Intraoperative Somatosensory Evoked Potential Diagnoses in Brachial Plexus Lesions

1990 ◽  
pp. 227-232
Author(s):  
P. Mailänder ◽  
E. Schaller ◽  
M. Möckel ◽  
A. Berger ◽  
G. F. Walter
Keyword(s):  
Brachial Plexus ◽  
Evoked Potential ◽  
Somatosensory Evoked Potential

EVALUATION OF BRACHIAL PLEXUS INJURY DURING STERNAL RETRACTION USING SOMATOSENSORY EVOKED POTENTIAL MONITORING

1994 ◽  
Vol 81 (SUPPLEMENT) ◽  
pp. A487
Author(s):  
W. S. Jellish ◽  
B. Blakeman ◽  
T. Sanders ◽  
B. Nulman ◽  
E. Hudson
Keyword(s):  
Brachial Plexus ◽  
Evoked Potential ◽  
Brachial Plexus Injury ◽  
Somatosensory Evoked Potential ◽  
Evoked Potential Monitoring ◽  
Potential Monitoring

Somatosensory evoked potential: Preventing brachial plexus injury in transaxillary robotic surgery

2019 ◽  
Vol 129 (11) ◽  
pp. 2663-2668 ◽  
Author(s):  
Shuo Huang ◽  
Meghan E. Garstka ◽  
Mohammed A. Murcy ◽  
Jeremey A. Bamford ◽  
Sang‐Wook Kang ◽  
...  
Keyword(s):  
Robotic Surgery ◽  
Brachial Plexus ◽  
Evoked Potential ◽  
Brachial Plexus Injury ◽  
Somatosensory Evoked Potential

Detection of Brachial Plexus Dysfunction by Somatosensory Evoked Potential Monitoring—A Report of Two Cases

1984 ◽  
Vol 60 (3) ◽  
pp. 248-252 ◽  
Author(s):  
Michael E. Mahla ◽  
Donlin M. Long ◽  
Joy McKennett ◽  
Carol Green ◽  
Robert W. McPherson
Keyword(s):  
Brachial Plexus ◽  
Evoked Potential ◽  
Somatosensory Evoked Potential ◽  
Evoked Potential Monitoring ◽  
Potential Monitoring

Acute Isovolemic Anemia Does Not Impair Peripheral or Central Nerve Conduction

2003 ◽  
Vol 99 (3) ◽  
pp. 546-551 ◽  
Author(s):  
Richard B. Weiskopf ◽  
Michael J. Aminoff ◽  
Harriet W. Hopf ◽  
John Feiner ◽  
Maurene K. Viele ◽  
...  
Keyword(s):  
Spinal Cord ◽  
Cerebral Cortex ◽  
Brachial Plexus ◽  
Conduction Velocity ◽  
Nerve Conduction ◽  
Evoked Potential ◽  
Cervical Spinal Cord ◽  
Hemoglobin Concentration ◽  
Somatosensory Evoked Potential ◽  
Acute Anemia

Background Previous studies have found subtle slowing of responses in tests of addition and digit-symbol substitution during acute severe isovolemic anemia to a hemoglobin concentration of 5 g/dl in healthy unmedicated humans. In this study, the authors tested the hypothesis that such changes relate to the slowing of afferent neural traffic. Methods The median nerve was stimulated at the wrist in seven healthy unmedicated volunteers before and after induction of acute isovolemic anemia to a nadir hemoglobin concentration of 5.1 +/- 0.3 g/dl (mean +/- SD). Times for neural impulses to travel from the stimulus site to the brachial plexus, cervical spinal cord, and cerebral cortex were measured using somatosensory evoked potentials. Tests were repeated during acute anemia with the subject breathing oxygen. As a control for time and intrasubject variation, the testing was repeated on a separate day when anemia was not produced at times equivalent to those on the experimental day. Results Induced acute severe isovolemic anemia decreased nerve conduction latencies from the wrist to the contralateral cerebral cortex (i.e., to the N20 peak) by 2.3 +/- 1.6% compared with values at a mean hemoglobin concentration of 12.7 g/dl (P < 0.01). These decreased latencies were due solely to an increased peripheral conduction velocity, from the wrist to the brachial plexus (P < 0.05), and were not altered when subjects breathed oxygen (P > 0.05). Conduction velocity from the brachial plexus or cervical spinal cord to the cerebral cortex did not change with acute anemia (P > 0.05). Latencies did not differ on the control day among the times of testing (all P > 0.05), nor did they differ at baseline between the control and experimental days (all P > 0.05). Conclusion Somatosensory evoked potential latencies were not increased by acute severe isovolemic anemia, making it unlikely that the afferent portion of the neural system is responsible for slowing of cognitive responses previously observed during acute anemia. Because severe isovolemic anemia did not increase somatosensory evoked potential latencies, etiologies other than anemia should be sought if latencies are increased during intraoperative monitoring.

Somatosensory evoked potential monitoring during cardiac surgery: An examination of brachial plexus dysfunction

1997 ◽  
Vol 11 (2) ◽  
pp. 187-191 ◽  
Author(s):  
Douglas Seal ◽  
John Balaton ◽  
Stuart G. Coupland ◽  
Chris J. Eagle ◽  
Charles MacAdams ◽  
...  
Keyword(s):  
Cardiac Surgery ◽  
Brachial Plexus ◽  
Evoked Potential ◽  
Somatosensory Evoked Potential ◽  
Evoked Potential Monitoring ◽  
Potential Monitoring
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