A case of duodenal ulcer with antral gastrin cell hyperplasia

1993 ◽  
Vol 26 (2) ◽  
pp. 105-110
Author(s):  
Michitoshi Osaka ◽  
Kikuo Aizawa ◽  
Tsutomu Suzuki ◽  
Jun Soga
Keyword(s):  
Duodenal Ulcer ◽  
Cell Hyperplasia ◽  
Gastrin Cell

Gastrin release after bombesin stimulation in Zollinger-Ellison syndrome, in antral gastrin cell hyperplasia and in duodenal ulcer patients

1980 ◽  
Vol 1 ◽  
pp. S116
Author(s):  
P. Tomassetti ◽  
G. Bonora ◽  
L. Bagnoli ◽  
A. Gruppioni ◽  
P. Vezzadini
Keyword(s):  
Duodenal Ulcer ◽  
Gastrin Release ◽  
Cell Hyperplasia ◽  
Gastrin Cell ◽  
Zollinger Ellison Syndrome

ANTRAL-GASTRIN-CELL HYPERPLASIA IN PEPTIC ULCER DISEASE

The Lancet ◽  
1974 ◽  
Vol 303 (7870) ◽  
pp. 1344-1345 ◽  
Author(s):  
Jack Hansky ◽  
P.C Ganguli ◽  
J.B Elder ◽  
J.M Polak ◽  
A.G.E Pearse
Keyword(s):  
Peptic Ulcer ◽  
Peptic Ulcer Disease ◽  
Cell Hyperplasia ◽  
Gastrin Cell ◽  
Ulcer Disease

The Relation between Functioning Parietal Cell and Gastrin Cell Masses in Two Groups of Duodenal Ulcer Patients

1976 ◽  
Vol 50 (5) ◽  
pp. 375-383 ◽  
Author(s):  
D. J. Byrnes ◽  
Shiu Kum Lam ◽  
W. Sircus
Keyword(s):  
Duodenal Ulcer ◽  
Parietal Cell ◽  
Serum Gastrin ◽  
Acid Output ◽  
Ulcer Patient ◽  
Cell Mass ◽  
Inverse Correlation ◽  
Normal Subjects ◽  
Gastrin Cell ◽  
Before And After

1. Serum gastrin concentrations before and after a standardized meal were determined in twenty-eight patients with duodenal ulcer and in ten normal control subjects. 2. In response to pentagastrin, thirteen of the duodenal ulcer subjects secreted acid within the limits of normal and fifteen secreted in excess. 3. The differences in the basal serum gastrin concentrations between the three groups, normal subjects, acid ‘normosecretors’ and hypersecretors were not statistically significant but that of the hypersecretors was suggestively low. 4. The integrated gastrin response and peak gastrin responses to meals were higher in duodenal ulcer patients with normal acid secretion than in the hypersecretors but the values for the latter were not different from normal subjects. 5. Stabilization of intragastric pH by infusion into the antrum of sodium bicarbonate during the test meal response period did not alter these differences between the two ulcer patient groups. 6. A significant inverse correlation exists between the maximal acid output and the integrated gastrin response in both normal subjects and hypersecreting duodenal ulcer patients. 7. The evidence (a) supports the existence of an inverse relationship between the functioning parietal cell and gastrin cell masses, (b) shows the gastrin response in normosecreting ulcer subjects to be inappropriately high, and (c) suggests that excessive vagotonia exerts trophic effects upon both parietal cell mass and gastrin cell mass.

Primary gastrin cell hyperplasia

1984 ◽  
Vol 8 (11) ◽  
pp. 821-832 ◽  
Author(s):  
Klaus J. Lewin ◽  
Kan Yang ◽  
Thomas Ulich ◽  
Janet D. Elashoff ◽  
John Walsh
Keyword(s):  
Cell Hyperplasia ◽  
Gastrin Cell

scholarly journals Adrenal Corticosteroids Cause Gastrin Cell Hyperplasia

1979 ◽  
Vol 76 (5) ◽  
pp. 913-916 ◽  
Author(s):  
John P. Delaney ◽  
Hector M. Michel ◽  
Margaret E. Bonsack ◽  
M. Michael Eisenberg ◽  
Daniel H. Dunn
Keyword(s):  
Cell Hyperplasia ◽  
Gastrin Cell

scholarly journals Antral Gastrin Cell Hyperplasia in Patients with Peptic Ulcer

1980 ◽  
Vol 191 (3) ◽  
pp. 276-281 ◽  
Author(s):  
F. KEUPPENS ◽  
G. WILLEMS ◽  
J. DE GRAEF ◽  
M. C. WOUSSEN-COLLE
Keyword(s):  
Peptic Ulcer ◽  
Cell Hyperplasia ◽  
Gastrin Cell

ANTRAL-GASTRIN-CELL HYPERPLASIA IN PEPTIC-ULCER DISEASE

The Lancet ◽  
1974 ◽  
Vol 303 (7866) ◽  
pp. 1118
Author(s):  
W. Sircus
Keyword(s):  
Peptic Ulcer ◽  
Peptic Ulcer Disease ◽  
Cell Hyperplasia ◽  
Gastrin Cell ◽  
Ulcer Disease

ANTRAL-GASTRIN-CELL HYPERPLASIA IN PEPTIC-ULCER DISEASE

The Lancet ◽  
1974 ◽  
Vol 303 (7863) ◽  
pp. 939-940 ◽  
Author(s):  
Shiu-Kum Lam ◽  
W. Sircus
Keyword(s):  
Peptic Ulcer ◽  
Peptic Ulcer Disease ◽  
Cell Hyperplasia ◽  
Gastrin Cell ◽  
Ulcer Disease
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