Chronic spinal subdural hematomas

1985 ◽  
Vol 63 (4) ◽  
pp. 636-639 ◽  
Author(s):  
Virender K. Khosla ◽  
Vijay K. Kak ◽  
Suresh N. Mathuriya
Keyword(s):  
Subdural Hematoma ◽  
Clinical Course ◽  
Complete Recovery ◽  
Pertinent Literature ◽  
Content Type ◽  
Subdural Hematomas ◽  
Surgical Evacuation ◽  
Spinal Subdural Hematoma ◽  
Fine Print

✓ Two patients with chronic spinal subdural hematomas are described. Both had a fluctuating clinical course, not previously documented in the literature. Surgical evacuation resulted in almost complete recovery. The pathogenesis of spinal subdural hematoma is discussed and the pertinent literature is reviewed.

Acute spinal subdural hematoma

1974 ◽  
Vol 41 (5) ◽  
pp. 627-630 ◽  
Author(s):  
Albert Zilkha ◽  
John M. Nicoletti
Keyword(s):  
Subdural Hematoma ◽  
Neurological Recovery ◽  
Content Type ◽  
Surgical Evacuation ◽  
Spinal Subdural Hematoma ◽  
Fine Print

✓ A case of an acute posttraumatic spinal subdural hematoma is presented. Complete neurological recovery followed surgical evacuation.

The contrast-enhanced CT scan in the diagnosis of isodense subdural hematoma

1979 ◽  
Vol 50 (1) ◽  
pp. 64-69 ◽  
Author(s):  
Fong Y. Tsai ◽  
James E. Huprich ◽  
Hervey D. Segall ◽  
James S. Teal
Keyword(s):  
Cerebral Cortex ◽  
Ct Scan ◽  
Subdural Hematoma ◽  
Correct Diagnosis ◽  
Content Type ◽  
Subdural Hematomas ◽  
Contrast Enhanced Ct ◽  
Contrast Enhanced ◽  
Enhanced Ct ◽  
Fine Print

✓ The authors review 29 cases of surgically-proven isodense subdural hematomas examined by non-contrast and contrast-enhanced computerized tomography scans. Three types of isodense collections were noted: homogeneous isodense collections, mixed-density collections, and gravitational layering within subdural collections. Contrast enhancement within the cerebral cortex, cortical vessels, and subdural membranes led to the correct diagnosis in each case. Contrast-enhanced scans are essential for the evaluation of isodense subdural hematomas.

Acute subdural hematoma

1982 ◽  
Vol 57 (2) ◽  
pp. 254-257 ◽  
Author(s):  
Henry A. Shenkin
Keyword(s):  
Computerized Tomography ◽  
Subdural Hematoma ◽  
Neurological Status ◽  
Neurological Dysfunction ◽  
Acute Subdural Hematoma ◽  
Consecutive Series ◽  
Content Type ◽  
Subdural Hematomas ◽  
Fine Print

✓ In a consecutive series of 39 cases of acute subdural hematoma (SDH), encountered since computerized tomography diagnosis became available, 61.5% were found to be the result of bleeding from a small cortical artery, 25.6% were of venous origin, 7.7% resulted from cerebral contusions, and 5% were acute bleeds into chronic subdural hematomas. Craniotomy was performed promptly on admission, but there was no difference in survival (overall 51.3%) between patients with arterial and venous bleeds. The only apparent factor affecting survival in this series was the preoperative neurological status: 67% of patients who were decerebrate and had fixed pupils prior to operation died. Of patients with less severe neurological dysfunction, only 20% failed to survive.

Isolated symptomatic cervical spinous process fracture requiring surgery

1991 ◽  
Vol 75 (1) ◽  
pp. 131-133 ◽  
Author(s):  
Leonard F. Hirsh ◽  
Luis E. Duarte ◽  
Eric H. Wolfson ◽  
Wilhelm Gerhard
Keyword(s):  
Medical Condition ◽  
Spinous Process ◽  
Complete Recovery ◽  
Neurological Injury ◽  
Pertinent Literature ◽  
Content Type ◽  
Residual Symptoms ◽  
Economic Forces ◽  
Spinous Process Fracture ◽  
Fine Print

✓ Isolated cervical spinous process fractures are common, but are usually considered to be inconsequential. Although such fractures may produce pain, complete recovery without residual symptoms is expected after conservative treatment, and neurological injury does not usually occur. The case of a patient with a persistently symptomatic C-2 spinous process fracture that required surgical treatment for pain relief is reported. A review of the pertinent literature illustrates with unusual clarity the interactions of social, political, and economic forces associated with this medical condition.

Brain scans and echoencephalography in the diagnosis of chronic subdural hematoma

1974 ◽  
Vol 40 (3) ◽  
pp. 347-350 ◽  
Author(s):  
Sheldon R. Hurwitz ◽  
Samuel E. Halpern ◽  
George Leopold
Keyword(s):  
Cerebral Angiography ◽  
Subdural Hematoma ◽  
Chronic Subdural Hematoma ◽  
Content Type ◽  
Brain Scans ◽  
Subdural Hematomas ◽  
Fine Print

✓ Eighteen patients with chronic subdural hematomas were studied by both brain scans and echoencephalography. All cases were verified by cerebral angiography. Brain scanning was accurate in predicting hematomas in 93% of the cases, and echoencephalography in 44%. When hematomas were bilateral or when frontal clots caused no shift in the diencephalic midline, the routine echoencephalogram often was negative. The two procedures are complementary, and serial studies may be helpful in the study of changing clinical situations.

