scholarly journals Pulmonary artery banding in isolated or complicated ventricular septal defects. Results and effects on growth.

Heart ◽  
1972 ◽  
Vol 34 (1) ◽  
pp. 87-94 ◽  
Author(s):  
S Menahem ◽  
A W Venables
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Artery Banding ◽  
Ventricular Septal Defects ◽  
Septal Defects

Pulmonary Artery Banding in Infants with Cardiac Anomalies Other Than Ventricular Septal Defects: Including an Evaluation of a New Technic for Determining a Critical Degree of Banding

1965 ◽  
Vol 47 (1) ◽  
pp. 88-97 ◽  
Author(s):  
GLEN G. CAYLER ◽  
G. RAINEY WILLIAMS ◽  
EDWARD A. SMELOFF ◽  
ROBERT S. CARTWRIGHT ◽  
NICHOLAS M. TASSOPOULOS ◽  
...  
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Artery Banding ◽  
Ventricular Septal Defects ◽  
Critical Degree ◽  
Cardiac Anomalies ◽  
Septal Defects

scholarly journals Diminution and Closure of Large Ventricular Septal Defects After Pulmonary Artery Banding

Circulation ◽  
1973 ◽  
Vol 48 (4) ◽  
pp. 847-855 ◽  
Author(s):  
ZOLTAN G. MESKO ◽  
JIMMY E. JONES ◽  
ALEXANDER S. NADAS
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Artery Banding ◽  
Ventricular Septal Defects ◽  
Septal Defects

scholarly journals Hyperoxic Vasoconstriction of Human Pulmonary Arteries: A Novel Insight into Acute Ventricular Septal Defects

2013 ◽  
Vol 2013 ◽  
pp. 1-4 ◽  
Author(s):  
Priyadharshanan Ariyaratnam ◽  
Mahmoud Loubani ◽  
Robert Bennett ◽  
Steven Griffin ◽  
Mubarak A. Chaudhry ◽  
...  
Keyword(s):  
Pulmonary Artery ◽  
Calcium Channels ◽  
Pulmonary Arteries ◽  
Bronchial Carcinoma ◽  
Isometric Tension ◽  
Ventricular Septal Defects ◽  
High Oxygen Content ◽  
Voltage Gated ◽  
Septal Defects ◽  
Voltage Gated Calcium Channels

Objectives. Acute rises in pulmonary artery pressures following postinfarction ventricular septal defects present a challenge. We hypothesised that the abnormally high oxygen content exposure to the pulmonary arteries may be a factor. We investigated the contractile responses of human pulmonary arteries to changes in oxygen tension. Methods. Isometric tension was measured in large and medium sized pulmonary artery rings obtained from lung resections for patients with bronchial carcinoma (n=30). Fresh rings were mounted in organ baths bubbled under basal conditions with hyperoxic or normoxic gas mixes and the gas tensions varied during the experiment. We studied whether voltage-gated calcium channels and nitric oxide signalling had any role in responses to oxygen changes. Results. Hypoxia caused a net mean relaxation of 18.1% ± 15.5 (P<0.005) from hyperoxia. Subsequent hyperoxia caused a contraction of 19.2% ± 13.5 (P<0.005). Arteries maintained in normoxia responded to hyperoxia with a mean constriction of 14.8% ± 3.9 (P<0.005). Nifedipine inhibited the vasoconstrictive response (P<0.05) whilst L-NAME had no effect on any hypoxic vasodilatory response. Conclusions. We demonstrate that hyperoxia leads to vasoconstriction in human pulmonary arteries. The mechanism appears to be dependent on voltage-gated calcium channels. Hyperoxic vasoconstriction may contribute to acute rises in pulmonary artery pressures.

Staged Surgical Management of Anatomically Corrected Malposition of Great Arteries

2020 ◽  
Vol 11 (3) ◽  
pp. 352-354
Author(s):  
Fabian A. Kari ◽  
Saira Siddiqui ◽  
Kanwal M. Farooqi ◽  
Michael P. DiLorenzo ◽  
Emile A. Bacha
Keyword(s):  
Pulmonary Artery ◽  
Surgical Management ◽  
Left Ventricular Outflow Tract ◽  
Ventricular Outflow Tract ◽  
Left Ventricular ◽  
Ventricular Septal Defects ◽  
Septal Defects ◽  
Left Ventricular Outflow ◽  
Anatomically Corrected Malposition ◽  
Atrioventricular Discordance

A nine-month-old girl diagnosed with anatomically corrected malposition (atrioventricular discordance, ventriculoarterial concordance, and malposed great arteries) complicated by multiple ventricular septal defects (VSD) and multifactorial left ventricular outflow tract obstruction (LVOTO) presented for management after pulmonary artery banding. She underwent interim palliation in the form of bilateral cavopulmonary connections, a modified Damus-Kaye-Stansel-type anastomosis, and subsequent staged one-and-a-half ventricle repair (1.5 repair) at the age of three years in the form of VSD closure, hemi-Mustard, and LVOTO resection.

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