scholarly journals 517 In vitro study of blood flow through a modeled aortic valve and in a modeled Valsalva sinus

2005 ◽  
Vol 2004.17 (0) ◽  
pp. 209-210
Author(s):  
Shinichi IIDA ◽  
Kenkichi OHBA ◽  
Tsutomu TAJIKAWA ◽  
Masataka YOSHIDA ◽  
Fuyo TSUKIYAMA
2004 ◽  
Vol 2004.5 (0) ◽  
pp. 185-186
Author(s):  
Shinnichi IIDA ◽  
Kenkichi OHBA ◽  
Tsutomu TAJIKAWA ◽  
Masataka YOSHIDA ◽  
Fuyo TSUKIYAMA

2004 ◽  
Vol 2004.79 (0) ◽  
pp. _9-7_-_9-8_
Author(s):  
Shinnichi IIDA ◽  
Kenkichi OHBA ◽  
Tsutomu TAJIKAWA ◽  
Masataka YOSHIDA ◽  
Fuyo TSUKIYAMA

2004 ◽  
Vol 24 (Supplement1) ◽  
pp. 139-140
Author(s):  
Shinnichi IIDA ◽  
Kenkichi OHBA ◽  
Tsutomu TAJIKAWA ◽  
Masataka YOSHIDA ◽  
Fuyo TSUKIYAMA

Author(s):  
Neelakantan Saikrishnan ◽  
Nicole C. Milligan ◽  
Ajit P. Yoganathan

In about 1–2% of all live births, the human aortic valve only consists of two anomalous leaflets and is known as the bicuspid aortic valve (BAV). BAVs are the most common congenital cardiac anomaly, and are associated with significant valvular dysfunction, including calcific aortic stenosis (AS) and aortic regurgitation (AR), as well as aortic wall abnormalities including coarctation of the aorta, ascending aortic dilatation and aneurysms [1]. Many studies have proposed a common underlying genetic defect in progression of complications with BAVs [2]. However, other recent studies have also suggested that the altered hemodynamic environment associated with BAVs could also be responsible for accelerated disease progression in these patients [3, 4]. A recent in vitro study showed elevated levels of turbulence associated with BAVs, and indicated that fluid flow patterns in the aortic sinuses are also affected due to the altered valve morphology [5]. The present work seeks to compare the levels of turbulence in BAVs to pure trileaflet aortic stenosis models.


2017 ◽  
Vol 10 (3) ◽  
pp. S66
Author(s):  
Matheus Simonato ◽  
Ali Azadani ◽  
Michael Reardon ◽  
Gabriel Aldea ◽  
Georg Nickenig ◽  
...  

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