The abnormal pupil in Cheyne-Stokes respiration

1990 ◽  
Vol 72 (5) ◽  
pp. 810-812 ◽  
Author(s):  
Edward F. Gonyea
Keyword(s):  
Subdural Hematoma ◽  
Sympathetic Nerve ◽  
Oculomotor Nerve ◽  
Nerve Compression ◽  
Nerve Paralysis ◽  
Content Type ◽  
Pupillary Dilatation ◽  
Surgical Evacuation ◽  
Fine Print

✓ Cheyne-Stokes respiration commonly induces a rhythmic pupillary dilatation during hyperpnea and constriction during apnea. Failure of a pupil to dilate during hyperventilation indicates underlying sympathetic nerve paralysis. This report deals with an instance in which one pupil failed to constrict during apnea due to oculomotor nerve compression. The periodic respirations and anisocoria disappeared following surgical evacuation of a large ipsilateral subdural hematoma.

Long-term follow-up review of patients with acute and subacute subdural hematomas

1978 ◽  
Vol 48 (3) ◽  
pp. 345-349 ◽  
Author(s):  
Jarl Rosenørn ◽  
Flemming Gjerris
Keyword(s):  
Survival Rate ◽  
Subdural Hematoma ◽  
Acute Subdural Hematoma ◽  
Content Type ◽  
Subdural Hematomas ◽  
Long Term Follow Up ◽  
Fine Print ◽  
Back To Work

✓ The authors present 149 patients suffering from acute (112) and subacute (37) subdural hematomas admitted during the 10-year period 1965 to 1974, with a follow-up period of 2 to 12 years. During the time of observation, 104 patients died and 45 survived; 73% of the patients with acute and 27% with subacute subdural hematomas died. Of the patients with an acute subdural hematoma, 11% went back to work, as against 32% of those with subacute subdural hematomas. The 5-year survival rate was 28% in patients with acute and 76% in patients with subacute subdural hematomas.

Subacute spinal subdural hematoma after spontaneous resolution of cranial subdural hematoma: causal relationship or coincidence?

2004 ◽  
Vol 100 (4) ◽  
pp. 372-374 ◽  
Author(s):  
Carlo Bortolotti ◽  
Huan Wang ◽  
Kenneth Fraser ◽  
Giuseppe Lanzino
Keyword(s):  
Causal Relationship ◽  
Subdural Hematoma ◽  
Spontaneous Resolution ◽  
Spine Injury ◽  
Lumbosacral Spine ◽  
Subdural Space ◽  
Content Type ◽  
Spinal Subdural Hematoma ◽  
Bridging Veins ◽  
Fine Print

✓ The etiopathogenesis of traumatic spinal subdural hematoma (SSH) is uncertain. Unlike the supratentorial subdural space, no bridging veins traverse the spinal subdural space. The authors describe a case of subacute SSH that occurred after spontaneous resolution of traumatic intracranial SDH and suggest a causal relationship between the two. A 23-year-old woman suffered an acute intracranial SDH after a snowboarding accident. There was no clinical or radiological evidence of spine injury. Conservative management of the supratentorial SDH resulted in spontaneous radiologically documented resolution with redistribution of blood in the subdural space. Four days after the injury, the patient started noticing new onset of mild low-back pain. The pain progressively worsened. Magnetic resonance imaging of the lumbosacral spine 10 days after the original injury revealed a large L4—S2 SDH. Ten days after the original injury, bilateral L5—S1 laminotomy and drainage of the subacute spinal SDH were performed. The patient experienced immediate pain relief. The authors hypothesize that in some cases spinal SDH may be related to redistribution of blood from the supratentorial subdural space.

Diagnosis and treatment of middle fossa arachnoid cysts and subdural hematomas

1981 ◽  
Vol 54 (3) ◽  
pp. 366-369 ◽  
Author(s):  
Ludwig M. Auer ◽  
Bernd Gallhofer ◽  
Gunther Ladurner ◽  
Wolf-Dieter Sager ◽  
Fritz Heppner ◽  
...  
Keyword(s):  
Ct Scan ◽  
Subdural Hematoma ◽  
Arachnoid Cysts ◽  
Middle Fossa ◽  
Skull Bone ◽  
Temporal Fossa ◽  
Content Type ◽  
Clear Cut ◽  
Subdural Hematomas ◽  
Fine Print

✓ Nine cases with temporal fossa arachnoid cysts were diagnosed by computerized tomography (CT). Five patients also had subdural hematomas, three of them following head trauma. When the hematoma was chronic and of equal hypodensity with the cyst, a clear-cut differentiation was not possible from the CT scan. The presence of a subdural hematoma could only be suggested by thickened arachnoid structures crossing the hypodense area, indicating the wall between cyst and hematoma. The cyst could often be diagnosed by bulging of the skull bone and a temporal lobe defect. Differences in density between cyst and hematoma, such as in subacute subdural hematoma, delineated both entities. Typical examples are demonstrated. Treatment consisted of evacuation of the hematoma and excision of the cyst in all cases.

Subdural hematoma secondary to metastatic dural carcinomatosis

1974 ◽  
Vol 41 (5) ◽  
pp. 610-613 ◽  
Author(s):  
Richard W. Leech ◽  
F. Tod Welch ◽  
George A. Ojemann
Keyword(s):  
Tumor Growth ◽  
Subdural Hematoma ◽  
Metastatic Adenocarcinoma ◽  
Content Type ◽  
Subdural Hematomas ◽  
Fine Print

✓ A case is reported in which bilateral subdural hematomas were caused by metastatic adenocarcinoma in the subdural membranes. This diffuse dural carcinomatosis was associated with widespread intravascular tumor growth and a marked vascular and fibrous proliferation, interpreted as a tumor-induced angiodesmoplasia. The hematomas resulted from separation and hemorrhage into this highly vascular abnormal areolar layer of the dura.

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