Predicting menstrual migraine with a home-use fertility monitor

Neurology ◽  
2005 ◽  
Vol 64 (3) ◽  
pp. 561-563 ◽  
Author(s):  
E. A. MacGregor ◽  
A. Frith ◽  
J. Ellis ◽  
L. Aspinall
Neurology ◽  
1971 ◽  
Vol 21 (8) ◽  
pp. 853-853 ◽  
Author(s):  
B. W. Somerville
Keyword(s):  

1979 ◽  
Vol 19 (2) ◽  
pp. 063-070 ◽  
Author(s):  
J. Lehtonen ◽  
M. T. Hyyppa ◽  
H-L. Kaihola ◽  
P. Kangasniemi ◽  
A. H. Lang

Cephalalgia ◽  
2000 ◽  
Vol 20 (3) ◽  
pp. 148-154 ◽  
Author(s):  
S D Silberstein ◽  
G R Merriam

The normal female life cycle is associated with a number of hormonal milestones: menarche, pregnancy, contraceptive use, menopause, and the use of replacement sex hormones. All these events and interventions alter the levels and cycling of sex hormones and may cause a change in the prevalence or intensity of headache. The menstrual cycle is the result of a carefully orchestrated sequence of interactions among the hypothalamus, pituitary, ovary, and endometrium, with the sex hormones acting as modulators and effectors at each level. Oestrogen and progestins have potent effects on central serotonergic and opioid neurons, modulating both neuronal activity and receptor density. The primary trigger of menstrual migraine appears to be the withdrawal of oestrogen rather than the maintenance of sustained high or low oestrogen levels. However, changes in the sustained oestrogen levels with pregnancy (increased) and menopause (decreased) appear to affect headaches. Headaches that occur with premenstrual syndrome appear to be centrally generated, involving the inherent rhythm of CNS neurons, including perhaps the serotonergic pain-modulating systems.


1999 ◽  
Vol 8 (7) ◽  
pp. 919-931 ◽  
Author(s):  
STEPHEN D. SILBERSTEIN
Keyword(s):  

Neurology ◽  
1997 ◽  
Vol 48 (1) ◽  
pp. 101-102 ◽  
Author(s):  
A. G. Herzog
Keyword(s):  

Cephalalgia ◽  
1994 ◽  
Vol 14 (6) ◽  
pp. 411-412 ◽  
Author(s):  
VK Gupta

Facchinetti and colleagues present epidemiological evidence regarding comorbidity between menstrual migraine and premenstrual syndrome, and suggest that premenstural symptoms should be incorporated in the diagnostic criteria for menstrual migraine (1). The crux of the matter, however, should be the concern regarding the nature or biological significance of the common neuroendocrine link of transient and cyclic failure of endogenous opioid activity in both premenstrual syndrome and menstrual migraine patients. Is this a primary event of pathogenetic importance (which would merit inclusion in the definition) or the concomitant side effect of a carefully orchestrated adaptive mechanism?


2003 ◽  
Vol &NA; (1386) ◽  
pp. 15-16
Author(s):  
RM Poole
Keyword(s):  

2007 ◽  
Vol 0 (0) ◽  
pp. 071120093031001-??? ◽  
Author(s):  
Vincent Martin ◽  
Roger Cady ◽  
Alexander Mauskop ◽  
Larry S. Seidman ◽  
Anthony Rodgers ◽  
...  

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