scholarly journals The Role of GluA1 in Ocular Dominance Plasticity in the Mouse Visual Cortex

2013 ◽  
Vol 33 (38) ◽  
pp. 15220-15225 ◽  
Author(s):  
A. Ranson ◽  
F. Sengpiel ◽  
K. Fox
2007 ◽  
Vol 58 ◽  
pp. S98
Author(s):  
Tomoki Fukai ◽  
Siu Kang ◽  
Yoko Yazaki-Sugiyama ◽  
Hideyuki Cateau ◽  
Takao K. Hensch

2006 ◽  
Vol 16 (4) ◽  
pp. 451-459 ◽  
Author(s):  
Sonja B Hofer ◽  
Thomas D Mrsic-Flogel ◽  
Tobias Bonhoeffer ◽  
Mark Hübener

2005 ◽  
Vol 8 (3) ◽  
pp. 380-388 ◽  
Author(s):  
Yoshiaki Tagawa ◽  
Patrick O Kanold ◽  
Marta Majdan ◽  
Carla J Shatz

1998 ◽  
Vol 31 ◽  
pp. S181
Author(s):  
Kazuhiko Nakadate ◽  
Kazuyuki Imamura ◽  
Masayuki Kobayashi ◽  
Peter A. Kaub ◽  
Yasuyoshi Watanabe

2017 ◽  
Vol 372 (1715) ◽  
pp. 20160504 ◽  
Author(s):  
Megumi Kaneko ◽  
Michael P. Stryker

Mechanisms thought of as homeostatic must exist to maintain neuronal activity in the brain within the dynamic range in which neurons can signal. Several distinct mechanisms have been demonstrated experimentally. Three mechanisms that act to restore levels of activity in the primary visual cortex of mice after occlusion and restoration of vision in one eye, which give rise to the phenomenon of ocular dominance plasticity, are discussed. The existence of different mechanisms raises the issue of how these mechanisms operate together to converge on the same set points of activity. This article is part of the themed issue ‘Integrating Hebbian and homeostatic plasticity’.


2019 ◽  
Vol 56 (9) ◽  
pp. 5987-5997 ◽  
Author(s):  
Elena Maria Boggio ◽  
Erich M. Ehlert ◽  
Leonardo Lupori ◽  
Elizabeth B. Moloney ◽  
Fred De Winter ◽  
...  

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