Orbital apex syndrome secondary to nasopharyngeal carcinoma

2015 ◽  
Vol 4 (2) ◽  
pp. 177
Author(s):  
CS Sandhya ◽  
DMurali Krishna ◽  
C Jagannath ◽  
G Srinivas ◽  
K Radhika
Author(s):  
Masood Bagheri ◽  
Ahad Jafari ◽  
Sasan Jafari

2021 ◽  
pp. 32-40
Author(s):  
Yusuke Murai ◽  
Takuji Kurimoto ◽  
Sotaro Mori ◽  
Kaori Ueda ◽  
Mari Sakamoto ◽  
...  

We report a rare case of granulomatosis with polyangiitis (GPA) presenting with bilateral orbital apex syndrome (OAS). A 73-year-old woman with a history of endoscopic sinus surgery for ethmoidal sinusitis experienced a sudden decrease in visual acuity (VA) of both eyes. At the initial examination, her VA had decreased to 0.01 in the right eye and 0.03 in the left eye, and eye movement in both eyes was mildly limited in all directions. Visual field tests of both eyes showed a large central scotoma. Laboratory tests revealed an elevation of myeloperoxidase-anti-neutrophil cytoplasmic antibody. Facial computed tomography demonstrated a thickened mucosal membrane in the entire ethmoidal sinus, and the posterosuperior walls of Onodi cells filled with infiltrative lesions had thinned. Orbital magnetic resonance imaging showed severe inflammation in the orbital apex. From these clinical findings, the patient was diagnosed with GPA presenting with OAS associated with ethmoid sinusitis. Emergent endoscopic sinus surgery was performed for biopsy and debridement of the ethmoidal and sphenoid sinusitis to decompress the optic nerve. One day after endoscopic sinus surgery, the patient’s VA and visual field were improved, and steroid pulse therapy was commenced postoperatively. Four days later, VA had recovered to 1.0 in both eyes, and eye movement and visual field had were improved. Although OAS is a rare manifestation, early surgical treatment should be considered when the orbital lesion presents as risk of rapid deterioration of visual function in patients with GPA.


2014 ◽  
Vol 54 (2) ◽  
pp. 158-161
Author(s):  
Hiroaki Iwanami ◽  
Hirotaka Katoh ◽  
Youhei Ohnaka ◽  
Masashi Nakajima ◽  
Mitsuru Kawamura

2005 ◽  
Vol 140 (2) ◽  
pp. 236.e1-236.e8 ◽  
Author(s):  
Kenneth S. Shindler ◽  
Grant T. Liu ◽  
Richard B. Womer

IDCases ◽  
2021 ◽  
pp. e01232
Author(s):  
Grace D. Cullen ◽  
Tara M. Davidson ◽  
Zachary Yetmar ◽  
Bobbi S. Pritt ◽  
Daniel C. DeSimone

1985 ◽  
Vol 99 (6) ◽  
pp. 597-599 ◽  
Author(s):  
R. C. Nayar ◽  
R. P. Mathur ◽  
A. Gulati ◽  
S. B. S. Mann

2008 ◽  
Vol 60 (1) ◽  
pp. 62-65 ◽  
Author(s):  
Vivek Sasindran ◽  
A. Ravikumar ◽  
Senthil

1997 ◽  
Vol 17 (3) ◽  
pp. 178???182 ◽  
Author(s):  
Kyle Balch ◽  
Paul H. Phillips ◽  
Nancy J. Newman

Author(s):  
AC Prado-Ribeiro ◽  
AC Luiz ◽  
MA. Montezuma ◽  
MP Mak ◽  
AR Santos-Silva ◽  
...  

2020 ◽  
Vol 1 (1) ◽  
pp. 20-24
Author(s):  
Daniela Vrînceanu ◽  
B. Bănică ◽  
Adriana Nica ◽  
Alina Popa-Cherecheanu

The superior orbital apex syndrome is a relatively uncommon complication of midface maxillofacial trauma. The clinical symptoms consist in ophthalmoplegia, palpebral ptosis, exophthalmia, fixed mydriasis, retrobulbar pain and supraorbital nerve hypoesthesia by involvement of the third (oculomotor nerve), fourth (trochlear), fifth (trigeminal) and sixth nerve (abducens). If there is involvement of the optical nerve, the syndrome is termed - orbital apex syndrome. In this article, we will present the case of a 33-years old male, victim of human aggression with traumatic superior orbital apex syndrome. We discuss details of diagnosis and surgical treatment. We will make, also, a review of literature on this subject. Even if the actual therapeutic algorithm is currently a matter of controversy, the generally accepted therapy plane initiated with a high dose of corticosteroids. Fine slice CT scan examination is mandatory for the correct planning. If the CT scan reveals a highly displaced maxillo-zygomatic complex fracture with or without orbital blow-out fracture, we recommend early surgical intervention after the resolving of the periorbital hematoma within 5 to 10 days ideally if concomitant intracranial injury or other conditions permit it. The early restoration of the orbital anatomy and volume will create the basis for cranial nerve decompression and function at the level of superior orbital fissure.


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