Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation
2000 ◽
Vol 1
(3)
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pp. 163-184
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1998 ◽
Vol 18
(4)
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pp. 363-369
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