Computational simulation of corrosion pit interactions under mechanochemical effects using a cellular automaton/finite element model

2016 ◽  
Vol 103 ◽  
pp. 305-311 ◽  
Author(s):  
Haitao Wang ◽  
En-Hou Han
Keyword(s):  
Finite Element ◽  
Finite Element Model ◽  
Cellular Automaton ◽  
Computational Simulation ◽  
Element Model ◽  
Corrosion Pit ◽  
Cellular Automaton Finite Element

scholarly journals A Cellular Automaton ∕ Finite Element model for predicting grain texture development in galvanized coatings

2011 ◽  
Author(s):  
G. Guillemot ◽  
M.-N. Avettand-Fènoël ◽  
A. Iosta ◽  
J. Foct ◽  
Francisco Chinesta ◽  
...  
Keyword(s):  
Finite Element ◽  
Finite Element Model ◽  
Cellular Automaton ◽  
Element Model ◽  
Texture Development ◽  
Grain Texture ◽  
Galvanized Coatings ◽  
Cellular Automaton Finite Element

scholarly journals A Computational Simulation of the Effect of Hemodilution on Oxygen Transport in Middle Cerebral Artery Vasospasm

2011 ◽  
Vol 31 (11) ◽  
pp. 2209-2217 ◽  
Author(s):  
Prashant Chittiboina ◽  
Bharat Guthikonda ◽  
Christian Wollblad ◽  
Steven A Conrad
Keyword(s):  
Finite Element ◽  
Blood Flow ◽  
Finite Element Model ◽  
Middle Cerebral Artery ◽  
Cerebral Artery ◽  
Oxygen Transport ◽  
Stokes Equations ◽  
Aneurysmal Subarachnoid Hemorrhage ◽  
Computational Simulation ◽  
Element Model

Cerebral vasospasm after aneurysmal subarachnoid hemorrhage is a potentially severe sequel. The induction of hypertension, hypervolemia, and hemodilution is advocated for vasospasm, but it is unclear whether hemodilution confers any benefit. A finite element model of oxygen transport in the proximal middle cerebral artery (MCA) was used to evaluate the complex relationship among hematocrit, viscosity, oxygen content, and blood flow in the setting of vasospasm. A single-phase non-Newtonian finite element model based on three-dimensional incompressible Navier–Stokes equations was constructed of the M1 segment. The model was solved at vessel stenoses ranging from 0% to 90% and hematocrit from 0.2 to 0.6. A small area of poststenotic recirculation was seen with mild (30%) stenosis. Poststenotic eddy formation was noted with more severe (60% to 90%) stenosis. Volumetric flow was inversely related to hematocrit at mild stenosis (0% to 30%). With near-complete stenosis (90%), a paradoxical increase in flow was seen with increasing hematocrit. Oxygen transport across the segment was related to hematocrit at all levels of stenosis with increasing oxygen transport despite a reduction in blood flow, suggesting that with clinically significant vasospasm in the MCA, hemodilution does not improve oxygen transport, but to the contrary, that ischemia may be worsened.

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