Hypoplasia of the Coronary Sinus with Coronary Venous Drainage into the Left Ventricle by Way of the Thebesian System

CHEST Journal ◽  
1975 ◽  
Vol 68 (3) ◽  
pp. 384-385 ◽  
Author(s):  
Sam A. Kinard
Heart ◽  
1981 ◽  
Vol 45 (1) ◽  
pp. 101-104 ◽  
Author(s):  
K M McGarry ◽  
J Stark ◽  
F J Macartney

2014 ◽  
Vol 86 (2) ◽  
pp. E99-E102
Author(s):  
Zachary M. Gertz ◽  
Jose-Luis E. Velazquez-Cecena ◽  
John. V. Ian Nixon

EP Europace ◽  
2017 ◽  
Vol 19 (suppl_3) ◽  
pp. iii211-iii212
Author(s):  
B. Papelbaum ◽  
SS. Galvao Filho ◽  
JT. Medeiros De Vasconcelos ◽  
C. Eduardo Duarte ◽  
R. Castro Galvao ◽  
...  

2012 ◽  
Vol 28 (5) ◽  
pp. 612.e9-612.e10
Author(s):  
Kiyotaka Watanabe ◽  
Kozo Hoshino ◽  
Kaoru Dohi ◽  
Naritatsu Saito ◽  
Takafumi Hashimoto ◽  
...  

1965 ◽  
Vol 208 (5) ◽  
pp. 946-953 ◽  
Author(s):  
Skoda Afonso ◽  
George G. Rowe ◽  
Jorge E. Lugo ◽  
Charles W. Crumpton

Only a part of heat produced by the left ventricle is removed by the coronary blood. During a cold saline infusion into the right ventricle, LV myocardial temperature decreases and the myocardium loses a measurable amount of heat. A part of this heat is also removed by the coronary blood. If simultaneous thermal curves are recorded from the aorta and coronary sinus during the infusion it is possible to calculate left ventricle heat production by the following formula: H = LV weight x ΔT x Δt x K x 60:A, where ΔT = myocardial temperature drop during the infusion; Δt = coronary sinus-aorta blood temperature difference prior to infusion; K = specific heat of myocardium; A = difference of areas of superimposed coronary sinus and aorta's thermal curves. Heat production estimated by the formula in 19 determinations has been compared with the heat production calculated from myocardial oxygen consumption. Measurements obtained by this method seem to be representative of left ventricle heat production.


1980 ◽  
Vol 6 (3) ◽  
pp. 247-254 ◽  
Author(s):  
Arthur S. Pickoff ◽  
Rafael Sequeira ◽  
Pedro L. Ferrer ◽  
Dolores Tamer ◽  
Vicki Bennett ◽  
...  

2019 ◽  
Vol 35 (5) ◽  
pp. 748-751
Author(s):  
Sou Takenaka ◽  
Jun Suzuki ◽  
Akihiko Ueno ◽  
Takashi Uchiyama

1978 ◽  
Vol 234 (2) ◽  
pp. H163-H166 ◽  
Author(s):  
H. K. Nakazawa ◽  
D. L. Roberts ◽  
F. J. Klocke

The fractions of left anterior descending (LAD) and circumflex (LC) inflow drainage into the canine great cardiac vein (GCV) and coronary sinus (CS) have been quantitated by use of a right heart bypass preparation in which GCV outflow was isolated from the remainder of CS outflow. Following direct LAD injection of indocyanine green dye (ICG), 63 +/- 8% (SD) of the total amount of dye recovered appeared in GCV outflow and the remainder in CS outflow. CS recovery of ICG was decreased appreciably by ligation of epicardial venous connections between the LAD and LC beds, but was not affected by selective reductions of LAD or LC inflow. Only 3 +/- 3% of ICG injected into the LC was recovered in GVC outflow under basal conditions, and these low values were not affected measurably by selective reductions of LAD or LC inflow. CS drainage of LAD inflow could be augmented by selective increments of GCV pressure exceeding 7-10 mmHg. Increments of LC drainage in GCV outflow required CS pressures that exceeded GCV pressures by greater than 10 mmHg.


2011 ◽  
Vol 22 (2) ◽  
pp. 145-151 ◽  
Author(s):  
Karolina M. G. Bilska ◽  
Claudia M. J. Kehrens ◽  
Gillian Riley ◽  
Robert H. Anderson ◽  
Jan Marek

AbstractReal-time three-dimensional echocardiography can surpass simple cross-sectional echocardiography in providing precise details of cardiac lesions. For the purpose of optimising treatment, we describe our findings with real-time three-dimensional echocardiography when interrogating different types of communications permitting interatrial shunting. A three-dimensional reconstruction of defects within the oval fossa enabled reliable identification of location, size, and integrity of surrounding rims. In the superior sinus venosus defect associated with partially anomalous pulmonary venous drainage, three-dimensional reconstruction helped to provide a better understanding of the relationship between the interatrial communication, the orifice of the superior caval vein, and the connections of the right upper pulmonary vein. In the defect opening infero-posteriorly within the oval fossa, three-dimensional reconstruction helped to avoid the risk of potentially inappropriate closure of the defect by suturing the hyperplastic Eustachian valve to the atrial wall, which could have diverted the inferior caval venous return into the left atrium, or obstructed the caval venous orifice. In the coronary sinus defect, three-dimensional echocardiography provided a ‘face to face’ view of the entire coronary sinus roof, showing a circular defect communicating with the cavity of the left atrium. Acquisition of the full-volume data sets took less than 2 minutes for the patients having defects within the oval fossa, and no more than 3 minutes for the patients with the sinus venosus and coronary sinus defects. Post-processing for the defects in the oval fossa took from 5 to 8 minutes, and from 12 to 16 minutes for the more complicated defects.ConclusionCross-sectional two-dimensional echocardiography can establish correct diagnosis in all types of atrial communications; however, real-time three-dimensional reconstruction provides additional value to the surgeon and interventionist for better understanding of spatial intracardiac morphology.


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