Effect of dopamine on regional myocardial function and oxygen consumption in experimental left ventricular hypertrophy

1991 ◽  
Vol 86 (5) ◽  
pp. 449-460 ◽  
Author(s):  
C. Wright ◽  
H. R. Weiss ◽  
J. Kedem ◽  
P. M. Schooz
1995 ◽  
Vol 25 (2) ◽  
pp. 251A ◽  
Author(s):  
Gerard P. Aurigemma ◽  
John S. Gottdiener ◽  
William H. Gaasch ◽  
David Williams ◽  
Zahid Jamal ◽  
...  

1990 ◽  
Vol 54 (5) ◽  
pp. 563-567 ◽  
Author(s):  
SUSUMU NAKANO ◽  
KAZUHIRO TANIGUCHI ◽  
TOMOHIDE KAWAMOTO ◽  
MASATAKA MITSUNO ◽  
HIKARU MATSUDA ◽  
...  

2014 ◽  
Vol 46 ◽  
pp. 21-21 ◽  
Author(s):  
A Decloedt ◽  
S Ven ◽  
N Van Der Vekens ◽  
D De Clercq ◽  
G van Loon

1976 ◽  
Vol 51 (s3) ◽  
pp. 455s-459s
Author(s):  
Suzanne Oparil ◽  
Lynda Erinoff ◽  
A. Cutilletta

1. Neither nerve-growth-factor antiserum (NGFAS) administered subcutaneously nor 6-hydroxydopamine administered intraventricularly to immature spontaneously hypertensive rats (SHR) inhibited the development of the hypertensive syndrome. In contrast, NGFAS did not affect blood pressure in normotensive Kyoto/Wistar rats. 2. Peripheral vascular resistance was increased and cardiac index decreased in both NGFAS and 6-hydroxydopamine-treated SHR despite preservation of normal blood pressure. 3. NGFAS treatment did not influence the development of left ventricular hypertrophy in SHR, despite the lowering of blood pressure. In contrast, 6-hydroxydopamine caused an attenuation in the development of left ventricular hypertrophy. 4. Indices of left ventricular contractility were depressed by NGFAS treatment but not by 6-hydroxydopamine. 5. Plasma renin activity was unaffected by NGFAS treatment and increased by 6-hydroxydopamine.


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