Ultra-high-resolution C-arm flat-detector CT angiography evaluation reveals 3-fold higher association rate for sporadic intracranial cavernous malformations and developmental venous anomalies: a retrospective study in consecutive 58 patients with 60 cavernous malformations

2016 ◽  
Vol 27 (6) ◽  
pp. 2629-2639 ◽  
Author(s):  
Burak Kocak ◽  
Osman Kizilkilic ◽  
Buge Oz ◽  
Dogu Vuralli Bakkaloglu ◽  
Cihan Isler ◽  
...  
Keyword(s):  
Retrospective Study ◽  
High Resolution ◽  
Ct Angiography ◽  
Association Rate ◽  
Flat Detector ◽  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Developmental Venous Anomalies

Cerebral cavernous malformations and developmental venous anomalies

2010 ◽  
pp. 189-220
Author(s):  
Philippe Metellus ◽  
Siddharth Kharkar ◽  
Doris Lin ◽  
Sumit Kapoor ◽  
Daniele Rigamonti
Keyword(s):  
Cerebral Cavernous Malformations ◽  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Developmental Venous Anomalies

scholarly journals Cerebellar cavernous malformations with and without associated developmental venous anomalies

BMC Neurology ◽  
2013 ◽  
Vol 13 (1) ◽  
Author(s):  
Peifeng Zhang ◽  
Lingtong Liu ◽  
Yong Cao ◽  
Shuo Wang ◽  
Jizong Zhao
Keyword(s):  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Developmental Venous Anomalies

Prevalence of cerebral cavernous malformations associated with developmental venous anomalies increases with age

2017 ◽  
Vol 33 (9) ◽  
pp. 1539-1543 ◽  
Author(s):  
Waleed Brinjikji ◽  
Ali El-Rida El-Masri ◽  
John T. Wald ◽  
Kelly D. Flemming ◽  
Giuseppe Lanzino
Keyword(s):  
Cerebral Cavernous Malformations ◽  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Developmental Venous Anomalies

scholarly journals What the Neurosurgeon needs to know about Cerebral Developmental Venous anomalies

2014 ◽  
Vol 21 (2) ◽  
pp. 175-185
Author(s):  
Hernando Raphael Alvis-Miranda ◽  
Nancy Duarte-Valdivieso ◽  
Gabriel Alcala-Cerra ◽  
Willem Calderon-Miranda ◽  
Marco Zenteno ◽  
...  
Keyword(s):  
Gray Matter ◽  
Arteriovenous Malformations ◽  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Developmental Venous Anomalies ◽  
Venous Angiomas ◽  
Cerebrovascular Malformation

Abstract Venous Angiomas or Developmental venous anomalies (DVA) are extreme variations of normal transmedullary veins that are necessary for the drainage of white and gray matter, also are one type of cerebrovascular malformation (CVM), sharing category with capillary telangiectesias, cavernous malformations (CM), and arteriovenous malformations (AVM), each of which may also be associated with a DVA. DVA are the most commonly encountered CVM, accounting for up to 60% of all CVM. We present a review of the literatura

DEVELOPMENTAL VENOUS ANOMALIES

Neurosurgery ◽  
2009 ◽  
Vol 65 (1) ◽  
pp. 20-30 ◽  
Author(s):  
Stylianos K. Rammos ◽  
Raffella Maina ◽  
Giuseppe Lanzino
Keyword(s):  
Magnetic Resonance Imaging ◽  
Magnetic Resonance ◽  
Intracerebral Hemorrhage ◽  
Vascular Malformations ◽  
Neurological Complications ◽  
Resonance Imaging ◽  
Cavernous Malformations ◽  
Venous Anomalies ◽  
Acute Infarction ◽  
Developmental Venous Anomalies

ABSTRACT DEVELOPMENTAL VENOUS ANOMALIES (DVAs), formerly known as venous angiomas, have become the most frequently diagnosed intracranial vascular malformation. DVAs are currently considered congenital cerebrovascular anomalies with mature venous walls that lack arterial or capillary elements. They are composed of radially arranged medullary veins, which converge in an enlarged transcortical or subependymal collector vein, and have characteristic appearances (caput medusae) on magnetic resonance imaging and angiography. DVAs were once thought to be rare lesions with substantial potential for intracerebral hemorrhage and considerable morbidity. The prevalence of incidental and asymptomatic DVAs has been more apparent since the advent of magnetic resonance imaging; recent cohort studies have challenged the once-held view of isolated DVAs as the cause of major neurological complications. The previously reported high incidence of intracerebral hemorrhage associated with DVAs is currently attributed to coexistent, angiographically occult cavernous malformations. Some patients may still have noteworthy neurological morbidity or die as a result of acute infarction or hemorrhage directly attributed to DVA thrombosis. DVAs can coexist with cavernous malformations and arteriovenous malformations. Such combination or transitional forms of malformations might suggest common pathways in pathogenesis. Recent data support a key role for DVAs in the pathogenesis of mixed vascular malformations.

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