The Local and Referred Pain From Myofascial Trigger Points in the Temporalis Muscle Contributes to Pain Profile in Chronic Tension-type Headache

2007 ◽  
Vol 23 (9) ◽  
pp. 786-792 ◽  
Author(s):  
César Fernández-de-las-Peñas ◽  
Hong-You Ge ◽  
Lars Arendt-Nielsen ◽  
Maria Luz Cuadrado ◽  
Juan A. Pareja
2010 ◽  
Vol 14 (4) ◽  
pp. 391-396 ◽  
Author(s):  
César Fernández-de-las-Peñas ◽  
Hong-You Ge ◽  
Cristina Alonso-Blanco ◽  
Javier González-Iglesias ◽  
Lars Arendt-Nielsen

2011 ◽  
Vol 12 (1) ◽  
pp. 35-43 ◽  
Author(s):  
César Fernández-de-las-Peñas ◽  
Daniel M. Fernández-Mayoralas ◽  
Ricardo Ortega-Santiago ◽  
Silvia Ambite-Quesada ◽  
Domingo Palacios-Ceña ◽  
...  

2009 ◽  
Vol 25 (6) ◽  
pp. 506-512 ◽  
Author(s):  
César Fernández-de-las-Peñas ◽  
Ana B. Caminero ◽  
Pascal Madeleine ◽  
Amparo Guillem-Mesado ◽  
Hong-You Ge ◽  
...  

2007 ◽  
Vol 23 (1) ◽  
pp. 23-27 ◽  
Author(s):  
Christian Coupp?? ◽  
Paola Torelli ◽  
Anders Fuglsang-Frederiksen ◽  
Kjeld Visti Andersen ◽  
Rigmor Jensen

Cephalalgia ◽  
2007 ◽  
Vol 27 (5) ◽  
pp. 383-393 ◽  
Author(s):  
C Fernández-de-las-Peñas ◽  
ML Cuadrado ◽  
L Arendt-Nielsen ◽  
DG Simons ◽  
JA Pareja

Present pain models for tension-type headache suggest that nociceptive inputs from peripheral tender muscles can lead to central sensitization and chronic tension-type headache (CTTH) conditions. Such models support that possible peripheral mechanisms leading to pericranial tenderness include activation or sensitization of nociceptive nerve endings by liberation of chemical mediators (bradikinin, serotonin, substance P). However, a study has found that nonspecific tender points in CTTH subjects were not responsible for liberation of algogenic substances in the periphery. Assuming that liberation of algogenic substances is important, the question arising is: if tender muscle points are not the primary sites of on-going neurogenic inflammation, which structure can be responsible for liberation of chemical mediators in the periphery? A recent study has found higher levels of algogenic substances, and lower pH levels, in active myofascial trigger point (TrPs) compared with control tender points. Clinical studies have demonstrated that referred pain elicited by head and neck muscles contribute to head pain patterns in CTTH. Based on available data, an updated pain model for CTTH is proposed in which headache can at least partly be explained by referred pain from TrPs in the posterior cervical, head and shoulder muscles. In this updated pain model, TrPs would be the primary hyperalgesic zones responsible for the development of central sensitization in CTTH.


2006 ◽  
Vol 46 (8) ◽  
pp. 1264-1272 ◽  
Author(s):  
Cesar Fernandez-de-las-Penas ◽  
Cristina Alonso-Blanco ◽  
Maria Luz Cuadrado ◽  
Robert D. Gerwin ◽  
Juan A. Pareja

2006 ◽  
Vol 14 (4) ◽  
pp. 222-231 ◽  
Author(s):  
César Fernández-de-las-Peñas ◽  
Lars Arendt-Nielsen ◽  
David G. Simons

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