Abduction Defect Associated With Aberrant Regeneration of the Oculomotor Nerve After Intracranial Aneurysm

1996 ◽  
Vol 121 (5) ◽  
pp. 580-582 ◽  
Author(s):  
Michael L. Slavin ◽  
Kenneth R. Einberg
Keyword(s):  
Intracranial Aneurysm ◽  
Oculomotor Nerve ◽  
Aberrant Regeneration

scholarly journals IMPLICIT OF CONTRALATERAL EYE SURGERY IN ABERRANT REGENERATION OF OCULOMOTOR NERVE PALSY (OCULOMOTOR SYNKINESIS)

2020 ◽  
Vol 8 (3) ◽  
pp. 1092-1094
Author(s):  
V Rajesh Prabu ◽  
◽  
Rajlaxmi B Wasnik ◽  
Parul Priyambada ◽  
Ranjini H ◽  
...  
Keyword(s):  
Nerve Palsy ◽  
Oculomotor Nerve ◽  
Oculomotor Nerve Palsy ◽  
Eye Surgery ◽  
Aberrant Regeneration ◽  
Contralateral Eye

scholarly journals Isolated Oculomotor Palsy Caused by Intracranial Aneurysm

1947 ◽  
Vol 40 (8) ◽  
pp. 419-432 ◽  
Author(s):  
Geoffrey Jefferson
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Intracranial Aneurysm ◽  
Subarachnoid Haemorrhage ◽  
Internal Carotid ◽  
Acute Stage ◽  
Oculomotor Nerve ◽  
Safe Procedure ◽  
Oculomotor Palsy ◽  
Correct Treatment

The present paper is concerned with the 55 aneurysms out of a total of 158 that caused isolated paralysis of the oculomotor nerve. The majority arose from the internal carotid artery after it had pierced the dura (supraclinoid). Rarely the aneurysm sprang from the basilar artery. In two-thirds of the cases there had been a subarachnoid hæmorrhage from leakage. Not more than 10% of patients had arteriosclerosis. Calcification of the sac is not a sign that the aneurysm has thrombosed. The only certain way of demonstrating the position and size of an intracranial aneurysm is by arteriography, which is a safe procedure. The correct treatment is by carotid ligature. In about 8% of normals the circle of Willis is incomplete, therefore percutaneous compression must first be tried. The only fatalities from ligature were in persons in the acute stage of subarachnoid hæmorrhage, not from meningeal bleeding alone. In this type of case a clip applied to the neck of the sac is probably a better method. In the more usual cases where the hæmorrhage has been spontaneously arrested common carotid ligature in the neck is probably a little safer than intracranial clipping.

scholarly journals ABERRANT REGENERATION OF OCULOMOTOR NERVE- LID GAZE DYSKINESIS: A CASE REPORT

2021 ◽  
pp. 1-1
Author(s):  
Sanjeev Krishan ◽  
Deepak Kumar Sharma
Keyword(s):  
Case Report ◽  
Signs And Symptoms ◽  
Oculomotor Nerve ◽  
Aberrant Regeneration

Aberrant regeneration of Oculomotor nerve results in the abnormal contraction of the muscles. It occurs because of failure to recover completely after injury to the oculomotor nerve. [1] Here we present a case who presented to us with the signs and symptoms of Aberrant Regeneration of Oculomotor Nerve.

Aberrant regeneration of the oculomotor nerve: implications for neurosurgeons

2007 ◽  
Vol 23 (5) ◽  
pp. E14 ◽  
Author(s):  
Eric D. Weber ◽  
Steven A. Newman
Keyword(s):  
Cranial Nerve ◽  
Clinical Signs ◽  
Treatment Strategies ◽  
Oculomotor Nerve ◽  
Oculomotor Palsy ◽  
Upper Eyelid ◽  
Aberrant Regeneration ◽  
Nerve Dysfunction ◽  
Neurosurgical Intervention

✓Aberrant regeneration of cranial nerve III, otherwise known as oculomotor synkinesis, is an uncommon but well-described phenomenon most frequently resulting from trauma, tumors, and aneurysms. Its appearance usually follows an oculomotor palsy, but it can also occur primarily without any preceding nerve dysfunction. It is vital that neurosurgeons recognize this disorder because it may be the only sign of an underlying cavernous tumor or PCoA aneurysm. The tumor most often implicated is a cavernous or parasellar meningioma, but any tumor that causes compression or disruption along the course of the oculomotor nerve may cause primary or secondary misdirection. The most common clinical signs of oculomotor synkinesis consist of elevation of the upper eyelid on attempted downward gaze or adduction, adduction of the eye on attempted upward or downward gaze, and constriction of the pupil on attempted adduction. The authors present the largest series of patients with oculomotor synkinesis, including those in whom it developed after neurosurgical intervention, to illustrate various presentations. In addition, the various mechanisms that contribute to synkinesis are reviewed. Last, the treatment strategies for both oculomotor palsies and synkinesis are discussed.

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