LEMG Findings of Vocal Fold Paresis in Voice Patients Who Play or Played Wind Instruments

Author(s):  
Valerie Trollinger ◽  
Ghiath Alnouri ◽  
Robert T. Sataloff
Author(s):  
Chelsea Ridgway ◽  
Sarah Bouhabel ◽  
Lisa Martignetti ◽  
Yo Kishimoto ◽  
Nicole Y. K. Li-Jessen

2007 ◽  
Vol 6 (2) ◽  
pp. 98-102
Author(s):  
D. I. Malinin ◽  
V. G. Petrov

The aim of the study was to improve surgical results of patients having thyroid gland pathology by the development of the surgery method directed to increasing possibility of injury and maintenance of the upper laryngeal nerve integrity. Method of performing extrafascial hemithyreoidectomy with visualization of recurrent nerve, parathyroid gland and upper thyroid artery is presented which is directed to decrease specific complications (vocal fold paresis, hypoparathyreosis) and complications associated with the upper laryngeal nerve injury. Using this method, 166 patients having node pathology of thyroid gland were operated on. This method resulted in decreased number of complications (from 15,0 to 2,6%).


2014 ◽  
Vol 151 (1_suppl) ◽  
pp. P69-P70
Author(s):  
Paul M. Paddle ◽  
Masaany Mansor ◽  
Phillip Song ◽  
Ramon A. Franco

2020 ◽  
pp. 000348942094254
Author(s):  
Jason R. Crossley ◽  
Nathan Aminpour ◽  
Jonathan P. Giurintano ◽  
Ann K. Jay ◽  
Brent T. Harris ◽  
...  

Objectives: To present a novel location in which neurosarcoidomatous inflammation is identified and its accompanying presentation. Methods: The authors present a case of bilateral vocal fold paresis associated with non-caseating granulomatous inflammation of the cervical and intra-axial portions of the vagus nerve masquerading as a cranial nerve tumor. Results: Examination revealed bilateral vocal fold paresis and asymmetric palate elevation. MRI demonstrated enhancing bilateral jugular foramen masses, and neck ultrasound demonstrated bilateral thickened appearance of the vagus nerves. Vagus nerve biopsy demonstrated non-caseating granulomas. Conclusions: Neurosarcoidosis may contribute to variable cranial neuropathies. Vocal fold paresis is usually thought to arise from mediastinal compression of the left recurrent laryngeal nerve. Rarely, though, lesions may arise in other parts of the vagus nerve. Failure of response to steroids does not rule out the diagnosis, making tissue diagnosis important in some cases.


2009 ◽  
Vol 23 (3) ◽  
pp. 396-398 ◽  
Author(s):  
C. Blake Simpson ◽  
Esther J. Cheung ◽  
Carlayne J. Jackson

2014 ◽  
Vol 28 (6) ◽  
pp. 799-808 ◽  
Author(s):  
Xuhui Chen ◽  
Ping Wan ◽  
Yabin Yu ◽  
Ming Li ◽  
Yanyan Xu ◽  
...  

2017 ◽  
Vol 131 (S2) ◽  
pp. S48-S52 ◽  
Author(s):  
G Harris ◽  
C O'Meara ◽  
C Pemberton ◽  
J Rough ◽  
P Darveniza ◽  
...  

AbstractObjectives:To review the clinical signs of vocal fold paresis on laryngeal videostroboscopy, to quantify its impact on patients’ quality of life and to confirm the benefit of laryngeal electromyography in its diagnosis.Methods:Twenty-nine vocal fold paresis patients were referred for laryngeal electromyography. Voice Handicap Index 10 results were compared to 43 patients diagnosed with vocal fold paralysis. Laryngeal videostroboscopy analysis was conducted to determine side of paresis.Results:Blinded laryngeal electromyography confirmed vocal fold paresis in 92.6 per cent of cases, with vocal fold lag being the most common diagnostic sign. The laryngology team accurately predicted side of paresis in 76 per cent of cases. Total Voice Handicap Index 10 responses were not significantly different between vocal fold paralysis and vocal fold paresis groups (26.08 ± 0.21 and 22.93 ± 0.17, respectively).Conclusion:Vocal fold paresis has a significant impact on quality of life. This study shows that laryngeal electromyography is an important diagnostic tool. Patients with persisting dysphonia and apparently normal vocal fold movement, who fail to respond to appropriate speech therapy, should be investigated for a diagnosis of vocal fold paresis.


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