Percutaneous recanalization of a segmental inferior vena cava occlusion in a patient with situs viscerum inversus and symptomatic Budd-Chiari syndrome

2019 ◽  
Vol 51 (6) ◽  
pp. 909
Author(s):  
Roberto Miraglia ◽  
Luigi Maruzzelli ◽  
Giuseppe Mamone ◽  
Angelo Luca
1995 ◽  
Vol 43 (04) ◽  
pp. 227-229 ◽  
Author(s):  
S. Kuki ◽  
S. Taketani ◽  
R. Matsumura ◽  
A. Okuda ◽  
T. Yamaguchi ◽  
...  

2006 ◽  
Vol 14 (5) ◽  
pp. e88-e90 ◽  
Author(s):  
Kazuhisa Rikitake ◽  
Tsuyoshi Itoh ◽  
Masafumi Natsuaki ◽  
Yuji Katayama ◽  
Satoshi Ohtsubo ◽  
...  

2004 ◽  
Vol 57 (11) ◽  
pp. 1121-1123
Author(s):  
Ángel Sánchez-Recalde ◽  
Nicolás Sobrino ◽  
Guillermo Galeote ◽  
Luis Calvo Orbe ◽  
José L. Merino ◽  
...  

2015 ◽  
Vol 11 (3) ◽  
pp. 73-77 ◽  
Author(s):  
Saima Karim ◽  
Mohammad M. Karim ◽  
Victor Lucas ◽  
Anil Verma ◽  
Nigel Girgrah ◽  
...  

2016 ◽  
Vol 12 (1) ◽  
pp. 124-128
Author(s):  
Saima Karim ◽  
Victor Lucas ◽  
Anil Verma ◽  
Nigel Girgrah ◽  
Stephen Ramee ◽  
...  

MedPharmRes ◽  
2018 ◽  
Vol 2 (3) ◽  
pp. 22-26
Author(s):  
Uyen Vo ◽  
Duc Quach ◽  
Luan Dang ◽  
Thao Luu ◽  
Luan Nguyen

Budd–Chiari syndrome (BCS), a rare and life-threatening disorder due to hepatic venous outflow obstruction, is occasionally associated with hypoproteinemia. We herein report the first case of BCS with segmental obstruction of the intrahepatic portion of inferior vena cava (IVC) and hepatic veins (HVs) successfully treated by endovascular stenting in Vietnam. A 32-year-old female patient presented with a 2-month history of massive ascites and leg swelling. She refused history of oral contraceptives use. Hepatosplenomegaly without tenderness was noted. Laboratory data showed polycythemia, mild hypoalbuminemia and hypoproteinemia, slightly high total bilirubin and normal transaminase level. The serum ascites albumin gradient was 1.9 g/dL and ascitic protein level was 1.1 g/dL. The other data were normal. BCS was suspected because of the discrepancy between mild liver failure and massive ascites; and the presence of hepatosplenomegaly and polycythemia. On abdominal magnetic resonance imaging, the segmental obstruction of three HVs and IVC was 2-3 cm long without thrombus. Cavogram revealed the severe segmental stenosis of intrahepatic portion of IVC with no visualized HV and extensive collateral veins. A Protégé stent was deployed to IVC. Leg swelling and ascites were completely resolved within 3 days after stenting. During 1-year follow-up, edema was not recurred and repeated laboratory results were all normal.


2021 ◽  
pp. 153857442110020
Author(s):  
Reza Talaie ◽  
Hamed Jalaeian ◽  
Nassir Rostambeigi ◽  
Anthony Spano ◽  
Jafar Golzarian

Budd-Chiari syndrome (BCS) results from the occlusion or flow reduction in the hepatic veins or inferior vena cava and can be treated with transjugular intrahepatic portosystemic shunt when hepatic vein recanalization fails.1-3 Hypercoagulable patients with primary BCS are predisposed to development of new areas of thrombosis within the TIPS shunt or IVC. This case details a patient with BCS, pre-existing TIPS extending to the right atrium, and chronic retrohepatic IVC thrombosis who underwent sharp recanalization of the IVC with stenting into the TIPS stent bridging the patient until his subsequent hepatic transplantation.


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