scholarly journals Atypical Anterior Spinal Artery Infarction due to Left Vertebral Artery Occlusion Presenting with Bilateral Hand Weakness

2014 ◽  
Vol 10 (2) ◽  
pp. 171 ◽  
Author(s):  
Min-Ji Kim ◽  
Mi-Hee Jang ◽  
Mi-Song Choi ◽  
Suk Yun Kang ◽  
Joo Yong Kim ◽  
...  
2019 ◽  
Vol 6 ◽  
pp. 2329048X1986780
Author(s):  
Abdulla Alawadhi ◽  
Christine Saint-Martin ◽  
Christine Sabapathy ◽  
Guillaume Sebire ◽  
Michael Shevell

Lateral medullary syndrome is rare in pediatrics. It is characterized by neurological deficits due to an ischemic lesion in the lateral medulla. The authors describe a 17-year-old boy who developed lateral medullary syndrome in the context of a hyperflexion neck injury while diving in shallow water with traumatic vascular injury. He had “crossed” neurological deficits above and below the neck. His magnetic resonance angiography showed intra- and extracranial left vertebral artery occlusion and his magnetic resonance imaging showed signal abnormality involving the left lateral medulla and inferomedial cerebellum in keeping with an infarct secondary to left vertebral artery and left posterior inferior cerebellar artery occlusion. Good neurological recovery was observed on heparin therapy started after surgical treatment of traumatic injury. To our knowledge, this is the first reported case of lateral medullary syndrome in a pediatric population related to a flexion neck injury. The authors emphasize the importance of a high level of suspicion for accurate diagnosis.


2021 ◽  
Vol 8 (1) ◽  
pp. 331-334
Author(s):  
Nobuhiko KAWAI ◽  
Masaki TATANO ◽  
Ryoji IMOTO ◽  
Koji HIRASHITA ◽  
Masatoshi YUNOKI ◽  
...  

Author(s):  
A Alawadhi ◽  
C Saint-Martin ◽  
G Sebire ◽  
M Shevell

Background: Wallenberg’s syndrome (WS), or lateral medullary syndrome is rare in pediatrics, but is not uncommon in adults. It is characterized by neurological deficits due to an ischemic lesion in the lateral medulla. Methods: Case report Results: We describe a 17-year-old boy who developed WS in the context of hyperflexion injury to the neck while diving in shallow water with vertebral dissection as a presumed etiology. He had ‘crossed’ neurological deficits above and below the neck. His MRA showed intra and extracranial left vertebral artery occlusion and his MRI showed T2W/FLAIR signal abnormality involving the left lateral medulla and inferomedial aspect of the cerebellum in keeping with infarcts secondary to the left vertebral artery thrombosis and occlusion of the left posterior inferior cerebellar artery. He was started on anti-coagulation after spinal surgery. On discharge, he had persistent dysphagia which prompt a gastrostomy tube placement prior to transfer to a rehabilitation center. Conclusions: Our case demonstrates that WS can occur post flexion injury in the pediatric population. The presence of crossed neurological findings above and below the neck in the context of neck injury is an important diagnostic clue that should prompt imaging study focusing on the brain stem and the posterior fossa vascular structures.


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