scholarly journals Infectious pancreatic necrosis virus inhibits infectious hematopoietic necrosis virus at the early stage of infection in a time dependent manner during Co-infection in Chinook salmon embryo cell lines

2020 ◽  
Vol 102 ◽  
pp. 361-367 ◽  
Author(s):  
Li-Ming Xu ◽  
Miao Liu ◽  
Jing-Zhuang Zhao ◽  
Guang-Ming Ren ◽  
Ying Dong ◽  
...  
Keyword(s):  
Pancreatic Necrosis ◽  
Early Stage ◽  
Infectious Pancreatic Necrosis Virus ◽  
Embryo Cell ◽  
Dependent Manner ◽  
Necrosis Virus ◽  
Infectious Hematopoietic Necrosis ◽  
Infectious Pancreatic Necrosis ◽  
Pancreatic Necrosis Virus ◽  
Chinook Salmon Embryo

Role of infectious pancreatic necrosis virus (IPNV) infection in co-infections with other viruses

2018 ◽  
Vol 74 (1) ◽  
pp. 6050-2018
Author(s):  
JOANNA MAJ-PALUCH ◽  
MICHAŁ REICHERT
Keyword(s):  
Defense Mechanisms ◽  
Pancreatic Necrosis ◽  
Infectious Pancreatic Necrosis Virus ◽  
Viral Hemorrhagic Septicemia Virus ◽  
Infectious Salmon Anaemia Virus ◽  
Necrosis Virus ◽  
Infectious Hematopoietic Necrosis ◽  
Infectious Pancreatic Necrosis ◽  
Pancreatic Necrosis Virus

Co-infection is an infection of more than one pathogen. In aquatic environment, the most common occurrence is appearance of infectious pancreatic necrosis virus (IPNV) in the presence of other viruses such as infectious hematopoietic necrosis virus (IHNV), viral hemorrhagic septicemia virus (VHSV), infectious salmon anaemia virus (ISAV), or salmonid alphavirus (SAV). In most cases, the IPN virus reduces the proliferation of other viruses in cell cultures or in the internal organs of salmonids, for example in IHNV or ISAV co-infections. However, it happens that there is no significant effect on the multiplication of the virus with which it coexists, e.g. IPNV-VHSV. Body's defense mechanisms, interferon and other interferon-like factors or mutations in the genome play an important role in co-infection..

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