A Case of Acute Esophageal Necrosis in a Patient with Diabetic Ketoacidosis

2012 ◽  
Vol 83 (1) ◽  
pp. 117 ◽  
Author(s):  
Jae Hyoung Im ◽  
Min Su Kim ◽  
Seung Yoen Lee ◽  
So Hun Kim ◽  
Seong Bin Hong ◽  
...  
2016 ◽  
Vol 111 ◽  
pp. S743-S744
Author(s):  
Russell Rosenblatt ◽  
Baris Alten ◽  
Shirley Cohen-Mekelburg ◽  
David Wan

2018 ◽  
Vol 2018 ◽  
pp. 1-3 ◽  
Author(s):  
Zachary Field ◽  
Jacqueline Kropf ◽  
Meghan Lytle ◽  
Giselle Castaneira ◽  
Mario Madruga ◽  
...  

Background. Acute esophageal necrosis is an uncommon clinical disorder diagnosed on endoscopy as a black esophagus. It has a multifactorial etiology that probably represents a combination of poor nutritional status, gastric outlet obstruction, and ischemia secondary to hypoperfusion of the distal esophagus. It typically occurs in older males with comorbidities. Case. A 37-year-old woman presented with diabetic ketoacidosis and hematemesis. At esophagogastroduodenoscopy acute esophageal necrosis was diagnosed. The treatment included fluid and electrolyte management, insulin, and a proton pump inhibitor. She improved and left the hospital on day 3. Conclusion. Diabetic ketoacidosis can result in a profound osmotic diuresis, fluid loss, and hypoperfusion of the distal esophagus. This condition can then lead to ischemic injury and acute esophageal necrosis. Awareness of the possibility of its presence in young women with hematemesis and poorly controlled diabetes is important since early identification with esophagogastroduodenoscopy is necessary to prevent serious postnecrotic complications.


2018 ◽  
Vol 190 (35) ◽  
pp. E1049-E1049 ◽  
Author(s):  
Hourmazd Haghbayan ◽  
Avijeet K. Sarker ◽  
Eric A. Coomes

2020 ◽  
Vol 2020 ◽  
pp. 1-3
Author(s):  
Linda P. Vien ◽  
Ho-Man Yeung

Acute esophageal necrosis (AEN) is a rare clinical diagnosis that primarily affects the distal third of the esophagus. AEN causes odynophagia, leading to decreased oral intake and food avoidance. AEN can arise in critically ill patients with multiple comorbidities and is an uncommon complication of diabetic ketoacidosis (DKA). We present a case of a young female with poorly controlled, insulin-dependent diabetes mellitus type 2 who developed odynophagia, small volume coffee-ground emesis, and inability to tolerate oral intake after resolution of DKA. She was found to have esophagitis with esophageal necrosis in the middle third of the esophagus on upper gastrointestinal endoscopy. She was subsequently treated with fluid resuscitation and gastric acid suppression and improved clinically with slow advancements in her diet. The location of her lesion in the more vascularized middle one-third of the esophagus and lack of significant blood pressure variations during her hospital stay make her case unique. Thus, AEN should be considered in the differential diagnosis for critically ill patients who present with vague symptoms such as odynophagia and gastrointestinal bleeding.


2021 ◽  
Vol 8 (5) ◽  
pp. e00568
Author(s):  
Aleksandar Gavric ◽  
Samo Plut ◽  
Rok Dezman ◽  
Gregor Novak

2020 ◽  
Vol 10 (2) ◽  
Author(s):  
Dustin J. Uhlenhopp ◽  
Gregory Pagnotta ◽  
Tagore Sunkara

Acute esophageal necrosis, also known as black esophagus or acute necrotizing esophagitis is a rare condition with roughly 154 cases reported in the literature. This condition is classically diagnosed on its endoscopic findings of a circumferentially black esophagus that abruptly ends at the gastroesophageal junction and transitions to normal gastric mucosa. When present, acute esophageal necrosis potentially signifies a poor prognosis with an overall mortality up to 36%. This case report describes a critically ill patient with multiple comorbidities that was found to have acute esophageal necrosis the entire length of the esophagus secondary to ischemia/hypoperfusion that was caused by diabetic ketoacidosis. The patient had a prolonged hospitalization but was ultimately discharged in stable condition. We also review the literature of this rare esophageal condition.


2014 ◽  
Vol 109 ◽  
pp. S233
Author(s):  
Xi Chen ◽  
Daniel Eshtiaghpour ◽  
Anuj Datta ◽  
Sofiya Reicher ◽  
Viktor Eysselein

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