Hemifacial spasm associated with intraparenchymal brain stem tumor

2011 ◽  
Vol 26 (13) ◽  
pp. 2325-2326 ◽  
Author(s):  
Athanassios Leonardos ◽  
Paul E. Greene ◽  
Louis H. Weimer ◽  
Alexander G. Khandji ◽  
Pietro Mazzoni

Cancer ◽  
2000 ◽  
Vol 89 (7) ◽  
pp. 1569-1576 ◽  
Author(s):  
Paul G. Fisher ◽  
Steven N. Breiter ◽  
Benjamin S. Carson ◽  
Moody D. Wharam ◽  
Jeffery A. Williams ◽  
...  


1976 ◽  
Vol 14 (2) ◽  
pp. 108-118 ◽  
Author(s):  
S. Negri ◽  
T. Caraceni ◽  
L. de Lorenzi
Keyword(s):  


1994 ◽  
Vol 8 (3) ◽  
Author(s):  
Kenji Kodama ◽  
Takato Morioka ◽  
Takrao Machi ◽  
Shosuke Takahashi


Neurology ◽  
1963 ◽  
Vol 13 (7) ◽  
pp. 607-607 ◽  
Author(s):  
R. L. Sogg ◽  
W. F. Hoyt ◽  
E. Boldrey
Keyword(s):  


1990 ◽  
Vol 14 (4) ◽  
pp. 662-664 ◽  
Author(s):  
Robert Sigal ◽  
François dʼAnthouard ◽  
Philippe David ◽  
Philippe Halimi ◽  
Michel Zerah ◽  
...  


1997 ◽  
Vol 37 (7) ◽  
pp. 525-532 ◽  
Author(s):  
Masafumi FUKUDA ◽  
Shigeki KAMEYAMA ◽  
Yoshiho HONDA ◽  
Hidetoshi YAMAZAKI ◽  
Tadashi KAWAGUCHI ◽  
...  


Neurosurgery ◽  
1983 ◽  
Vol 13 (2) ◽  
pp. 141-146 ◽  
Author(s):  
John D. Loeser ◽  
James Chen

Abstract The literature on hemifacial spasm and its surgical therapy is reviewed, and the authors' experiences with 20 patients are described. Vascular cross compression of the facial nerve adjacent to the brain stem is seen in 90% of the surgical patients. Mobilizing the offending vessel will cure or significantly improve approximately 80% of the patients. Complications occur in 25% of the patients and usually involve decreased hearing or facial weakness. Mortality is virtually zero, and this operation is vastly superior to any other medical or surgical therapy for hemifacial spasm. The pathophysiology is not yet understood.



1982 ◽  
Vol 92 (8) ◽  
pp. 848???852
Author(s):  
MARGARETA B. M??LLER ◽  
AAGE R. M??LLER ◽  
PETER J. JANNETTA


2021 ◽  
Author(s):  
Ehab El Refaee ◽  
Steffen Fleck ◽  
Marc Matthes ◽  
Sascha Marx ◽  
Joerg Baldauf ◽  
...  

Abstract BACKGROUND Microvascular decompression (MVD) is the most effective treatment option for hemifacial spasm (HFS). However, deeply located forms of compression would require proper identification to allow for adequate decompression. OBJECTIVE To describe the usefulness of endoscopic visualization in one of the most challenging compression patterns in HFS, where the posterior inferior cerebellar artery (PICA) loop is severely indenting the brain stem at the proximal root exit zone of facial nerve along the pontomedullary sulcus. METHODS Radiological and operative data were checked for all patients in whom severe indentation of the brainstem by PICA at pontomedullary sulcus was recorded and endoscope-assisted MVD was performed. Clinical correlation and outcome were analyzed. RESULTS A total of 58 patients with HFS were identified with radiological and surgical evidence proving brainstem indentation at the VII transitional zone. In 31 patients, PICA was the offending vessel to the facial nerve. In 3 patients, the PICA loop was mobilized under visualization of a 45° endoscope. A total of 31 patients had a mean follow-up duration of 52.1 mo. The mean duration between start of complaints and surgery was 7.2 yr. In the last follow-up, all patients had remarkable spasm improvement. A total of 5 patients had more than 90% disappearance of spasms and 26 patients experienced spasm-free outcome. CONCLUSION Although severe indentation of brain stem implies morphological damage, outcome after MVD is excellent. A 45° endoscope is extremely helpful to identify compression down at the pontomedullary sulcus. Deeply located compression site can easily be missed with microscopic inspection alone.



Neurosurgery ◽  
1985 ◽  
Vol 16 (6) ◽  
pp. 839-842 ◽  
Author(s):  
Toshio Matsushima ◽  
Masashi Fukui ◽  
Masayuki Matsunaga ◽  
Katsutoshi Kitamura ◽  
Kanehiro Hasuo

Abstract A case of accessory nerve neurinoma presenting with a so-called “intraaxial mass sign” on the angiograms is reported. The tumor originated from the intracranial portion of the accessory nerve and displaced the lateral medullary segment of the posterior inferior cerebellar artery posterolaterally. The displacement of the artery can be understood by the microanatomical relations.



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