Horner’s syndrome and dissection of the internal carotid artery after chiropractic manipulation of the neck

2001 ◽  
Vol 131 (4) ◽  
pp. 523-524 ◽  
Author(s):  
Bobbie L. Parwar ◽  
Amani A. Fawzi ◽  
Anthony C. Arnold ◽  
Steven D. Schwartz
2009 ◽  
Vol 24 (1) ◽  
pp. 101-104 ◽  
Author(s):  
Carmen Fons ◽  
Monica Vasconcelos ◽  
Mariona Vidal ◽  
Ramón Puy ◽  
Antonio Capdevila ◽  
...  

2021 ◽  
Vol 14 (2) ◽  
pp. e234973
Author(s):  
Saadat Ali Saleemi ◽  
Ramesh Sahathevan

Horner’s syndrome results from interruption of the sympathetic innervation to the eye. This interruption may occur at three anatomical levels along the sympathetic trunk pathway. There are numerous causes of Horner’s syndrome, including injury to the carotid artery, of which arterial dissection is the commonest pathology. Occlusive carotid disease secondary to atherosclerosis is a relatively rare cause of Horner’s syndrome. We describe a patient with Horner’s syndrome due to complete occlusion of the ipsilateral internal carotid artery.


2011 ◽  
Vol 12 (04) ◽  
pp. 266-269 ◽  
Author(s):  
Hüseyin Özdemir ◽  
Kamran Mahmutyazicioğlu ◽  
Aysun Ünal ◽  
Ahmet Savranlar ◽  
H. Tuğrul Atasoy ◽  
...  

2015 ◽  
Vol 86 (11) ◽  
pp. e4.76-e4
Author(s):  
Ambika Kapoor ◽  
Krishna Chinthapalli ◽  
Graham Warner

Bilateral internal carotid artery (ICA) dissections are rare. Typical clinical presentation includes cerebral ischaemia, neck/headache, Horner's syndrome & pulsatile tinnitus1.We present a forty-two year old right-handed man admitted to hospital with a ten day gradual onset headache, altered taste, widespread cognitive impairments, horizontal diplopia and a right Horner's syndrome. There was no history of trauma. Brain magnetic resonance imaging revealed multiple small embolic infarcts in carotid territories of both cerebral hemispheres. Computed tomography angiogram (CTA) of intracranial and extracranial vessels revealed occlusion of the left ICA and attenuation of the right ICA below the skull base.Symptoms resolved within 3–5 days of starting sub-cutaneous low molecular weight heparin and he was then anticoagulated with warfarin for six months. Repeat CTA at 4 months showed persisting complete left ICA occlusion but complete recanalisation of the right ICA.This is the first description of cognitive changes following bilateral ICA artery dissections. Whilst ICA dissection presentations are usually explained by embolic phenomena or local effects upon ICA walls, the mechanism here is probably due to cerebral hypoperfusion. Cognitive assessment should be performed in all patients presenting with ICA dissection especially when it is bilateral.


2016 ◽  
Vol 2016 ◽  
pp. 1-3
Author(s):  
Jose Enrique Alonso Formento ◽  
Jose Luis Fernández Reyes ◽  
Blanca Mar Envid Lázaro ◽  
Teresa Fernández Letamendi ◽  
Ryth Yeste Martín ◽  
...  

Internal carotid artery dissection (ICAD) is a rare entity that either results from traumatic injury or can be spontaneously preceded or not by a minor trauma such as sporting activities. It represents a major cause of stroke in young patients. The diagnosis should be suspected with the combination of Horner’s syndrome, headache or neck pain, and retinal or cerebral ischaemia. The confirmation is frequently made with a magnetic resonance angiography (MRA). Although anticoagulation with heparin followed by vitamin-K-antagonists is the most common treatment, there is no difference in efficacy of antiplatelet and anticoagulant drugs at preventing stroke and death in patients with symptomatic carotid dissection. We describe a patient with ICAD following deep sea scuba diving, who presented with Horner’s syndrome and neck pain and was successfully treated with anticoagulants.


Author(s):  
N. Guy ◽  
D. Deffond ◽  
N. Carriere ◽  
G. Dordain ◽  
P. Clavelou ◽  
...  

Background:Typical presentation of spontaneous internal carotid artery (ICA) dissection is an ipsilateral pain in neck and face with Horner's syndrome and contralateral deficits. Although rare, lower cranial nerve palsy have been reported in association with an ipsilateral spontaneous ICA dissection.Case studies:We report three new cases of ICA dissection with lower cranial nerve palsies.Results:The first symtom to appear was headache in all three patients. Examination disclosed a Horner's syndrome in two cases (1 and 2), an isolated XIIth nerve palsy in two patients (case 1 and 3) and IX, X, and XIIth nerve palsies (case 2) revealing an ipsilateral carotid dissection, confirmed by MRI and angiography. In all cases, prognosis was good after a few weeks.Conclusion:These cases, analysed with those in the literature, led us to discuss two possible mechanisms: direct compression of cranial nerves by a subadventitial haematoma in the parapharyngeal space or ischemic palsy by compression of the ascending pharyngeal artery.


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