Femoral artery occlusion after using a suture-mediated arterial puncture closure device

2008 ◽  
Vol 72 (4) ◽  
pp. 522-524 ◽  
Author(s):  
Sung-Min Park ◽  
Young-Tae Kwak
Keyword(s):  
Femoral Artery ◽  
Artery Occlusion ◽  
Arterial Puncture ◽  
Closure Device ◽  
Femoral Artery Occlusion

Cases of Femoral Artery Occlusion after using the Exoseal Closure Device: Including Experiment in Vascular Model

2015 ◽  
Vol 7 (3) ◽  
pp. 81-85
Author(s):  
Yosuke Takasawa ◽  
Sumio Mizuno ◽  
Junya Yamaguchi ◽  
Masatomo Suzuki ◽  
Masayuki Tsuchida ◽  
...  
Keyword(s):  
Femoral Artery ◽  
Artery Occlusion ◽  
Closure Device ◽  
Vascular Model ◽  
Femoral Artery Occlusion

scholarly journals Femoral artery occlusion with a percutaneous arterial closure device after a routine coronary angiogram: a case report and literature review

2011 ◽  
Vol 93 (6) ◽  
pp. e102-e104 ◽  
Author(s):  
T Hussain ◽  
S Al-Hamali
Keyword(s):  
Femoral Artery ◽  
Common Femoral Artery ◽  
Arterial Disease ◽  
Vessel Diameter ◽  
Artery Occlusion ◽  
Arterial Puncture ◽  
Closure Device ◽  
Coronary Angiogram ◽  
Puncture Site ◽  
Vascular Closure Devices

Groin vessels are most commonly used to obtain vascular access for angiography because of their size and accessibility. Haemostasis at the puncture site can be achieved with manual compression alone or by using a vascular closure device. We highlight the case of a 68-year-old woman who developed acute claudication in the right leg after a routine diagnostic coronary angiogram when an Angio-Seal™ device had been employed to close a relatively low arterial puncture. On exploring the common femoral artery, fragments of the device were found occluding the bifurcation. A patch angioplasty was carried out and the patient’s claudicant symptoms improved. The Angio-Seal™ device has a polylactide and polyglycolide polymer anchor, a collagen plug and a suture contained within a carrier system. Haemostasis is achieved by compressing the arterial puncture site between the anchor and the collagen plug. The manufacturer’s recommended criterion for using the device safely permits its use only for common femoral artery punctures with an internal vessel diameter of 4mm. Anatomical confirmation of the puncture site and evidence of any arterial disease or stenosis in the artery is detected on fluoroscopy during the procedure. Recent meta-analyses have cast doubt on the assumption that vascular closure devices are superior to mechanical compression alone and serious complications do occur occasionally but are under-reported. Clinicians should be aware of the potentially serious problems that may occur when deciding to employ vascular closure devices, especially with an anatomically low puncture site.

scholarly journals Angioscopic Observation of Acute Femoral Artery Occlusion Complicated by Use of Angio-seal Vascular Closure Device

Angioscopy ◽  
2015 ◽  
Vol 1 (1) ◽  
pp. 35-37
Author(s):  
Ryo Munakata ◽  
Masamichi Takano ◽  
Tomohito Hada ◽  
Tetsuro Shimura ◽  
Osamu Kurihara ◽  
...  
Keyword(s):  
Femoral Artery ◽  
Artery Occlusion ◽  
Closure Device ◽  
Vascular Closure Device ◽  
Femoral Artery Occlusion

scholarly journals A Novel Intervention to Treat Failed Angio-Seal Footplate Deployment: Two Case Series

2019 ◽  
Vol 12 ◽  
pp. 117954761982871 ◽  
Author(s):  
Thomas C Hall ◽  
Said Habib
Keyword(s):  
Femoral Artery ◽  
Case Series ◽  
Arterial Occlusion ◽  
Artery Occlusion ◽  
Critical Limb Ischaemia ◽  
Closure Device ◽  
General Anaesthetic ◽  
Limb Ischaemia ◽  
Retrograde Approach ◽  
Femoral Artery Occlusion

Introduction: Vascular closure devices are commonly used to achieve rapid haemostasis and early ambulation following arterial puncture for endovascular procedures. Although device failure rates are low, the consequences of arterial occlusion include severe limb ischaemia. We describe a novel endovascular technique for the treatment of Angio-Seal arterial closure device (Terumo, Europe NV) failure causing femoral artery occlusion. Materials and methods: We describe 2 cases of lower limb angioplasty performed for critical limb ischaemia where the access site was closed using an Angio-Seal according to the manufacturer instructions for use (IFU). In both cases, however, ultrasound could not be used during deployment of the Angio-Seal due to body habitus and small subcutaneous haematoma. In both cases, the device failed and occluded the femoral artery. Results: Access was achieved via a retrograde approach from the contralateral limb in one case and a retrograde approach from the ipsilateral profunda artery in the other case. Angiography confirmed that the footplate of the Angio-Seal had occluded the femoral artery. Subsequently, the occlusion was crossed and a short balloon-mounted bare metal stent placed to push the footplate against the arterial wall that resulted in resolution of the occlusion and haemorrhage control. Conclusions: Crossing the occlusion caused by failure of the Angio-Seal closure device and subsequent stenting resulted in satisfactory relief of the femoral artery occlusion and haemostasis without the added risks of open surgical revascularisation and general anaesthetic.

Acute compensation to abrupt occlusion of rat femoral artery is prevented by NO synthase inhibitors

1994 ◽  
Vol 267 (6) ◽  
pp. H2523-H2530 ◽  
Author(s):  
J. L. Unthank ◽  
J. C. Nixon ◽  
M. C. Dalsing
Keyword(s):  
Femoral Artery ◽  
Arterial Occlusion ◽  
Artery Occlusion ◽  
No Synthase ◽  
Artery Blood Flow ◽  
Relaxing Factor ◽  
Femoral Artery Occlusion ◽  
Before And After ◽  
Endothelium Derived Relaxing Factor ◽  
Oxide Synthase

The hemodynamic significance of endothelium-derived relaxing factor (EDRF)-mediated mechanisms in vascular responses to abrupt rat femoral artery occlusion was investigated. Temporary arterial occlusion was produced before and after inhibition of nitric oxide synthase by N omega-nitro-L-arginine methyl ester (L-NAME) or NG-monomethyl-L-arginine (L-NMMA). Iliac artery blood flow and arterial pressures proximal and distal to the occlusion were measured. Normal vascular compensation included a return of resistance to preocclusion levels and a rise in distal pressure to a plateau within 5 min postocclusion. After treatment with L-NAME and L-NMMA, postocclusion resistance remained elevated by 53 and 36%, respectively. Collateral dilation after occlusion, as indicated by the rise in distal pressure, was prevented by L-NAME but not L-NMMA. Increases in adrenergic tone and mean arterial pressure by phenylephrine did not prevent compensation, suggesting the effects of L-NAME and L-NMMA did not result from elevated sympathetic activation or pressure. The results are consistent with the hypothesis that the stimulated release of endothelium-derived relaxing factor mediates the acute vascular compensation to abrupt arterial occlusion.

Sign in / Sign up

Export Citation Format

Share Document