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Death receptor 5 (DR5) marks the highly pathogenic interacting GM-CSF+ T helper cells and IL-23+ macrophages rendering it as an attractive therapeutic target of Rheumatoid arthrit
Orthopedic & Muscular System
◽
10.4172/2161-0533.s1.015
◽
2014
◽
Vol s3
(01)
◽
Keyword(s):
Therapeutic Target
◽
T Helper Cells
◽
Death Receptor
◽
Death Receptor 5
◽
T Helper
◽
Highly Pathogenic
◽
Gm Csf
◽
Helper Cells
◽
Attractive Therapeutic Target
Download Full-text
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◽
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Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature
◽
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◽
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◽
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◽
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Faculty Opinions recommendation of STAT5 programs a distinct subset of GM-CSF-producing T helper cells that is essential for autoimmune neuroinflammation.
Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature
◽
10.3410/f.725244071.793526942
◽
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◽
Author(s):
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◽
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IL-17 and GM-CSF Expression Are Antagonistically Regulated by Human T Helper Cells
Science Translational Medicine
◽
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◽
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◽
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(241)
◽
pp. 241ra80-241ra80
◽
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◽
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◽
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Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature
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◽
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◽
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◽
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Type 17 T-helper cells might be a promising therapeutic target for systemic lupus erythematosus
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◽
10.1038/ncprheum0815
◽
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◽
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◽
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◽
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◽
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◽
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