal Subjects ◽  
Control Of Breathing ◽  
O2 Consumption ◽  
Arterial Blood ◽  
Skeletal Muscle Mitochondria ◽  
Ventilatory Response To Co2

The oxygen cost of exercise and chemical control of breathing were studied in a subject with Luft's syndrome, a disorder in which skeletal muscle mitochondria have a high “resting” O2 consumption which is imcreased only slightly by stimulation with excess phosphate acceptor, but a normal P/O ratio. The O2 consumption was more than three times normal (1.05 1/min) at rest but could be doubled when stimulated by maximal exercise. The O2 cost of exercise was similar to that of normal subjects. At rest, arterial blood PCO2 and ventilatory response to CO2 were normal, while ventilatory response to hypoxia was four times the predicted value. The data 1) confirm, in vivo, the normal respiratory efficiency of skeletal muscles in this disorder; 2) suggest that in vitro estimates of the extent to which mitochondrial respiration can be stimulated may not correlate with in vivo determinations; and 3) suggests that hypermetabolism per se can cause the ventilatory adjustments which are associated with exercise in normal subjects.

scholarly journals Effect of Feeding on the Chemical Control of Breathing in the Newborn Infant

1981 ◽  
Vol 15 (12) ◽  
pp. 1509-1512 ◽  
Author(s):  
M Durand ◽  
F N Leahy ◽  
M Maccallum ◽  
D B Cates ◽  
H Rigatto ◽  
...  
Keyword(s):  
Newborn Infant ◽  
Chemical Control ◽  
Control Of Breathing ◽  
Effect Of Feeding
Sign in / Sign up

Export Citation Format

Share Document