Hemodynamic Changes in Essential Hypertension and the Hemodynamic Effects of Calcium Antagonists

1986 ◽  
pp. 199-211 ◽  
Author(s):  
P. Lund-Johansen ◽  
P. Omvik
Keyword(s):  
Essential Hypertension ◽  
Calcium Antagonists ◽  
Hemodynamic Effects ◽  
Hemodynamic Changes

Immediate hemodynamic effects of urapidil in patients with essential hypertension

1985 ◽  
Vol 55 (6) ◽  
pp. 722-725 ◽  
Author(s):  
Isaac Kobrin ◽  
Celso Amodeo ◽  
Hector O. Ventura ◽  
Franz H. Messerli ◽  
Edward D. Frohlich
Keyword(s):  
Essential Hypertension ◽  
Hemodynamic Effects

scholarly journals HEMODYNAMIC EFFECTS OF ACUTE AND PROLONGED β-ADRENERGIC BLOCKADE IN ESSENTIAL HYPERTENSION

2009 ◽  
Vol 196 (1-6) ◽  
pp. 27-34 ◽  
Author(s):  
Lennart Hansson ◽  
Andrew J. Zweifler ◽  
Stevo Julius ◽  
Stephen N. Hunyor
Keyword(s):  
Essential Hypertension ◽  
Adrenergic Blockade ◽  
Hemodynamic Effects

IMMEDIATE HEMODYNAMIC EFFECTS OF ISOPROTERENOL

1963 ◽  
Vol 41 (1) ◽  
pp. 1949-1953 ◽  
Author(s):  
Margaret Beznák ◽  
P. Hacker
Keyword(s):  
Blood Pressure ◽  
Cardiac Output ◽  
Oxygen Consumption ◽  
Subcutaneous Injection ◽  
Direct Action ◽  
Peripheral Resistance ◽  
Oxygen Demand ◽  
Hemodynamic Effects ◽  
Hemodynamic Changes ◽  
Histological Damage

Subcutaneous injection of 40 mg/kg isoproterenol is followed within 2 minutes by a fall in blood pressure and peripheral resistance, by tachycardia, and by an increase in cardiac output. It seems likely that these hemodynamic changes are a consequence of a direct action of isoproterenol on the myocardium. Isoproterenol also causes a significant increase in the oxygen consumption of the rats. The increased oxygen demand of the tissues may play a role in maintaining the hemodynamic changes for periods of more than an hour after isoproterenol. Signs of histological damage in the myocardium begin to appear without affecting the function of the cardiovascular system, as measured by the tests used.

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