Operative Methods of Chronic Anteromedial Rotatory Instability of Knee: Advancement and Bony Reinsertion Operation of Tibial Collateral Ligament

1983 ◽  
Vol 18 (2) ◽  
pp. 281
Author(s):  
Jung Man Kim ◽  
Won Hyung Shin
2020 ◽  
Vol 28 (12) ◽  
pp. 3700-3708 ◽  
Author(s):  
S. Ball ◽  
J. M. Stephen ◽  
H. El-Daou ◽  
A. Williams ◽  
Andrew A. Amis

Abstract Purpose The purpose of this study was to determine the contribution of each of the ACL and medial ligament structures in resisting anteromedial rotatory instability (AMRI) loads applied in vitro. Methods Twelve knees were tested using a robotic system. It imposed loads simulating clinical laxity tests at 0° to 90° flexion: ±90 N anterior–posterior force, ±8 Nm varus–valgus moment, and ±5 Nm internal–external rotation, and the tibial displacements were measured in the intact knee. The ACL and individual medial structures—retinaculum, superficial and deep medial collateral ligament (sMCL and dMCL), and posteromedial capsule with oblique ligament (POL + PMC)—were sectioned sequentially. The tibial displacements were reapplied after each cut and the reduced loads required allowed the contribution of each structure to be calculated. Results For anterior translation, the ACL was the primary restraint, resisting 63–77% of the drawer force across 0° to 90°, the sMCL contributing 4–7%. For posterior translation, the POL + PMC contributed 10% of the restraint in extension; other structures were not significant. For valgus load, the sMCL was the primary restraint (40–54%) across 0° to 90°, the dMCL 12%, and POL + PMC 16% in extension. For external rotation, the dMCL resisted 23–13% across 0° to 90°, the sMCL 13–22%, and the ACL 6–9%. Conclusion The dMCL is the largest medial restraint to tibial external rotation in extension. Therefore, following a combined ACL + MCL injury, AMRI may persist if there is inadequate healing of both the sMCL and dMCL, and MCL deficiency increases the risk of ACL graft failure.


1935 ◽  
Vol 27 (2) ◽  
pp. 245-247 ◽  
Author(s):  
S.H. Wetzler ◽  
D.V. Elconin

2018 ◽  
Vol 100-B (8) ◽  
pp. 1060-1065 ◽  
Author(s):  
J-T. Hwang ◽  
M. N. Shields ◽  
L. J. Berglund ◽  
A. W. Hooke ◽  
J. S. Fitzsimmons ◽  
...  

Aims The aim of this study was to evaluate two hypotheses. First, that disruption of posterior bundle of the medial collateral ligament (PMCL) has to occur for the elbow to subluxate in cases of posteromedial rotatory instability (PMRI) and second, that ulnohumeral contact pressures increase after disruption of the PMCL. Materials and Methods Six human cadaveric elbows were prepared on a custom-designed apparatus which allowed muscle loading and passive elbow motion under gravitational varus. Joint contact pressures were measured sequentially in the intact elbow (INTACT), followed by an anteromedial subtype two coronoid fracture (COR), a lateral collateral ligament (LCL) tear (COR + LCL), and a PMCL tear (COR + LCL + PMCL). Results There was no subluxation or joint incongruity in the INTACT, COR, and COR + LCL specimens. All specimens in the COR + LCL + PMCL group subluxated under gravity-varus loads. The mean articular contact pressure of the COR + LCL group was significantly higher than those in the INTACT and the COR groups. The mean articular contact pressure of the COR + LCL + PMCL group was significantly higher than that of the INTACT group, but not higher than that of the COR + LCL group. Conclusion In the presence of an anteromedial fracture and disruption of the LCL, the posterior bundle of the MCL has to be disrupted for gross subluxation of the elbow to occur. However, elevated joint contact pressures are seen after an anteromedial fracture and LCL disruption even in the absence of such subluxation. Cite this article: Bone Joint J 2018;100-B:1060–5.


2017 ◽  
Vol 26 (9) ◽  
pp. 1636-1643 ◽  
Author(s):  
Enrico Bellato ◽  
Youngbok Kim ◽  
James S. Fitzsimmons ◽  
Alexander W. Hooke ◽  
Lawrence J. Berglund ◽  
...  

2004 ◽  
Vol 9 (4) ◽  
pp. 380-385 ◽  
Author(s):  
Makoto Kurimura ◽  
Hideo Matsumoto ◽  
Kyosuke Fujikawa ◽  
Yoshiaki Toyama

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