Pacemaker Lead-induced Tricuspid Valve Stenosis and Reverse Lutembacher Syndrome

Author(s):  
Yuya Yamazaki ◽  
Takuya Wada ◽  
Takayuki Kadohama ◽  
Daichi Takagi ◽  
Kentaro Kiryu ◽  
...  
CASE ◽  
2021 ◽  
Author(s):  
Sharoojan Gunaseelan ◽  
Per Lehnert ◽  
Steen Hvitfeldt Poulsen ◽  
Jesper Khedri Jensen

2007 ◽  
Vol 9 (2) ◽  
pp. 123-128 ◽  
Author(s):  
David J. Connolly ◽  
Gina Neiger-Aeschbacher ◽  
Daniel J. Brockman

2011 ◽  
Vol 18 (3) ◽  
pp. e48-e49
Author(s):  
Jean-Francois Morin ◽  
Richard Sheppard ◽  
Patrick Chamoun

The present report is the first to describe a case of hemoptysis caused by an endocardial pacemaker lead. In addition, the patient presented with endocarditis and tricuspid valve stenosis. Aggressive treatment consisted of surgical extraction of two pacemaker leads and one pacemaker battery, replacement of the tricuspid valve and implantation of a DDD-R epicardial pacemaker.


2010 ◽  
Vol 63 (12) ◽  
pp. 1486
Author(s):  
Enrique Villagrán ◽  
Jacobo Silva ◽  
José E. Rodríguez

2010 ◽  
Vol 3 (3) ◽  
pp. 465-467 ◽  
Author(s):  
Ruben Uijlings ◽  
Jolanda Kluin ◽  
Remy Salomonsz ◽  
Mark Burgmans ◽  
Maarten-Jan Cramer

2013 ◽  
Vol 16 (4) ◽  
pp. 187
Author(s):  
Ali Kemal Gür ◽  
Özgür Gürsu ◽  
Helin El ◽  
Esra Eker ◽  
Mustafa Etli

The aim of the present study was to describe a successful method for treating rheumatic tricuspid valve stenosis with autologous pericardium, commissurotomy, and a Kalangos ring without replacing the tricuspid valve.


2011 ◽  
Vol 41 (7) ◽  
pp. 399 ◽  
Author(s):  
Hyun Sang Lee ◽  
Won Suk Choi ◽  
Kyun Hee Kim ◽  
Jung Kyu Kang ◽  
Na Young Kim ◽  
...  

2019 ◽  
Vol 3 (3) ◽  
Author(s):  
Kazuhito Hirata ◽  
Toshiho Tengan ◽  
Minoru Wake ◽  
Takanori Takahashi ◽  
Toru Ishimine ◽  
...  

Abstract Background Bioprosthetic tricuspid valve stenosis is a late sequela of tricuspid valve replacement (TVR); however, detailed information regarding its clinical picture is lacking. Case summary Thirty-one patients with bioprosthetic TVR (mean age: 60.5 ± 16.6 years, male/female: 11/20) were followed-up for 79.5 ± 49.1 months (14–188 months). Eleven patients developed bioprosthetic tricuspid valve stenosis (mean tricuspid gradient >5 mmHg) at a median interval of 96 months (interquartile range: 61–114 months). The mean tricuspid gradient at the time of tricuspid valve stenosis diagnosis was 10.9 ± 3.9 mmHg. Although the mid-term tricuspid valve stenosis-free survival was favourable (92.4% at 60 and 78.7% at 84 months), it had declined steeply to 31.5% by 120 months. Ten out of 11 tricuspid valve stenosis patients showed signs of right heart failure (RHF) as manifested by oedema and elevated jugular venous pressure, requiring moderate-to-high doses of diuretics. Diastolic rumble was audible in 10 patients. Five of the 11 tricuspid valve stenosis patients required redo TVR as a result of refractory RHF. Examination of the five excised bioprostheses showed pannus in four, fusion of the commissure in three, native valve attachment in two, and sclerosis in one. Detailed clinical pictures and pathology of the explanted valves in three cases that underwent surgery are presented in this case series. Discussion Bioprosthetic tricuspid valve stenosis is not uncommon after 8 years. Tricuspid valve replacement performed at the second surgery was associated with a higher incidence of bioprosthetic tricuspid valve stenosis.


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