The effect of isradipine in acute altitude hypoxia on cerebral, renal, and splanchnic blood flow evaluated by Doppler measurements

2011 ◽  
Vol 4 (04) ◽  
pp. 212-217 ◽  
Author(s):  
Michel Court-Payen ◽  
Aram Ter Minassian ◽  
Niels Olsen ◽  
Inge Kanstrup ◽  
Claude Dubray ◽  
...  
Keyword(s):  
Blood Flow ◽  
Splanchnic Blood Flow ◽  
Altitude Hypoxia

SPLANCHNIC BLOOD FLOW AND OXYGEN DELIVERY ARE DECREASED IN ALCOHOL (EtOH) AND BURN INJURY.

Shock ◽  
2004 ◽  
Vol 21 ◽  
pp. 3-4
Author(s):  
M. A. Choudhry ◽  
Z. F. Ba ◽  
S. Rana ◽  
I. H. Chaudry
Keyword(s):  
Blood Flow ◽  
Oxygen Delivery ◽  
Burn Injury ◽  
Splanchnic Blood Flow

Splanchnic blood flow, O2 consumption, removal of lactate, and output of glucose in highlanders

1977 ◽  
Vol 43 (2) ◽  
pp. 204-210 ◽  
Author(s):  
A. Capderou ◽  
J. Polianski ◽  
J. Mensch-Dechene ◽  
L. Drouet ◽  
G. Antezana ◽  
...  
Keyword(s):  
Blood Flow ◽  
Oxygen Consumption ◽  
Splanchnic Blood Flow ◽  
O2 Consumption ◽  
Arterial Lactate ◽  
Oxygen Breathing ◽  
Arterial Blood ◽  
Splanchnic Hemodynamics ◽  
Arterial Blood Glucose ◽  
Arteriovenous Difference

An impairment of gluconeogenesis has been proposed to explain the low arterial blood glucose of highlanders. Therefore, we studied splanchnic blood flow, splanchnic uptake of oxygen and lactate, and output of glucose in nine normal and six anemic highlanders at an altitude of 3,750 m. Splanchnic blood flow, arteriovenous difference for oxygen, and oxygen consumption were comparable at rest in both groups and in lowlanders from the literature, whereas splanchnic output of glucose, and uptake of lactate were approximately twice those in lowlanders. After 10 min of mild exercise in 12 subjects (7 normals, 5 anemic), no significant changes in splanchnic hemodynamics and metabolism were found. During 29% oxygen breathing in 8 subjects (5 normals, 3 anemics), arterial lactate, splanchnic uptake of lactate and output of glucose fell to normal sea-level values. We concluded that splanchnic hemodynamics are similar in adapted highlanders and in lowlanders, and that there is no evidence of an impaired gluconeogenesis at the altitude of the present study.

Splanchnic Blood Flow in Experimental Shock

1972 ◽  
pp. 87-107 ◽  
Author(s):  
Kenneth G. Swan ◽  
Randall W. Barton ◽  
David G. Reynolds
Keyword(s):  
Blood Flow ◽  
Splanchnic Blood Flow ◽  
Experimental Shock

Adenosine blockade reduces splanchnic blood flow in cirrhotic rats

1989 ◽  
Vol 9 ◽  
pp. S52
Author(s):  
Samuel S. Lee ◽  
Lisa Chilton
Keyword(s):  
Blood Flow ◽  
Splanchnic Blood Flow

Augmentation of Splanchnic Blood Flow

1995 ◽  
pp. 153-164 ◽  
Author(s):  
J.-L. Vincent ◽  
D. Schmartz ◽  
D. De Backer
Keyword(s):  
Blood Flow ◽  
Splanchnic Blood Flow

Splanchnic blood flow in the monkey during hemorrhagic shock

1965 ◽  
Vol 208 (2) ◽  
pp. 265-269 ◽  
Author(s):  
Francis L. Abel ◽  
John A. Waldhausen ◽  
Ewald E. Selkurt
Keyword(s):  
Blood Flow ◽  
Arterial Pressure ◽  
Portal Vein ◽  
Hemorrhagic Shock ◽  
Hepatic Vein ◽  
Venous Pressure ◽  
Portal Pressure ◽  
Mesenteric Arteries ◽  
Splanchnic Blood Flow

Blood flow in the celiac and superior mesenteric arteries was measured in nine Macaca monkeys during a standardized hemorrhagic shock procedure. Simultaneous pressures were obtained from the hepatic vein, portal vein, and aorta. Each animal was bled rapidly to an arterial pressure of 40 mm Hg and maintained at this level until 30% of the bled volume had spontaneously reinfused. The remaining blood was then rapidly reinfused and the animal observed until death. The results show a lack of overshoot of venous pressure on reinfusion, grossly pale intestines with some microscopic congestive changes, and a decrease in splanchnic conductance throughout the postinfusion period. Hepatic venous pressure exceeded portal pressure in six of the nine animals during the period of hemorrhage. The results are interpreted as indicative of insignificant splanchnic pooling during hemorrhagic shock in this animal.

Circulatory effects of RES depression in the dog

1965 ◽  
Vol 209 (3) ◽  
pp. 532-538 ◽  
Author(s):  
Edward F. Banaszak ◽  
John P. Kampine ◽  
Barbara A. Brault ◽  
James J. Smith
Keyword(s):  
Blood Flow ◽  
Mammalian Species ◽  
Traumatic Shock ◽  
Splanchnic Blood Flow ◽  
Phagocytic Index ◽  
Phagocytic Capacity ◽  
Thorium Dioxide ◽  
Circulatory Effects ◽  
Repeated Doses ◽  
Dose Levels

The response of the anesthetized dog to single and repeated doses of colloidal carbon gel (CCG) is quite similar to that reported in other mammalian species. At doses less than 30 mg/kg the clearance rate is high and limited only by splanchnic blood flow. At dose levels above 30 mg/kg the phagocytic index (K) approaches a constant. The minimal dose of CCG for assessment of phagocytic capacity was adjudged to be 40 mg/kg. Large doses of CCG (160 mg/kg) and colloidal SiO2 (350 mg/kg) induced a phagocytic depression accompanied by a marked granulocytopenia as well as prompt and delayed hypotensive reactions. There was a decline of estimated splanchnic blood flow incident to test doses of CCG but no further reduction after an RES-depressing dose of CCG. Colloidal thorium dioxide (4 and 8 ml/kg) did not depress the phagocytic capacity of the canine RES for CCG. It is suggested that the delayed hypotensive reaction may be related to the lesser tolerance to traumatic shock previously reported in RES-depressed animals.

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