Bladder prolapse through a patent urachus presenting as an umbilical mass in the newborn: characteristic prenatal sonographic findings and the diagnostic benefit of postnatal cystography

Author(s):  
Aaron D. Hodes ◽  
Gerard Weinberg ◽  
Steven H. Borenstein ◽  
Mark C. Liszewski ◽  
Terry L. Levin
2021 ◽  
pp. 006-009
Author(s):  
Warchol Stanislaw ◽  
Slomska Sylwia ◽  
Roszkowski Tomasz ◽  
Dudek-Warchol Teresa

2012 ◽  
Vol 39 (2) ◽  
pp. 241-242 ◽  
Author(s):  
F. Raga ◽  
F. Bonilla-Musoles ◽  
J. C. Castillo

2010 ◽  
Vol 17 (2) ◽  
pp. 262
Author(s):  
Hyun Shin Choi ◽  
Hae Eun Kim ◽  
Eun Sun Kim ◽  
Soo-Young Oh ◽  
Yun Sil Chang ◽  
...  

Urology ◽  
2021 ◽  
Author(s):  
German Fernando Falke ◽  
Sebastian Tobia Gonzalez ◽  
Leandro Berberian ◽  
Sofia Marchionatti ◽  
Soledad Heredia ◽  
...  

2006 ◽  
Vol 41 (5) ◽  
pp. e5-e7 ◽  
Author(s):  
Brian Lugo ◽  
Jennifer McNulty ◽  
Sherif Emil

2007 ◽  
Vol 42 (12) ◽  
pp. e7-e10 ◽  
Author(s):  
Futoshi Matsui ◽  
Fumi Matsumoto ◽  
Kenji Shimada

2017 ◽  
Vol 24 ◽  
pp. 17-20 ◽  
Author(s):  
Sergey Vavilov ◽  
Janani Krishnan ◽  
Ashish Jiwane ◽  
Antonia W. Shand

2014 ◽  
Vol 34 (1) ◽  
pp. 68-70 ◽  
Author(s):  
B Thapa ◽  
MS Pun

We report a case of bladder prolapse through a patent urachus in a term male neonate with a large, red, tubular, mucosa lined mass inferior to the umbilical cord. A cystic mass communicating with fetal urinary bladder was detected in an antenatal ultrasound in a 26 years primigravida at 18 and 26 weeks gestation. The cyst disappeared at 35 weeks and a new solid mass was noted at the fetal abdominal wall. After birth a protruded mucosal mass inferior to the umbilical cord was noted. Urethral catherisation confirmed communication with bladder. On the second day of life excision of urachus, repair, reduction of bladder and reconstruction of abdominal wall was performed. The patient voided well and was discharged on ninth day without any complication. DOI: http://dx.doi.org/10.3126/jnps.v34i1.7877   J Nepal Paediatr Soc 2014;34(1):68-70  


1998 ◽  
Vol 37 (02) ◽  
pp. 76-79 ◽  
Author(s):  
T. D. Kirchhoff ◽  
W. Burchert ◽  
J. v. d. Hoff ◽  
H. Zeidler ◽  
H. Hundeshagen ◽  
...  

SummaryA 61-year-old female patient presenting with mixed connective tissue disease (Sharp syndrome), underwent a long-term high dose glucocorticoid treatment because of multiple organ manifestations. Under steroid therapy she developed severe osteoporosis resulting in multiple fractures. A dynamic [18F]fluoride PET study in this patient revealed reduced fluoride influx in non-fractured vertebrae. This finding corresponds to pathogenetic concepts which propose an inhibition of bone formation as major cause of glucocorticoid-induced osteoporosis. In the light of the presented case it seems to be promising to evaluate the diagnostic benefit of [18F]fluoride PET in osteoporosis.


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