Surgical technique of double switch procedure: Senning with arterial switch operation for congenitally corrected transposition of the great arteries with ventricular septal defect

2016 ◽  
Vol 2016 ◽  
pp. mmw007
Author(s):  
Alexey S. Ilin ◽  
Pavel V. Teplov ◽  
Valeriy A. Sakovich ◽  
Richard G. Ohye
2004 ◽  
Vol 14 (6) ◽  
pp. 661-663 ◽  
Author(s):  
Nobuyuki Ishibashi ◽  
Mitsuru Aoki ◽  
Tadashi Fujiwara

We performed a combined Senning and arterial switch operation on a 2-month-old patient with congenitally corrected transposition, Ebstein's malformation producing severe tricuspid regurgitation, ventricular septal defect, pulmonary hypertension, and congestive heart failure. The tricuspid regurgitation was improved. The double switch operation has the advantage of improving the function of the systemic atrioventricular valve, especially in newborns or young infants in whom the outcome of the valvar repair is poor.


1999 ◽  
Vol 9 (3) ◽  
pp. 319-322 ◽  
Author(s):  
Nikolaos Nikoloudakis ◽  
Angelika Lindinger ◽  
Hans-Joachim Schäfers

AbstractAn infant is described with congenitally corrected transposition and Ebstein's malformation. Banding of the pulmonary trunk had been previously performed because of a muscular ventricular septal defect. The patient underwent the double-switch procedure with the intention of unloading the morphologically right ventricle and the malformed tricuspid valve. This resulted in prompt postoperative functional and haemodynamic improvement.


1995 ◽  
Vol 3 (3-4) ◽  
pp. 103-108
Author(s):  
KG Jaya Prasanna ◽  
Krishna Subramony Iyer ◽  
Rajesh Sharma ◽  
Balram Airan ◽  
Ivatury Mrityonjaya Rao ◽  
...  

From January 1991' to May 1994, 29 patients with double outlet right ventricle with ventricular septal defect, without pulmonary stenosis underwent primary intracardiac repair at the All India Institute of Medical Sciences, New Delhi. Patients were classified into 4 groups based on location of the ventricular septal defect. The ventricular septal defect was subaortic in 11, subpulmonary in 13, doubly committed subarterial in 1, and noncommitted in 4 patients. Surgical treatment consisted of intraventricular routing of the left ventricle to the aorta (17), and the left ventricle to the pulmonary artery followed by an arterial switch operation (12). There were 4 (13.9%) early deaths. Follow-up ranged from 3 months to 3 years (mean, 1.5 years). There was no late mortality. Three patients had residual ventricular septal defect, one of whom has undergone reoperation. One patient has a gradient of 25 mmHg across the left ventricular outflow tract. Double outlet right ventricle with subpulmonic ventricular septal defect was found to be a significant risk factor for early mortality (p = 0.03). The subgroup of double outlet right ventricle with subpulmonic ventricular septal defect who had a combination of single coronary artery and post arterial switch operation was particularly prone to pulmonary hypertensive crisis and hospital death (p = 0.002).


1997 ◽  
Vol 64 (2) ◽  
pp. 495-502 ◽  
Author(s):  
Tom R. Karl ◽  
Robert G. Weintraub ◽  
Christian P. Brizard ◽  
Andrew D. Cochrane ◽  
Roger B.B. Mee

2011 ◽  
Vol 21 (4) ◽  
pp. 383-391
Author(s):  
J. Gabriel ◽  
H.-H. Scheld ◽  
T.D.T. Tjan ◽  
N. Osada ◽  
Thomas Krasemann

AbstractA ventricular septal defect in transposition of the great arteries is frequently closely related to the cardiac valves. The valvar function after arterial switch operation of patients with transposition of the great arteries and ventricular septal defect or intact ventricular septum was compared. We analysed the function of all cardiac valves in patients who underwent the arterial switch operations pre- and post-operatively, 1 year after the procedure and on follow-up. The study included 92 patients – 64 with transposition of the great arteries/intact ventricular septum and 28 with transposition of the great arteries/ventricular septal defect. The median age at surgery was 5.5 days in transposition of the great arteries/intact ventricular septum (0–73 days) and 7.0 days in transposition of the great arteries/ventricular septal defect (4–41 days). Follow-up was 51.7 months in transposition of the great arteries/intact ventricular septum (3.3–177.3 months) and 55 months in transposition of the great arteries/ventricular septal defect (14.6–164.7 months). Neo-aortic, neo-pulmonary, and mitral valvar function did not differ. Tricuspid regurgitation was more frequent 1 year post-operatively in transposition of the great arteries/ventricular septal defect (n = 4) than in transposition of the great arteries/intact ventricular septum. The prevalence of neo-aortic regurgitation and pulmonary stenosis increased over time, especially in patients with transposition of the great arteries/intact ventricular septum. The presence of a ventricular septal defect in patients undergoing arterial switch operation for transposition of the great arteries only has a minor bearing for the development of valvar dysfunction on the longer follow-up.


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