gast-2. Mesenteric Vein Thrombosis in a Patient with Pancreatitis and Protein C Deficiency.

2003 ◽  
Vol 96 (Supplement) ◽  
pp. S55
Author(s):  
Andrew I. Rackoff
2007 ◽  
Vol 40 (2) ◽  
pp. 204-208 ◽  
Author(s):  
Toshiki Yamakawa ◽  
Yuji Onoda ◽  
Ryuichirou Ohashi ◽  
Sadanobu Izumi ◽  
Ichio Suzuka ◽  
...  

1992 ◽  
Vol 9 (5) ◽  
pp. 257-261
Author(s):  
R.J. Weinel ◽  
J. Kussmann ◽  
M. Rothmund

2008 ◽  
Vol 41 (6) ◽  
pp. 682-687 ◽  
Author(s):  
Akira Toyoshima ◽  
Takeshi Endo ◽  
Sakae Nagaoka ◽  
Yoshikazu Maruyama ◽  
Keisuke Sakai ◽  
...  

2012 ◽  
Vol 73 (9) ◽  
pp. 2290-2294
Author(s):  
Itaru ISHIBASHI ◽  
Masaki KAWAHARA ◽  
Hiroaki KOBAYASHI ◽  
Yoko TAKINO ◽  
Takashi KODAMA ◽  
...  

Diagnostics ◽  
2021 ◽  
Vol 11 (8) ◽  
pp. 1348
Author(s):  
Ying-Ying Chen ◽  
Sheng-Mao Wu ◽  
Russell Oliver Kosik ◽  
Yi-Chien Hsieh ◽  
Tzu-I Wu ◽  
...  

Acute abdominal pain during pregnancy is challenging, both from a diagnostic and management perspective. A non-localized, persistent pain out of proportion to physical examination is a sign that advanced imaging may be necessary. Mesenteric venous thrombosis in a pregnant patient is extremely rare, but if diagnosis is delayed, can be potentially fatal to both the mother and the fetus. We present here a pregnant patient in the tenth week of gestation with classic clinical manifestations of mesenteric vein thrombosis and the corresponding findings on magnetic resonance imaging (MRI) and computed tomography (CT).


TH Open ◽  
2021 ◽  
Vol 05 (02) ◽  
pp. e171-e173
Author(s):  
Kiyoko Kanosue ◽  
Satomi Nagaya ◽  
Eriko Morishita ◽  
Masayoshi Yamanishi ◽  
Shinsaku Imashuku

AbstractA 78-year-old Japanese male with Clostridium perfringens septicemia and cholecystitis was found to have thrombosis in the left branch of intrahepatic portal vein as well as superior mesenteric vein. Visceral vein thrombosis (VVT) in this case was associated with protein C deficiency, due to a heterozygous mutation, p. Arg185Met. Our experience emphasizes that VVT, or other thromboembolic events, may occur in later life, triggered by environmental thrombosis risk factors, together with underlying hereditary protein C gene mutation.


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