Current and Future Management Strategies for Intracranial Aneurysms and Carotid Artery Stenosis

2005 ◽  
pp. 1-17
Author(s):  
Eric M. Deshaies ◽  
Alan S. Boulos
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Management Strategies ◽  
Artery Stenosis ◽  
Future Management

Management of carotid artery stenosis in patients with coexistent unruptured intracranial aneurysms

2020 ◽  
Vol 132 (1) ◽  
pp. 94-97 ◽  
Author(s):  
Tiziano Tallarita ◽  
Thomas J. Sorenson ◽  
Lorenzo Rinaldo ◽  
Gustavo S. Oderich ◽  
Thomas C. Bower ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Early Period ◽  
Aneurysm Rupture ◽  
Artery Stenosis ◽  
Consecutive Series ◽  
Carotid Revascularization ◽  
Unruptured Intracranial Aneurysms ◽  
Risk Of Rupture

OBJECTIVEConcomitant unruptured intracranial aneurysms (UIAs) are present in patients with carotid artery stenosis not infrequently and result in unique management challenges. Thus, we investigated the risk of rupture of an aneurysm after revascularization of a carotid artery in a contemporary consecutive series of patients seen at our institution.METHODSData from patients who underwent a carotid revascularization in the presence of at least one concomitant UIA at our institution from 1991 to 2018 were retrospectively reviewed. Patients were evaluated for the incidence of aneurysm rupture within 30 days (early period) and after 30 days (late period) of carotid revascularization, as well as for the incidence of periprocedural complications from the treatment of carotid stenosis and/or UIA.RESULTSOur study included 53 patients with 63 concomitant UIAs. There was no rupture within 30 days of carotid revascularization. The overall risk of rupture was 0.87% per patient-year. Treatment (coiling or clipping) of a concomitant UIA, if pursued, could be performed successfully after carotid revascularization.CONCLUSIONSCarotid artery revascularization in the setting of a concomitant UIA can be performed safely without an increased 30-day or late-term risk of rupture. If indicated, treatment of the UIA can take place after the patient recovers from the carotid procedure.

scholarly journals Incidental Intracranial Aneurysms in Patients With Internal Carotid Artery Stenosis

Stroke ◽  
2009 ◽  
Vol 40 (4) ◽  
pp. 1341-1346 ◽  
Author(s):  
Laura M. Héman ◽  
Lisa M. Jongen ◽  
H. Bart van der Worp ◽  
Gabriel J.E. Rinkel ◽  
Jeroen Hendrikse
Keyword(s):  
Carotid Artery ◽  
Internal Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Internal Carotid ◽  
Internal Carotid Artery Stenosis ◽  
Artery Stenosis

Small, unruptured intracranial aneurysms and management of symptomatic carotid artery stenosis

Neurology ◽  
2000 ◽  
Vol 55 (2) ◽  
pp. 307-309 ◽  
Author(s):  
L. J. Kappelle ◽  
M. Eliasziw ◽  
A. J. Fox ◽  
H. J. M. Barnett
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Artery Stenosis ◽  
Symptomatic Carotid ◽  
Unruptured Intracranial Aneurysms

Concomitant Unruptured Intracranial Aneurysms and Carotid Artery Stenosis: An Institutional Review of Patients Undergoing Carotid Revascularization

2014 ◽  
Vol 28 (1) ◽  
pp. 102-107 ◽  
Author(s):  
Matthew J. Borkon ◽  
Han Hoang ◽  
Caron Rockman ◽  
Firas Mussa ◽  
Neal S. Cayne ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Artery Stenosis ◽  
Carotid Revascularization ◽  
Institutional Review ◽  
Unruptured Intracranial Aneurysms

scholarly journals Management of Carotid Artery Stenosis in Patients With Coexistent Unruptured Intracranial Aneurysms

2019 ◽  
Vol 69 (6) ◽  
pp. 2001
Author(s):  
T. Tallarita ◽  
T.J. Sorenson ◽  
L. Rinaldo ◽  
G.S. Oderich ◽  
T.C. Bower ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Intracranial Aneurysms ◽  
Artery Stenosis ◽  
Unruptured Intracranial Aneurysms

scholarly journals PC108. Comparison of Management Strategies for Asymptomatic Carotid Artery Stenosis

2015 ◽  
Vol 61 (6) ◽  
pp. 146S
Author(s):  
Alok Aggarwal ◽  
Dhiren Patel ◽  
Amy M. Maselli ◽  
Michael Shapiro ◽  
Donald A. Risucci ◽  
...  
Keyword(s):  
Carotid Artery ◽  
Carotid Artery Stenosis ◽  
Management Strategies ◽  
Artery Stenosis ◽  
Asymptomatic Carotid ◽  
Asymptomatic Carotid Artery Stenosis

Oxidative injury associated with stenting of asymptomatic carotid artery stenosis

VASA ◽  
2017 ◽  
Vol 46 (4) ◽  
pp. 268-274
Author(s):  
Erhan Saraçoğlu ◽  
Ertan Vuruşkan ◽  
Yusuf Çekici ◽  
Salih Kiliç ◽  
Halil Ay ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Carotid Artery ◽  
Oxidative Damage ◽  
Carotid Artery Stenosis ◽  
Artery Stenosis ◽  
Oxidative Stress Marker ◽  
Asymptomatic Carotid ◽  
Imaging Methods ◽  
Neurological Findings ◽  
Asymptomatic Carotid Artery Stenosis

Abstract. Background: After carotid artery stenting (CAS), neurological complications that cannot be explained with imaging methods may develop. In our study we aimed to show, using oxidative stress markers, isolated oxidative damage and resulting neurological findings following CAS in patients with asymptomatic carotid artery stenosis. Patients and methods: We included 131 neurologically asymptomatic patients requiring CAS. The neurological findings were evaluated using the modified Rankin Scale (mRS) prior to the procedure, one hour post-procedure, and two days after. Patients with elevated mRS scores but with or without typical hyperintense lesions observed on an MRI and with changes of oxidative stress marker levels at the time (Δtotal-thiol, Δtotal antioxidative status [TAS], and Δtotal oxidant status [TOS]) were evaluated. Results: In the neurological examination carried out one hour prior to the procedure, there were 92 patients with mRS = 0, 20 with mRS = 1, and 12 with mRS = 2. When Δtotal-thiol, ΔTAS, and ΔTOS values and the mRS were compared, it was observed that as the difference in oxidative parameters increased, clinical deterioration also increased proportionally (p = 0.001). Conclusions: We demonstrate a possible correlation between oxidative damage and neurological findings after CAS which could not be explained by routine imaging methods.

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