scholarly journals Psychotic Disorder or Complex Partial Seizure Disorder Due to a Sequelae of Herpes Simplex Encephalitis and Coincidental Parasagittal Meningioma

Author(s):  
Elif Akcay ◽  
Filiz Karadag ◽  
Tugba Tunc
1997 ◽  
Vol 145 (1) ◽  
pp. 154-164 ◽  
Author(s):  
Kost Elisevich ◽  
Sandra A. Rempel ◽  
Brien J. Smith ◽  
Klaus Edvardsen

1994 ◽  
Vol 6 (3) ◽  
pp. 499-517 ◽  
Author(s):  
Rochelle Caplan ◽  
Donald Guthrie ◽  
W. Donald Shields ◽  
Sue Yudovin

AbstractBased on findings that, like schizophrenic children, children with complex partial seizure disorder have illogical thinking, this study examined whether or not these same epileptic subjects had other thought processing impairments found in schizophrenic children. Compared to normal children (N = 38), both the epileptic (N = 27) and schizophrenic children (N = 31) underutilized discourse (cohesive) devices that make speech coherent. The severity of the discourse deficits and formal thought disorder of the schizophrenic children was associated with age, but not with IQ scores. Age, IQ, and seizure-related, not behavioral variables, were associated, however, with the severity of the discourse deficits of the epileptic subjects. The study' findings suggest that measures of impaired thought processing and communication in schizophrenic children could be a model to study the developmental impact of complex partial seizure disorder in middle childhood. The clinical implications of the study's findings are discussed.


1998 ◽  
Vol 39 (8) ◽  
pp. 1145-1152 ◽  
Author(s):  
Rochelle Caplan ◽  
Donald Guthrie ◽  
Scott Komo ◽  
W. Donald Shields

1992 ◽  
Vol 33 (8) ◽  
pp. 1399-1412 ◽  
Author(s):  
Rochelle Caplan ◽  
Donald Guthrie ◽  
W. Donald Shields ◽  
Lisa Mori

1994 ◽  
Vol 8 (1) ◽  
pp. 55-64 ◽  
Author(s):  
Barbara Pendleton Jones ◽  
Connie C. Duncan ◽  
Allan F. Mirsky ◽  
Robert M. Post ◽  
William H. Theodore

2017 ◽  
Vol 2017 ◽  
pp. 1-5 ◽  
Author(s):  
Kenneth Asogwa ◽  
Kwame Buabeng ◽  
Amarjit Kaur

Historically, psychotic disorder has been associated with viral infection. Herpes simplex infections and Epstein-Barr virus (EBV) among other viral infections have been implicated in psychotic disorder. Of note in this case report is psychotic disorder that occurred following reactivation of herpes simplex infection in a background of mannose-binding lecithin (MBL) deficiency, childhood EBV infection, and severe psychosocial stress. Herpes simplex encephalitis (HSE) remains a significant cause of morbidity and mortality despite advancement in its treatment with intravenous acyclovir. Many studies have reported psychiatric and neurological manifestation of herpes simplex infection following primary or reactivated infection, while others suggest milder clinical course of herpes simplex encephalitis in a background of immunosuppression. Another contributory factor to psychotic disorder in this case is childhood EBV exposure which has been reported to increase the risk of psychosis in adolescence and adulthood. This case report describes a 15-year-old female with MBL deficiency who presented with psychosis caused by reactivated herpes simplex infection and had good clinical recovery. Based on childhood Epstein-Barr virus exposure and psychosis in adolescence (current case), she is at increased risk of psychotic disorder in adulthood, which underscores the importance of long-term monitoring.


1988 ◽  
Author(s):  
Richard J. Roberts ◽  
Jane S. Paulsen ◽  
James N. Marchman ◽  
Nils R. Varney

1996 ◽  
Vol 76 (02) ◽  
pp. 184-186 ◽  
Author(s):  
Kenji lijima ◽  
Fumiyo Murakami ◽  
Yasushi Horie ◽  
Katsumi Nakamura ◽  
Shiro Ikawa ◽  
...  

SummaryA 74-year-old female developed pneumonia following herpes simplex encephalitis. Her white blood cell counts reached 28,400/μl, about 90% of which consisted of granulocytes. The polymorphonuclear (PMN) elastase/α1-arantitrypsin complex levels increased and reached the maximum of 5,019 ng/ml, indicating the release of a large amount of elastase derived from the granulocytes. The mechanism of PMN elastase release was most likely to be granulocyte destruction associated with phagocytosis. The cleavage of fibrinogen and fibrin by PMN elastase, independent of plasmin, was indicated by the presence of the fragments in immunoprecipitated plasma from the patient corresponding to elastase-induced FDP D and DD fragments and the absence of fragments corresponding to plasmin-induced FDP D and DD fragments on SDS-PAGE. These findings suggested that the large amount of PMN elastase released from the excessive numbers of granulocytes in this patient with herpes simplex encephalitis and pneumonia, induced the cleavage of fibrinogen and fibrin without the participation of plasmin.


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