Giant-cell Arteritis Producing an Aortic Arch Syndrome

1967 ◽  
Vol 66 (3) ◽  
pp. 578 ◽  
Author(s):  
GENE G. HUNDER
Keyword(s):  
Giant Cell Arteritis ◽  
Aortic Arch ◽  
Giant Cell ◽  
Aortic Arch Syndrome

Polymyalgia Rheumatica and Systemic Giant Cell Arteritis. Bioptic Findings of the Subclavian Arteries in a Case of Aortic Arch Syndrome

Angiology ◽  
1984 ◽  
Vol 35 (8) ◽  
pp. 528-533 ◽  
Author(s):  
V. Di Giacomo ◽  
A. Fraioli ◽  
G. Carmenini ◽  
M. Schietroma ◽  
F. Meloni ◽  
...  
Keyword(s):  
Giant Cell Arteritis ◽  
Aortic Arch ◽  
Giant Cell ◽  
Polymyalgia Rheumatica ◽  
Aortic Arch Syndrome

MRI and FDG-PET in the assessment of inflammatory aortic arch syndrome in complicated courses of giant cell arteritis

2008 ◽  
Vol 67 (7) ◽  
pp. 1030-1033 ◽  
Author(s):  
M Both ◽  
K Ahmadi-Simab ◽  
M Reuter ◽  
O Dourvos ◽  
E Fritzer ◽  
...  
Keyword(s):  
Giant Cell Arteritis ◽  
Aortic Arch ◽  
Giant Cell ◽  
Fdg Pet ◽  
Aortic Arch Syndrome

scholarly journals Thoracic endovascular repair of a rare case of leaking aortic arch intramural hematoma secondary to Giant cell arteritis

2019 ◽  
Vol 2 (1) ◽  
Author(s):  
Abhijit Salaskar ◽  
Farzad Najam ◽  
Elizabeth Pocock ◽  
Shawn Sarin
Keyword(s):  
Giant Cell Arteritis ◽  
Aortic Arch ◽  
Giant Cell ◽  
Endovascular Repair ◽  
Intramural Hematoma ◽  
Aortic Aneurysms ◽  
Active Extravasation ◽  
History Of ◽  
One Year ◽  
Aortic Intramural Hematoma

Abstract Background Traditionally thoracic aortic aneurysms (TAA) secondary to Giant Cell Arteritis (GCA) were treated with resection and open repair. However no prior studies have reported an aortic intramural hematoma (IMH) as a presentation of GCA or outcome of thoracic endovascular aortic repair (TEVAR) in TAA or IMH secondary to GCA. Case presentation A 59 year old female, nonsmoker, non-hypertensive, non-diabetic with a known history of GCA, temporal arteritis on prednisone presented with shortness of breath & chest pain. Chest CT revealed aortic arch IMH and large left hemothorax. CTA confirmed distal aortic arch focal dilation, a focal intimal irregularity in the distal aortic arch and extensive IMH without any active extravasation or signs of aortitis. Patient underwent an urgent TEVAR without oversizing the aortic landing zones. Post TEVAR aortogram showed exclusion of the site of IMH origin and dilated aortic arch segment by the stent and absence of active extravasation. One month post-TEVAR CTA showed patent stent graft with resolution of IMH and hemothorax. One year after TEVAR, patient remained asymptomatic. Conclusion GCA can present as an IMH secondary to underlying chronic vasculitis. When endovascular repair is considered, great care should be taken not to grossly oversize aortic landing zones.

scholarly journals FRI0193 ULTRASONOGRAPHY IN THE DIAGNOSIS OF GIANT CELL ARTERITIS

2020 ◽  
Vol 79 (Suppl 1) ◽  
pp. 680.1-680
Author(s):  
R. Jese ◽  
Z. Rotar ◽  
M. Tomsic ◽  
A. Hocevar
Keyword(s):  
Giant Cell Arteritis ◽  
Aortic Arch ◽  
Giant Cell ◽  
Control Group ◽  
Occipital Artery ◽  
Positive Finding ◽  
Temporal Arteries ◽  
Linear Probe ◽  
Aortic Arch Arteries ◽  
Vessel Involvement

Background:Objectives:To evaluate the frequency of cranial and aortic arch artery involvement in GCA using color Doppler ultrasonography (CDS).Methods:We performed CDS of cranial and aortic arch arteries in 248 incipient, clinically diagnosed, GCA patients (64.9% females, median (IQR) age 75 (67-80) years) between October 2013 and September 2019, using a Philips IU22 with 5–17.5 MHz linear probe or Philips Epiq 7 with 5–18.5 MHz linear probe. Temporal, facial, occipital, carotid, vertebral, subclavian, and axillary arteries were examined bilaterally. A halo with positive compression sign was considered a positive finding. Additionally, the thickness of intima-media complex (IMT) of individual vessel was measured, and compared to the IMT of 97 consecutive suspected GCA cases (60.8% females median (IQR) age 74 (65-81) years), in whom GCA was excluded, that served as a control group.Results:The CDS was positive in 244 (98.4%) patients in at least one of the examined arteries. Temporal arteries were most commonly affected, and were involved in 192 (77.4%) patients, followed by facial and occipital arteries, involved in 122 (49.2) and 72 (29.0%) patients, respectively. Extracranial large vessel involvement (LVV) was found in 87 (35.1%) patients (32 patients had isolated LVV, and 55 concomitant cranial and LVV artery involvement). Among the 161 patients without LVV, 12 (4.8% of the studied cohort) had involvement of cranial arteries other than temporal arteries (we found facial and occipital artery involvement in 11 and 3 patients, respectively). Table 1 shows the frequency of individual vessel involvement in GCA, and the IMT of CDS inflamed and non-inflamed arteries in GCA, and in controls.Table 1.The involvement of cranial and aortic arch arteries in GCA assessed by CDS and intima-media thickness of inflamed and non-inflamed arteries in GCA, and controlsArteryGCA No (%)IMT (mm) in GCAIMT (mm) in ControlsPositive CDSPositive CDS*; minimalNegative CDSNegative CDS; maximalTemporal192 (77.4)0.71±0.19; 0.330.25±0.070.23±0.05; 0.46Facial122 (49.2)0.75±0.27; 0.410.29±0.070.26±0.07; 0.47Occipital72 (29.0)0.73±0.33; 0.450.26±0.060.23±0.05; 0.46Carotid34 (13.7)1.53±0.44; 0.880.78±0.180.72±0.15; 1.09Vertebral25 (10.1)1.33±0.47; 0.740.45±0.100.42±0.08; 0.63Subclavian67 (27.0)1.65±0.45; 0.910.70±0.140.70±0.13; 0.99Axillary59 (23.8)1.74±0.65; 1.000.61±0.170.57±0.13; 0.97Any artery244 (98.4)---Legend: GCA giant cell arteritis; IMT thickness of intima-media complex; * mean±SD;Conclusion:CDS of seven preselected cranial and aortic arch arteries provides a high diagnostic yield in GCA.Disclosure of Interests:Rok Jese: None declared, Ziga Rotar Consultant of: Speaker and consulting fees from Abbvie, Amgen, Biogen, Eli Lilly, Medis, MSD, Novartis, Pfizer, Roche, Sanofi., Speakers bureau: Speaker and consulting fees from Abbvie, Amgen, Biogen, Eli Lilly, Medis, MSD, Novartis, Pfizer, Roche, Sanofi., Matija Tomsic: None declared, ALOJZIJA HOCEVAR: None declared

Sign in / Sign up

Export Citation Format

Share Document