Subclavian steal syndrome in a post-coronary artery bypass patient

2013 ◽  
Vol 23 (6) ◽  
pp. 738-739
Author(s):  
Min Er Ching ◽  
Paul Jau Lueng Ong ◽  
Hee Hwa Ho
Cardiology ◽  
2020 ◽  
Vol 145 (9) ◽  
pp. 601-607
Author(s):  
Hassan M. Lak ◽  
Rohan Shah ◽  
Beni Rai Verma ◽  
Eric Roselli ◽  
Francis Caputo ◽  
...  

Coronary subclavian steal syndrome (CSSS) is a rare cause of angina. It occurs in patients with prior coronary artery bypass grafting and, specifically, a left internal mammary artery (LIMA) to left anterior descending artery (LAD) graft and co-existent significant subclavian artery stenosis. In this context, there is retrograde blood flow through the LIMA to LAD graft to supply the subclavian artery beyond the significant stenosis. This potentially occurs at the cost of compromising coronary artery perfusion dependent on the LIMA graft. In this review, we present a case of a middle-aged female who suffered from CSSS and review the literature for the contemporary diagnosis and management of this condition.


2014 ◽  
Vol 2014 ◽  
pp. 1-6 ◽  
Author(s):  
Usman Younus ◽  
Brandon Abbott ◽  
Deepika Narasimha ◽  
Brian J. Page

Coronary subclavian steal syndrome is a rare complication of coronary artery bypass grafting surgery (CABG) when a left internal mammary artery (LIMA) graft is utilized. This syndrome is characterized by retrograde flow from the LIMA to the left subclavian artery (SA) when a proximal left SA stenosis is present. We describe a unique case of an elderly male who underwent CABG 6 years ago who presented with prolonged chest pain, mildly elevated troponins, and unequal pulses in his arms. A CTA of the chest demonstrated a severely calcified occluded proximal left SA jeopardizing his LIMA graft. Subclavian angiography was performed with an attempt to revascularize the patient’s occluded left SA which was unsuccessful. We referred the patient for nuclear stress testing which demonstrated a moderate size area of anterior ischemia on imaging; the patient exercised to a fair exercise capacity of 7 METS with no chest pain and no ECG changes. Subsequent coronary angiography showed severe native three-vessel coronary artery disease with intermittent retrograde blood flow from the LIMA to the left SA distal to the occlusion, jeopardizing perfusion to the left anterior descending (LAD) coronary artery distribution. He declined further options for revascularization and was discharged with medical management.


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