scholarly journals Reverse Total Shoulder Replacement with Minimal ACJ Excision Arthroplasty for Management of Massive ACJ Cyst - A Case Report

2014 ◽  
Vol 8 (1) ◽  
pp. 298-301 ◽  
Author(s):  
Shahril R Shaarani ◽  
Hannan Mullett

Massive acromioclavicular joint (ACJ) cysts are an extremely rare cause of shoulder impairment and with limited consensus in its management. We present the first published case report of a patient with a massive ACJ cyst treated with a reverse total shoulder replacement with minimal ACJ excision arthroplasty.

2020 ◽  
Vol 16 (2) ◽  
pp. 102-105
Author(s):  
Ji-An Choi ◽  
Jung-Ha Kwak ◽  
Kwang-Ryeol Lim ◽  
Chung-Min Yoon

Prosthetic joint infection is a rare but serious complication of total shoulder replacement. After infection control, shoulder reconstruction is also required to repair the shoulder defect. The shoulder is a complex structure consisting of mechanical soft tissue and the joint, making reconstruction challenging. A 78-year-old female patient was diagnosed of wound necrosis and exposed prosthesis due to prosthetic joint infection after reverse total shoulder replacement. The infection was controlled with appropriate antibiotic treatment, and the necrotic tissue was removed by radical debridement. To reduce the size of the massive shoulder defect and minimize exposure of the prosthesis, negativepressure wound therapy (NPWT) was administered for 8 weeks and shoulder reconstruction was performed using a pedicled latissimus dorsi (LD) flap and split-thickness skin graft. The patient recovered without any complications, and no reinfection of the surgical site or other complications were observed during follow-up visits. This report demonstrates that antibiotic treatment, radical debridement of necrotic tissue, NPWT, and shoulder reconstruction using a pedicled LD flap can be an effective method of treatment for prosthetic joint infection with exposed prosthesis, tissue necrosis, and massive shoulder defect.


Author(s):  
Peter B MacDonald ◽  
Martin J Petrak ◽  
Trevor C Gascoyne ◽  
Eric Richard Bohm

Failure analysis was performed for two humeral tray components which fractured in vivo after only 6 and 9 months. Implant retrieval analysis indicated that the components failed due to fatigue failure initiating from a small radius fillet at the interface of the trunnion and tray regions. Finite element simulations revealed the small radius fillet to have resulted in a large stress concentration and confirmed the possibility for fatigue failure in 6 months. The stress concentration caused by both the small radius fillet and the insufficient tray thickness contributed to the premature fatigue failure of the humeral trays.


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