Faculty Opinions recommendation of Sox2 regulates cholinergic amacrine cell positioning and dendritic stratification in the retina.

Author(s):  
Robert Burgess ◽  
Abby Tadenev
2014 ◽  
Vol 34 (30) ◽  
pp. 10109-10121 ◽  
Author(s):  
I. E. Whitney ◽  
P. W. Keeley ◽  
A. J. St. John ◽  
A. G. Kautzman ◽  
J. N. Kay ◽  
...  

1985 ◽  
Vol 20 (2) ◽  
pp. 286-290 ◽  
Author(s):  
Colin J. Barnstable ◽  
Raphael Hofstein ◽  
Kimio Akagawa

2016 ◽  
Vol 416 (1) ◽  
pp. 123-135 ◽  
Author(s):  
Stéphanie Grimbert ◽  
Kyria Tietze ◽  
Michalis Barkoulas ◽  
Paul W. Sternberg ◽  
Marie-Anne Félix ◽  
...  

2011 ◽  
Vol 136 (6) ◽  
pp. 617-636 ◽  
Author(s):  
Kazushige Ogawa ◽  
Natsuki Takemoto ◽  
Maki Ishii ◽  
Elena B. Pasquale ◽  
Takayuki Nakajima

2005 ◽  
Vol 94 (6) ◽  
pp. 4196-4208 ◽  
Author(s):  
Ajithkumar Warrier ◽  
Salvador Borges ◽  
David Dalcino ◽  
Cameron Walters ◽  
Martin Wilson

The Ca2+ that promotes transmitter release is generally thought to enter presynaptic terminals through voltage-gated Ca2+channels. Using electrophysiology and Ca2+ imaging, we show that, in amacrine cell dendrites, at least some of the Ca2+ that triggers transmitter release comes from endoplasmic reticulum Ca2+ stores. We show that both inositol 1,4,5-trisphosphate receptors (IP3Rs) and ryanodine receptors (RyRs) are present in these dendrites and both participate in the elevation of cytoplasmic [Ca2+] during the brief depolarization of a dendrite. Only the Ca2+ released through IP3Rs, however, seems to promote the release of transmitter. Antagonists for the IP3R reduced transmitter release, whereas RyR blockers had no effect. Application of an agonist for metabotropic glutamate receptor, known to liberate Ca2+ from internal stores, enhanced both spontaneous and evoked transmitter release.


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