The thyrohyoid membrane as a target for ultrasonography-guided block of the internal branch of the superior laryngeal nerve

2015 ◽  
Vol 27 (7) ◽  
pp. 548-552 ◽  
Author(s):  
Tatjana Stopar-Pintaric ◽  
Kamen Vlassakov ◽  
Josip Azman ◽  
Erika Cvetko
Neurosurgery ◽  
2001 ◽  
Vol 49 (4) ◽  
pp. 925-933 ◽  
Author(s):  
Ashkan Monfared ◽  
Daniel Kim ◽  
Sivakumar Jaikumar ◽  
Goutham Gorti ◽  
Andrew Kam

Abstract OBJECTIVE To study the microsurgical anatomy of the superior laryngeal nerve (SLN) and recurrent laryngeal nerve (RLN) with respect to anatomic landmarks, and to identify their vascular supplies. METHODS The microsurgical anatomy of the anterior neck, the course of the right and left SLN and RLN and their variations were studied in 21 cadavers. Fresh cadavers were perfused with colored silicon dye to investigate the microvasculature in detail. RESULTS SLN originates from the inferior vagal ganglion at the C2 level and descends medially toward the thyrohyoid membrane. It branches into an external and an internal branch deep to the internal carotid artery at the C3 level. The external branch, along with the cricothyroid artery, descends deep to the superior thyroid artery toward the cricothyroid muscle. Accompanied by the superior laryngeal artery, the internal branch passes deep to the loop of the superior thyroid artery and pierces the thyrohyoid membrane. Both nerves reside in the fascia covering longus colli muscles and are supplied by their accompanying arteries. The loop of RLN is found at the T1–T3 level on the right, and more caudally at the T3–T6 level on the left, entering the larynx between C5–C7 levels on both sides. RLN receives arterial supply from the esophageal and tracheal branches of the inferior thyroid artery proximally, and by the inferior laryngeal artery distally. CONCLUSION Incidental intraoperative injury to the SLN and RLN potentially could be avoided by understanding the detailed course of each nerve with respect to the surrounding anatomic landmarks and by recognizing their blood supplies.


2021 ◽  
pp. 014556132110291
Author(s):  
George K. Paraskevas ◽  
Alexandros Poutoglidis ◽  
Nikolaos Lazaridis ◽  
Nikolaos Anastasopoulos ◽  
Nikolaos Tsetsos

Internal branch of superior laryngeal nerve (ibSLN) provides sensory innervation mostly to the supraglottic part of the larynx and thus prevents aspiration during ingestion. Normally, it is distributed to the larynx after piercing the thyrohyoid membrane above the superior laryngeal artery. Multiple anatomical variations in the course of ibSLN have been reported. An early ibSLN bifurcation and course through double thyroid foramen constitutes an interesting anatomical variation that may easily lead to an injury during procedures in the thyroid gland and the larynx. Knowledge of the anatomical variability is essential in order to prevent surgical complications that could potentially impact the patient’s quality of life.


2006 ◽  
Vol 15 (9) ◽  
pp. 1320-1325 ◽  
Author(s):  
Amac Kiray ◽  
Sait Naderi ◽  
Ipek Ergur ◽  
Esin Korman

1994 ◽  
Vol 110 (1) ◽  
pp. 122-125
Author(s):  
Paul A. Levine ◽  
Daniel G. Deschler ◽  
Jeffrey A. McKenna ◽  
Thomas A. Tami

1999 ◽  
Vol 12 (2) ◽  
pp. 79-83 ◽  
Author(s):  
Robert E. Stephens ◽  
Karen Haas Wendel ◽  
W. Robert Addington

1975 ◽  
Vol 84 (2) ◽  
pp. 206-212 ◽  
Author(s):  
Masahiro Tanabe ◽  
Kazutomo Kitajima ◽  
Wilbur J. Gould

The laryngeal phonatory reflex through the internal branch of the superior laryngeal nerve (SLN) was investigated by means of anesthetization of the nerve, after which acoustic signals were subjected to computer analysis to determine how anesthesia affected basic vocal parameters. Results showed that the anesthetization did not affect the abrupt cycle-to-cycle frequency changes and also did not influence the gross control of the fundamental frequency. But slower fluctuation of the fundamental frequency increased following anesthesia. From these results, it is suggested that the anesthetization of the internal branch of the SLN may derange the fine control mechanism of the larynx without affecting overall or gross performance of the phonatory apparatus.


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