Percutaneous closure of an Iatrogenic fistula from the left ventricle to the coronary sinus

2014 ◽  
Vol 86 (2) ◽  
pp. E99-E102
Author(s):  
Zachary M. Gertz ◽  
Jose-Luis E. Velazquez-Cecena ◽  
John. V. Ian Nixon
Heart ◽  
1981 ◽  
Vol 45 (1) ◽  
pp. 101-104 ◽  
Author(s):  
K M McGarry ◽  
J Stark ◽  
F J Macartney

EP Europace ◽  
2017 ◽  
Vol 19 (suppl_3) ◽  
pp. iii211-iii212
Author(s):  
B. Papelbaum ◽  
SS. Galvao Filho ◽  
JT. Medeiros De Vasconcelos ◽  
C. Eduardo Duarte ◽  
R. Castro Galvao ◽  
...  

2012 ◽  
Vol 28 (5) ◽  
pp. 612.e9-612.e10
Author(s):  
Kiyotaka Watanabe ◽  
Kozo Hoshino ◽  
Kaoru Dohi ◽  
Naritatsu Saito ◽  
Takafumi Hashimoto ◽  
...  

Author(s):  
Giulia Poretti ◽  
Mauro Lo Rito ◽  
Alessandro Varrica ◽  
Alessandro Frigiola

Abstract Background Isolated coronary arteriovenous fistulas are extremely rare, accounting for 0.08–0.4% of all congenital heart disease. Closure of the fistula is recommended in cases of large dimensions, relevant left–right shunt, or ischaemic events. Thrombosis of the coronary aneurysms may occur as a postoperative complication. Case summary We report a case of a coronary fistula between the circumflex artery and coronary sinus with giant aneurysm. After a failed percutaneous closure attempt, the patient was surgically treated without major postoperative complications. Despite therapeutic anticoagulation and antiplatelet therapy, she presented at clinical follow-up with thrombosis of the dilated coronary artery without signs or symptoms of ischaemia. Discussion Management of coronary artery fistula may be challenging in cases in which initial percutaneous closure is unsuccessful. This particular case also highlights the importance of close follow-up, despite optimal therapy, to detect potentially lethal complications related to the low flow in the dilated coronary aneurysm.


1965 ◽  
Vol 208 (5) ◽  
pp. 946-953 ◽  
Author(s):  
Skoda Afonso ◽  
George G. Rowe ◽  
Jorge E. Lugo ◽  
Charles W. Crumpton

Only a part of heat produced by the left ventricle is removed by the coronary blood. During a cold saline infusion into the right ventricle, LV myocardial temperature decreases and the myocardium loses a measurable amount of heat. A part of this heat is also removed by the coronary blood. If simultaneous thermal curves are recorded from the aorta and coronary sinus during the infusion it is possible to calculate left ventricle heat production by the following formula: H = LV weight x ΔT x Δt x K x 60:A, where ΔT = myocardial temperature drop during the infusion; Δt = coronary sinus-aorta blood temperature difference prior to infusion; K = specific heat of myocardium; A = difference of areas of superimposed coronary sinus and aorta's thermal curves. Heat production estimated by the formula in 19 determinations has been compared with the heat production calculated from myocardial oxygen consumption. Measurements obtained by this method seem to be representative of left ventricle heat production.


2019 ◽  
Vol 35 (5) ◽  
pp. 748-751
Author(s):  
Sou Takenaka ◽  
Jun Suzuki ◽  
Akihiko Ueno ◽  
Takashi Uchiyama

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