Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutation

2001 ◽  
Vol 21 (1) ◽  
pp. 67-81 ◽  
Author(s):  
Shinsuke Kato ◽  
Kenji Nakashima ◽  
Seikoh Horiuchi ◽  
Ryoji Nagai ◽  
Don W. Cleveland ◽  
...  
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