Nesidioblastosis and pancreatic islet cell hyperplasia arising in a mediastinal mature teratoma

2016 ◽  
Author(s):  
Ji-Yun Jeong
Keyword(s):  
Pancreatic Islet ◽  
Islet Cell ◽  
Mature Teratoma ◽  
Pancreatic Islet Cell ◽  
Cell Hyperplasia ◽  
Islet Cell Hyperplasia

Mild chronic watery diarrhea-hypokalemia syndrome associated with pancreatic islet cell hyperplasia

1979 ◽  
Vol 66 (5) ◽  
pp. 883-888 ◽  
Author(s):  
Gerald S. Kidd ◽  
Mark Donowitz ◽  
Thomas O'Dorisio ◽  
Samuel Cataland ◽  
Forest Newman
Keyword(s):  
Pancreatic Islet ◽  
Islet Cell ◽  
Watery Diarrhea ◽  
Pancreatic Islet Cell ◽  
Cell Hyperplasia ◽  
Islet Cell Hyperplasia

[68Ga]-DOTATOC-PET and visualization of pancreatic islet cell hyperplasia / nesidioblastosis

2007 ◽  
Vol 115 (S 1) ◽  
Author(s):  
AAR Starke ◽  
WH Knapp ◽  
R Köster ◽  
L Mansfeld ◽  
PE Goretzki
Keyword(s):  
Pancreatic Islet ◽  
Islet Cell ◽  
Pancreatic Islet Cell ◽  
Cell Hyperplasia ◽  
Islet Cell Hyperplasia

scholarly journals Chronic laxative abuse associated with pancreatic islet cell hyperplasia.

Gut ◽  
1977 ◽  
Vol 18 (12) ◽  
pp. 1032-1035 ◽  
Author(s):  
M Lesna ◽  
A N Hamlyn ◽  
C W Venables ◽  
C O Record
Keyword(s):  
Pancreatic Islet ◽  
Islet Cell ◽  
Pancreatic Islet Cell ◽  
Cell Hyperplasia ◽  
Laxative Abuse ◽  
Islet Cell Hyperplasia

scholarly journals In Vitro Studies of Insulin Inactivation with Reference to Erythroblastosis Fetalis

Blood ◽  
1967 ◽  
Vol 30 (3) ◽  
pp. 359-363 ◽  
Author(s):  
J. STEINKE ◽  
F. A. GRIES ◽  
S. G. DRISCOLL ◽  
Maija Grinbush
Keyword(s):  
Pancreatic Islet ◽  
Islet Cell ◽  
In Vitro Studies ◽  
Pancreatic Islet Cell ◽  
Cell Hyperplasia ◽  
Compensatory Response ◽  
Reversible Binding ◽  
Pancreatic Insulin ◽  
Islet Cell Hyperplasia

Abstract It was demonstrated that hemolysed blood and/or crystalline hemoglobin inactivated insulin either by irreversible destruction due to the presence of SH compounds or by reversible binding to hemoglobin. The increased pancreatic insulin content and pancreatic islet cell hyperplasia observed in infants with erythroblastosis fetalis may represent a compensatory response to inactivation of circulating insulin.

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