Dynamic Wrinkle Reduction Strategies for Cable Suspended Membrane Structures

SummaryAttempts were made to demonstrate ultrastructural changes of the tissue thromboplastin after intravenous injection, as a model experiment on the pulmonary microthrombi formation induced by the tissue thromboplastin circulating from venous return.Concentrically arranged membrane structures of the injected thromboplastin disappeared in extremely short time after the injection of the thromboplastin in rabbits. The long sheet membrane of the injected thromboplastin was frequently seen as adhered to the vascular endothelium or to the surface of blood corpuscles. Furthermore, fibrin fibres were formed in contact with the long sheet membrane of the thromboplastin. Membrane structures were not found anywhere in the control rabbits.

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Ultramicroscopic and Coagulation-Fibrinolytic Findings Induced by Tissue Thromboplastin

10.1055/s-0038-1665796 ◽

1979 ◽

Author(s):

H Nagata ◽

T Seya ◽

Y Oguma ◽

M Yamauchi ◽

T Murakoshi ◽

...

Keyword(s):

Endothelial Cells ◽

Electron Microscope ◽

Transmission Electron Microscope ◽

Hypercoagulable State ◽

Membrane Structures ◽

Transmission Electron ◽

Single Sheet ◽

Tissue Thromboplastin ◽

Short Time

We have studied the ultrastructures of tissue thromboplastin (T.Tbp) to demonstrate how It changes during coagulation.[Materials and Methods] T.Tbp from lungs of rabbits was used for these studies. It was injected into ear veins of rabbits. Lungs were resected at several seconds, 10sec, 1 min, 5 min, 24 hrs or 48 hrs after the injection. They were examined by transmission electron microscope.[Results] Concentrically arranged membrane structures of the injected T.Tbp disappeared in extremely short time after the injection. 1 min after the injection, fibrin fibers were seen between single sheet of membrane and endothelial cells of capillaries. In the rabbit which had died suddenly after the injection of T.Tbp, multiple pulmonary thrombi made of fibrin and platelets were seen in capillaries. The endothelial cells of capillaries were destroyed and interstitial tissues were edematous.The hypercoagulable state was seen 10~30sec after the start of the injection, indicating the shortening of r of TEG. Then, it gradually returned the level before injection. Moreover, changes of the measurements of fibrinogen, antiplasmin and prekallikrein were also seen after the injection.

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Ischemic Complications of Pregnancy: Who is at Risk?

US Cardiology Review ◽

10.15420/usc.2016.10.1.14 ◽

2016 ◽

Vol 10 (1) ◽

pp. 14

Author(s):

Sara C Martinez ◽

◽

Sharonne N Hayes ◽

Keyword(s):

Artery Dissection ◽

Cardiac Events ◽

Coronary Artery Dissection ◽

Reduction Strategies ◽

Ischemic Complications ◽

Substantial Risk ◽

History Of ◽

Invasive Management ◽

In Pregnancy

The physiologic demands of pregnancy may either trigger or uncover ischemic heart disease (IHD) via largely unknown mechanisms, leading to an increased mortality compared with nonpregnant individuals. Risk factors for IHD in pregnancy are age, smoking, multiparity, and prior cardiac events. A multidisciplinary team at a referral center is key to coordinating medical or invasive management and inpatient observation. Etiologies may be revealed by experienced angiographers, and are predominantly spontaneous coronary artery dissection, followed by atherosclerotic disease and thrombus, while a significant percentage of women are found to have normal coronary arteries by angiogram. The management of these conditions is varied and, in general, conservative management is preferred with adequate coronary flow and stable hemodynamics. A woman with a history of IHD in pregnancy is at a substantial risk for further complications in future pregnancies and beyond; therefore, aggressive risk factor-reduction strategies and regular cardiology follow-up are imperative to decrease adverse events.

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