Cervical dural arteriovenous malformation and large epidural venous varices in a rare adult presentation of congenital vascular bone syndrome

2015 ◽  
Vol 39 (4) ◽  
pp. 677-681 ◽  
Author(s):  
Vasant Garg ◽  
Sunil Manjila ◽  
Mark Corriveau ◽  
Nicholas C. Bambakidis ◽  
Jeffrey L. Sunshine
Neurosurgery ◽  
1993 ◽  
Vol 33 (1) ◽  
pp. 139-141 ◽  
Author(s):  
Howard C. Chandler ◽  
William A. Friedman

Neurosurgery ◽  
1983 ◽  
Vol 13 (2) ◽  
pp. 129-135 ◽  
Author(s):  
Leland A. Albright ◽  
Richard E. Latchaw ◽  
Robert A. Price

Abstract A 23-month-old infant with an extensive dural arteriovenous malformation (AVM) developed a heart murmur and cardiomegaly. The AVM involved the occipital and suboccipital dura mater and the tentorium, bilaterally. We embolized the AVM with Gelfoam and polyvinyl alcohol particulates, subtotally resected it, and embolized residual vessels with isobutyl cyanoacrylate, In spite of this extensive therapy, the malformation was not totally eradicated and an occipital pial AVM developed. This infant and the eight infants with posterior dural AVMs reported previously emphasize the difficulty of eradicating these lesions in infancy. We contrast the clinical features and therapy of infants and adults with posterior dural AVMs. Infants develop heart failure and cranial bruits beacause of arteriovenous shunts, whereas adults complain of headache and intracranial bruits. Infants need therapy to prevent progressive heart failure, intracranial hypertension, and perhaps cerebral ischemia. Ligation of arterial tributaries is inadequate therapy for these lesions. Embolization and resection of the malformation, when feasible, offers the best chance of curing posterior dural AVMs with extensive arterial tributaries.


1979 ◽  
Vol 51 (5) ◽  
pp. 715-717 ◽  
Author(s):  
Shunro Endo ◽  
Keiji Koshu ◽  
Jiro Suzuki

✓ The authors report a case of infratentorial arteriovenous malformation that regressed spontaneously within 25 months without bleeding episodes or surgical intervention. The patient's clinical symptoms, left-sided tinnitus, cranial bruit, and right homonymous hemianopsia, disappeared completely over the same period.


1981 ◽  
Vol 21 (1) ◽  
pp. 131-134 ◽  
Author(s):  
Shoji BITOH ◽  
Hidemitsu NAKAGAWA ◽  
Norio ARITA ◽  
Masaaki FUJIWARA

Neurosurgery ◽  
1991 ◽  
Vol 29 (1) ◽  
pp. 109-113 ◽  
Author(s):  
Yutaka Sawamura ◽  
Robert C. Janzer ◽  
Heinz Fankhauser ◽  
Nicolas de Tribolet

Abstract An unusual association of a meningioma and an arteriovenous malformation is reported. A 68-year-old man developed left homonymous hemianopsia. left hemiparesis. and gaze palsy. Magnetic resonance imaging showed a right occipital mass lesion containing multiple signal-void areas with tubular and honeycomb appearance, suggesting a marked vascular component. An angiogram showed abnormal vasculature in the mass supplied by the posterior cerebral artery and a dural arteriovenous malformation on the tentorium. Neuropathological examination after total removal of the mass revealed a meningothelial meningioma including major portions of an arteriovenous malformation that extended from the dura and leptomeninges. through the meningioma, and into the occipital lobe, where the tumor was located. (Neurosurgery 29:109-113, 1991)


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