OBLITERATION OF SPINAL-CORD ARTERIOVENOUS MALFORMATION BY PERCUTANEOUS EMBOLISATION

The Lancet ◽  
1968 ◽  
Vol 291 (7540) ◽  
pp. 477 ◽  
Author(s):  
JohnL Doppman ◽  
GiovanniDi Chiro ◽  
Ayub Ommaya
Neurosurgery ◽  
1983 ◽  
Vol 12 (6) ◽  
pp. 691-693 ◽  
Author(s):  
Don S. Lee ◽  
Arthur Kobrine

Abstract A case of neurogenic pulmonary edema (NPE) associated with a ruptured spinal cord arteriovenous malformation (AVM) is presented. The mechanisms involved in the development of NPE are discussed briefly. The possible role of preganglionic sympathetic fibers in the spinal cord in the etiology of NPE is suggested.


1986 ◽  
Vol 64 (2) ◽  
pp. 322-324 ◽  
Author(s):  
T. S. Park ◽  
Wayne S. Cail ◽  
Johnny B. Delashaw ◽  
John Kattwinkel

✓ A 2-day-old neonate with a spinal cord arteriovenous malformation developed severe paraparesis. The abrupt neurological deterioration was not associated with hemorrhage or aneurysmal dilatation. Ischemic damage of the spinal cord is suggested as the cause of the clinical manifestation.


2009 ◽  
Vol 10 (5) ◽  
pp. 436-442 ◽  
Author(s):  
Kittipong Srivatanakul ◽  
Dittapong Songsaeng ◽  
Augustin Ozanne ◽  
Frédérique Toulgoat ◽  
Pierre Lasjaunias

The authors describe 4 cases of syringomyelia-associated spinal cord arteriovenous malformation (AVM). All cases were managed with embolization of the AVM. Treatments were aimed to stabilize the AVM itself and not directed toward the syrinx. In 3 of the 4 cases the syringomyelia resolved after treatment. Reports concerning AVM as a cause of syringomyelia is very scarce and lacks posttreatment clinical information. In light of the clinical course and imaging findings, the authors propose a theory that venous hypertension in the spinal cord is the trigger for the development of syringomyelia, which may reverse after AVM treatment.


1995 ◽  
Vol 34 (5) ◽  
pp. 281-283 ◽  
Author(s):  
Robert S. B. Clark ◽  
Richard A. Orr ◽  
C. Scott Atkinson ◽  
Richard B. Towbin ◽  
Dachling Pang

2008 ◽  
Vol 14 (2) ◽  
pp. 185-190 ◽  
Author(s):  
Y. Iizuka ◽  
M. Suzuki ◽  
S. Komura ◽  
T. Takada ◽  
K. Shimoji

We describe a 24-year-old woman who presented with twice previously unverified subarachnoid hemorrhages from the conus medullaris spinal arteriovenous malformation with Parkes-Weber-syndrome. Spinal MRI examination is considered to be necessary for the diagnosis of Klippel-Trenaunay-Weber syndrome. For diagnosis of the spinal cord arteriovenous malformation, it is indispensable to search carefully for the presence of accompanying lesions. Transarterial glue embolization is effective for the management of the spinal vascular lesion.


Sign in / Sign up

Export Citation Format

Share Document